Decoding the complex web: Cellular and molecular interactions in the lung tumor microenvironment DOI

Bonnie Issa,

Ayat Hussein Adhab,

Morug Salih Mahdi

и другие.

Journal of drug targeting, Год журнала: 2024, Номер unknown, С. 1 - 44

Опубликована: Дек. 21, 2024

The lung tumor microenvironment (TME) or stroma is a dynamic space of numerous cells and their released molecules. This complicated web regulates progression resistance to different modalities. Lung cancer in conjunction with liberate wide range factors that dampen antitumor attacks by innate immunity like natural killer (NK) also adaptive responses effector T cells. These include growth factors, exosomes epigenetic regulators, anti-inflammatory cytokines. Understanding the intricate interactions between various elements within TME, such as immune stromal can help provide novel strategies for better management treatment malignancies. current article discusses complex network signaling molecules, which mediate communications TME. By elucidating these multifaceted interactions, we aim insights into potential therapeutic targets treatment.

Язык: Английский

Identification of cancer cell-intrinsic biomarkers associated with tumor progression and characterization of SFTA3 as a tumor suppressor in lung adenocarcinomas DOI Creative Commons
Yu Zhao, Chengcheng Zhou, Ling Zuo

и другие.

BMC Cancer, Год журнала: 2025, Номер 25(1)

Опубликована: Янв. 8, 2025

Recent advancements in contemporary therapeutic approaches have increased the survival rates of lung cancer patients; however, long-term benefits remain constrained, underscoring pressing need for novel biomarkers. Surfactant-associated 3 (SFTA3), a long non-coding RNA predominantly expressed normal epithelial cells, plays crucial role development. Nevertheless, its function adenocarcinoma (LUAD) remains inadequately understood. Single-cell sequencing data were utilized to identify cell-intrinsic gene signatures associated with progression LUAD, and their roles LUAD comprehensively analyzed. Serum samples collected quantify expression levels SFTA3 patients. Furthermore, series biological experiments, including cell viability assays, scratch wound healing colony formation conducted demonstrate tumor-suppressive effects SFTA3. was performed elucidate molecular mechanisms underlying cells. We constructed prognostic model comprising eight genes: ALDOA, ATP5MD, SERPINH1, SFTA3, SLK, U2SURP, SCGB1A1, SCGB1A3. The effectively stratified patients into high- low-risk categories, revealing that experienced superior clinical outcomes, exhibited an immunologically hot tumor microenvironment (TME), had greater probability responding immunotherapy. In contrast, high-risk group cold TME may benefit more from chemotherapy. our study revealed progressive decrease cells correlated advancement. Notably, serum significantly decreased suggesting potential utility liquid biopsy diagnosis. Additionally, knockdown enhances proliferation migration whereas overexpression inhibits these phenotypes. epithelial-mesenchymal transition pathway enriched following silencing, impact by modulating this process. also identified key transcription factors epigenetic implicated downregulation LUAD. developed robust as biomarker applications diagnosis, prognosis, personalized treatment findings offer new insights tumorigenesis immune evasion.

Язык: Английский

Процитировано

0

Redefining Hepatocellular Carcinoma Treatment: Nanotechnology Meets Tumor Immune Microenvironment DOI
Chen Mi, Sai Liu, Zhida Chen

и другие.

Journal of drug targeting, Год журнала: 2025, Номер unknown, С. 1 - 47

Опубликована: Март 13, 2025

Hepatocellular carcinoma (HCC) is one of the most lethal malignancies worldwide, characterized by its complex pathogenesis and poor therapeutic outcomes. Despite recent advances in targeted molecular therapies, immune checkpoint inhibitors (ICIs), radiotherapy, conventional chemotherapy, five-year survival rate for this neoplasm remains dismally low. The progress nanotechnology has revolutionized cancer treatment years. These provide unprecedented opportunities to overcome current limitations different modalities. This review provides a comprehensive analysis how interfaces with tumor microenvironment (TIME) HCC can present new frontier interventions HCC. We critically overview latest developments nanoparticle-based delivery systems various drugs also other antitumor agents like thermal therapy radiotherapy. highlight unique ability nanoparticles modulate immunosuppressive (TME) enhance efficacy. Furthermore, we analyze emerging strategies that exploit nanoformulations biological barriers drug bioavailability treatment.

Язык: Английский

Процитировано

0

Targeting the lung tumor stroma: harnessing nanoparticles for effective therapeutic interventions DOI

Shushu Zhang,

Hui Wang

Journal of drug targeting, Год журнала: 2024, Номер 33(1), С. 60 - 86

Опубликована: Окт. 2, 2024

Lung cancer remains an influential global health concern, necessitating the development of innovative therapeutic strategies. The tumour stroma, which is known as microenvironment (TME) has a central impact on expansion and treatment resistance. stroma lung tumours consists numerous cells molecules that shape environment for expansion. This not only protects tumoral against immune system attacks but also enables to attenuate action antitumor drugs. stromal like cancer-associated fibroblasts (CAFs), suppressive cells, cytotoxic cells. Additionally, presence stem endothelial pericytes can facilitate volume Nanoparticles are hopeful tools targeted drug delivery because their extraordinary properties capacity devastate biological obstacles. review article provides comprehensive overview contemporary advancements in targeting using nanoparticles. Various nanoparticle-based approaches, including passive active targeting, stimuli-responsive systems, highlighting potential improve efficiency. role nanotechnology modulating by key components such extracellular matrix (ECM), hypoxia, elements stroma.

Язык: Английский

Процитировано

1

Decoding the complex web: Cellular and molecular interactions in the lung tumor microenvironment DOI

Bonnie Issa,

Ayat Hussein Adhab,

Morug Salih Mahdi

и другие.

Journal of drug targeting, Год журнала: 2024, Номер unknown, С. 1 - 44

Опубликована: Дек. 21, 2024

The lung tumor microenvironment (TME) or stroma is a dynamic space of numerous cells and their released molecules. This complicated web regulates progression resistance to different modalities. Lung cancer in conjunction with liberate wide range factors that dampen antitumor attacks by innate immunity like natural killer (NK) also adaptive responses effector T cells. These include growth factors, exosomes epigenetic regulators, anti-inflammatory cytokines. Understanding the intricate interactions between various elements within TME, such as immune stromal can help provide novel strategies for better management treatment malignancies. current article discusses complex network signaling molecules, which mediate communications TME. By elucidating these multifaceted interactions, we aim insights into potential therapeutic targets treatment.

Язык: Английский

Процитировано

1