The cardio-oncologic burden of breast cancer: molecular mechanisms and importance of preclinical models DOI Creative Commons
Jürgen Bräuer,

M. Tumani,

Norbert Frey

и другие.

Basic Research in Cardiology, Год журнала: 2024, Номер unknown

Опубликована: Дек. 2, 2024

Abstract Breast cancer, the most prevalent cancer affecting women worldwide, poses a significant cardio-oncological burden. Despite advancements in novel therapeutic strategies, anthracyclines, HER2 antagonists, and radiation remain cornerstones of oncological treatment. However, each carries risk cardiotoxicity, though molecular mechanisms underlying these adverse effects differ. Common include DNA damage response, increased reactive oxygen species, mitochondrial dysfunction, which are key areas ongoing research for potential cardioprotective strategies. Since also essential effective tumor cytotoxicity, we explore tumor-specific effects, particularly hereditary breast linked to BRCA1 BRCA2 mutations. These genetic variants impair repair mechanisms, increase tumorigenesis possibly cardiotoxicity from treatments such as anthracyclines antagonists. Novel therapies, including immune checkpoint inhibitors, used clinic triple-negative improve outcomes patients. This review discusses BRCA dysfunction associated pathological pathways. It gives an overview preclinical models genetically engineered mouse models, syngeneic murine humanized various vitro ex vivo systems study cardiovascular side therapies. Understanding mechanism developing strategies improving treatment reducing long-term risks

Язык: Английский

Immuno-related cardio-vascular adverse events associated with immuno-oncological treatments: an under-estimated threat for cancer patients DOI Creative Commons
Giuseppe Panuccio, Pierpaolo Correale, Maria D’Apolito

и другие.

Basic Research in Cardiology, Год журнала: 2024, Номер unknown

Опубликована: Сен. 3, 2024

Abstract Immunotherapy represents an emergent and heterogeneous group of anticancer treatments harnessing the human immune-surveillance system, including immune-checkpoint inhibitor monoclonal antibodies (mAbs), Chimeric Antigen Receptor T Cells (CAR-T) therapy, cancer vaccines lymphocyte activation gene-3 (LAG-3) therapy. While remarkably effective against several malignancies, these therapies, often in combination with other treatments, have showed unforeseen toxicity, cardiovascular complications. The occurrence immuno-mediated adverse (irAEs) events has been progressively reported last 10 years. These irAEs present extended range severity, from self-limiting to life-threatening conditions. Although recent guidelines CardioOncology provided important evidence managing they encompass general approaches. However, a specific focus is required due particular etiology, unique risk factors, associated side effects immunotherapy. This review aims deepen understanding prevalence nature issues patients undergoing immunotherapy, offering insights into strategies for stratification management.

Язык: Английский

Процитировано

7

Cardio-oncology: chances and challenges DOI
Adriana E. Viñas-Mendieta,

A. Gallardo-Grajeda,

Teresa López‐Fernández

и другие.

Basic Research in Cardiology, Год журнала: 2024, Номер unknown

Опубликована: Сен. 30, 2024

Язык: Английский

Процитировано

6

Baicalin reduced vandetanib induced myocardial injury by regulating redox balance and NLRP3 inflammasome pathway DOI
Fen Wang, Jianwei Li, Zhixuan Zhang

и другие.

Tissue and Cell, Год журнала: 2025, Номер 94, С. 102795 - 102795

Опубликована: Фев. 16, 2025

Язык: Английский

Процитировано

0

Autoimmune-related adverse events induced by immune checkpoint inhibitors DOI
Yini Sun, Ziyang Zhang, Jia Ke

и другие.

Current Opinion in Immunology, Год журнала: 2025, Номер 94, С. 102556 - 102556

Опубликована: Апрель 11, 2025

Язык: Английский

Процитировано

0

The cardio-oncologic burden of breast cancer: molecular mechanisms and importance of preclinical models DOI Creative Commons
Jürgen Bräuer,

M. Tumani,

Norbert Frey

и другие.

Basic Research in Cardiology, Год журнала: 2024, Номер unknown

Опубликована: Дек. 2, 2024

Abstract Breast cancer, the most prevalent cancer affecting women worldwide, poses a significant cardio-oncological burden. Despite advancements in novel therapeutic strategies, anthracyclines, HER2 antagonists, and radiation remain cornerstones of oncological treatment. However, each carries risk cardiotoxicity, though molecular mechanisms underlying these adverse effects differ. Common include DNA damage response, increased reactive oxygen species, mitochondrial dysfunction, which are key areas ongoing research for potential cardioprotective strategies. Since also essential effective tumor cytotoxicity, we explore tumor-specific effects, particularly hereditary breast linked to BRCA1 BRCA2 mutations. These genetic variants impair repair mechanisms, increase tumorigenesis possibly cardiotoxicity from treatments such as anthracyclines antagonists. Novel therapies, including immune checkpoint inhibitors, used clinic triple-negative improve outcomes patients. This review discusses BRCA dysfunction associated pathological pathways. It gives an overview preclinical models genetically engineered mouse models, syngeneic murine humanized various vitro ex vivo systems study cardiovascular side therapies. Understanding mechanism developing strategies improving treatment reducing long-term risks

Язык: Английский

Процитировано

1