Synergistic effects of repeated transcranial magnetic stimulation and mesenchymal stem cells transplantation on alleviating neuroinflammation and PANoptosis in cerebral ischemia DOI Creative Commons

Shimei Cheng,

Qiying Lu,

Qiuli Liu

и другие.

Journal of Neuroinflammation, Год журнала: 2024, Номер 21(1)

Опубликована: Ноя. 30, 2024

Neuronal death is the primary cause of poor outcomes in cerebral ischemia. The inflammatory infiltration early phase ischemic stroke plays a vital role triggering neuronal death. Either transplantation mesenchymal stem cells (MSCs) derived from humans or repetitive transcranial magnetic stimulation (rTMS) have respectively proved to be neuroprotective and anti-inflammatory However, either treatment above has its limitations. Whether these two therapies synergistic effects on improving neurological function underlying mechanisms remains unclear. This investigation aims elucidate MSCs combined with rTMS recovery post-ischemia. A Sprague-Dawley rat model infarction was induced via transient middle artery occlusion (tMCAO). rats were divided into five groups (n = 50): sham, tMCAO, rTMS, MSCs, + groups. Transplantation human umbilical cord intervention performed 24 h post-stroke. Neurological further assessed several behavioral tests 2,3,5-triphenyltetrazolium chloride (TTC) staining companied Nissl used assess survival. TUNEL staining, western blotting, immunofluorescence, immunohistochemistry, ELISA, flow cytometry employed measure levels neuroinflammation PANoptosis. molecular special therapy distinguished transcriptome sequencing PC12 oxygen-glucose deprivation/reoxygenation (OGD/R) conditions. efficiently reduced lesion volume improved survival (P < 0.05), subsequently functional after stroke. ameliorated PANoptosis neurons accompanied by decreased factors tissue serum during subacute infarction. To explore roles effect, we found that transplanted primarily localized spleen ischemia suppressed splenic inflammation. Meanwhile, significantly protects MSCs-inhibited conditions downregulating REST unveiled sequencing. Our study elucidates an unidentified mechanism which combination could synergistically promote suppress infarction, thus outcomes. may potentially contribute effect against These results are expected provide novel insights protecting injury potential targets

Язык: Английский

A CPApoptosis nano-actuator switches immune-off solid tumors to immune-on for fueling T-cell- based immunotherapy DOI Creative Commons
Ying Luo, Yi Wang, Bo Liu

и другие.

Theranostics, Год журнала: 2025, Номер 15(9), С. 3797 - 3820

Опубликована: Март 3, 2025

Background: Most anticancer agents induce tumor apoptosis, but they often lack immunogenicity and display limited success when combined with mainstream immunotherapies, thus killing cancer cells through multiple cell death modalities as well switching immune-off tumors to immune-on is a strategy great promise. To this end, we developed CPApoptosis (cuproptosis, pyroptosis, apoptosis) nano-actuator for immunologically cold solid tumors. Methods: In study, elesclomol (ES), mitochondrial targeting copper transporter, was encapsulated within bacterial outer membrane vesicles (OMVs). These OMVs were then surface-modified via metal-phenolic self-assembly using Cu2+ tannic acid (TA). Results: The ES released from the in pH-dependent manner. OMV activated non-canonical pyroptotic pathway, leading rupture. on one hand transported mitochondria cuproptosis facilitated by ES, other hand, reduced into Cu+ TA, which catalyzed ROS production oxidative apoptosis. Simultaneously, TA degraded glutathione (GSH), sensitizing cuproptosis. multifactorial mechanisms led release of immunogenic factors lysed cells, stimulating dendritic maturation recruiting cytotoxic T cells. This immune response further amplified αPD-L1 antibody treatment. Conclusion: represents promising approach enhance current therapies, inducing both robust response, potential long-lasting protective effects.

Язык: Английский

Процитировано

0

Ultrasound-mediated nanomaterials for the treatment of inflammatory diseases DOI Creative Commons

Kai Zhang,

Tingting Wang, Xingyong Huang

и другие.

Ultrasonics Sonochemistry, Год журнала: 2025, Номер 114, С. 107270 - 107270

Опубликована: Фев. 13, 2025

Sterile and infection-associated inflammatory diseases are becoming increasingly prevalent worldwide. Conventional drug therapies often entail significant drawbacks, such as the risk of overdose, development resistance in pathogens, systemic adverse reactions, all which can undermine effectiveness treatments for these conditions. Nanomaterials (NMs) have emerged a promising tool treatment due to their precise targeting capabilities, tunable characteristics, responsiveness external stimuli. Ultrasound (US), non-invasive effective method, has been explored combination with NMs achieve enhanced therapeutic outcomes. This review provides comprehensive overview recent advances use US-mediated treating diseases. A introduction application classification US was first presented, emphasizing advantages mechanisms through interact enhance anti-inflammatory therapy. Subsequently, specific applications sterile inflammation were summarized. Finally, challenges prospects clinical translation discussed, along an outline future research directions. aims provide insights guide improvement more interventions

Язык: Английский

Процитировано

0

Non-central symmetric 2D bismuth‐based perovskites for piezoelectric-enhanced sonodynamic immunotherapy DOI

Longji Zhu,

Qian Wang, Junhui Du

и другие.

Journal of Colloid and Interface Science, Год журнала: 2025, Номер 687, С. 386 - 401

Опубликована: Фев. 13, 2025

Язык: Английский

Процитировано

0

Ruthenium-doped carbon dots with “three-in-one” chemodynamic, photodynamic, and photothermal activity induce panoptosis for tumor therapy DOI

Xiaokang Lu,

Weijie Sun,

Xingye Zheng

и другие.

Chemical Engineering Journal, Год журнала: 2025, Номер unknown, С. 161355 - 161355

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

Unravelling the Role of PANoptosis in Liver Diseases: Mechanisms and Therapeutic Implications DOI Open Access
Wanyuan Xiong, Junfeng Li, Aiping Tian

и другие.

Liver International, Год журнала: 2025, Номер 45(4)

Опубликована: Март 21, 2025

PANoptosis is a multimodal form of cell death that involves inflammatory, apoptotic, and necroptotic pathways, playing key role in the development liver diseases. This article first outlines definition characteristics PANoptosis, then explores its mechanisms action different types diseases, including acute injury, failure, metabolic dysfunction-associated fatty disease, hepatocellular carcinoma. Furthermore, this analyses molecular regulatory network potential therapeutic targets. Finally, summarises current research on diseases future directions, it reviews emerging mechanism

Язык: Английский

Процитировано

0

Polydopamine-Encapsulated Cyanine Dye Nanoplatform for Near-Infrared Fluorescence Imaging-Guided Photothermal Therapy of Hepatocellular Carcinoma DOI

Ping Hu,

Xu Hou, Xiali Wang

и другие.

ACS Applied Polymer Materials, Год журнала: 2025, Номер unknown

Опубликована: Апрель 10, 2025

Язык: Английский

Процитировано

0

Nanomaterials in the diagnosis and treatment of gastrointestinal tumors: New clinical choices and treatment strategies DOI Creative Commons
Liping Chen,

Qingqing Li

Materials Today Bio, Год журнала: 2025, Номер unknown, С. 101782 - 101782

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

Nanomaterials‐Induced PANoptosis: A Promising Anti‐Tumor Strategy DOI

Guanghui Hou,

Youdong Chen,

Huali Lei

и другие.

Angewandte Chemie International Edition, Год журнала: 2024, Номер unknown

Опубликована: Ноя. 19, 2024

Abstract Malignant tumors pose a significant threat to global public health. Promoting programmed cell death in cancer cells has become critical strategy for treatment. PANoptosis, newly discovered form of regulated death, integrates key molecular components pyroptosis, apoptosis, and necroptosis, activating these three pathways simultaneously achieve synergistic multi‐mechanistic killing. PANoptosis significantly inhibits growth resistance activates strong anti‐tumor immune response, making tumor‐specific induction potential therapeutic strategy. Currently, treatment research related is focused mainly on the development small molecules cytokines. However, approaches still face limitations terms metabolic stability tumor specificity. The unique physicochemical properties biological activities nanomaterials hold promise optimizing strategies. This review summarizes concept mechanisms highlights latest applications nanoagents PANoptosis‐based anti‐cancer therapy, discusses challenges future directions clinical translation. It hoped that this will inspire further exploration treatments, providing new perspectives researchers field.

Язык: Английский

Процитировано

1

Nanomaterials‐Induced PANoptosis: A Promising Anti‐Tumor Strategy DOI Open Access

Guanghui Hou,

Youdong Chen,

Huali Lei

и другие.

Angewandte Chemie, Год журнала: 2024, Номер unknown

Опубликована: Ноя. 19, 2024

Abstract Malignant tumors pose a significant threat to global public health. Promoting programmed cell death in cancer cells has become critical strategy for treatment. PANoptosis, newly discovered form of regulated death, integrates key molecular components pyroptosis, apoptosis, and necroptosis, activating these three pathways simultaneously achieve synergistic multi‐mechanistic killing. PANoptosis significantly inhibits growth resistance activates strong anti‐tumor immune response, making tumor‐specific induction potential therapeutic strategy. Currently, treatment research related is focused mainly on the development small molecules cytokines. However, approaches still face limitations terms metabolic stability tumor specificity. The unique physicochemical properties biological activities nanomaterials hold promise optimizing strategies. This review summarizes concept mechanisms highlights latest applications nanoagents PANoptosis‐based anti‐cancer therapy, discusses challenges future directions clinical translation. It hoped that this will inspire further exploration treatments, providing new perspectives researchers field.

Язык: Английский

Процитировано

0

Synergistic effects of repeated transcranial magnetic stimulation and mesenchymal stem cells transplantation on alleviating neuroinflammation and PANoptosis in cerebral ischemia DOI Creative Commons

Shimei Cheng,

Qiying Lu,

Qiuli Liu

и другие.

Journal of Neuroinflammation, Год журнала: 2024, Номер 21(1)

Опубликована: Ноя. 30, 2024

Neuronal death is the primary cause of poor outcomes in cerebral ischemia. The inflammatory infiltration early phase ischemic stroke plays a vital role triggering neuronal death. Either transplantation mesenchymal stem cells (MSCs) derived from humans or repetitive transcranial magnetic stimulation (rTMS) have respectively proved to be neuroprotective and anti-inflammatory However, either treatment above has its limitations. Whether these two therapies synergistic effects on improving neurological function underlying mechanisms remains unclear. This investigation aims elucidate MSCs combined with rTMS recovery post-ischemia. A Sprague-Dawley rat model infarction was induced via transient middle artery occlusion (tMCAO). rats were divided into five groups (n = 50): sham, tMCAO, rTMS, MSCs, + groups. Transplantation human umbilical cord intervention performed 24 h post-stroke. Neurological further assessed several behavioral tests 2,3,5-triphenyltetrazolium chloride (TTC) staining companied Nissl used assess survival. TUNEL staining, western blotting, immunofluorescence, immunohistochemistry, ELISA, flow cytometry employed measure levels neuroinflammation PANoptosis. molecular special therapy distinguished transcriptome sequencing PC12 oxygen-glucose deprivation/reoxygenation (OGD/R) conditions. efficiently reduced lesion volume improved survival (P < 0.05), subsequently functional after stroke. ameliorated PANoptosis neurons accompanied by decreased factors tissue serum during subacute infarction. To explore roles effect, we found that transplanted primarily localized spleen ischemia suppressed splenic inflammation. Meanwhile, significantly protects MSCs-inhibited conditions downregulating REST unveiled sequencing. Our study elucidates an unidentified mechanism which combination could synergistically promote suppress infarction, thus outcomes. may potentially contribute effect against These results are expected provide novel insights protecting injury potential targets

Язык: Английский

Процитировано

0