Εosinophilic Chronic Obstructive Pulmonary Disease. What Do We Know So Far? DOI Creative Commons
Κonstantinos Bartziokas, Andriana Ι. Papaioannou, Νikoletta Ρovina

и другие.

Pulmonary Therapy, Год журнала: 2024, Номер 11(1), С. 7 - 24

Опубликована: Дек. 11, 2024

The latest advances in asthma treatment have highlighted the significance of eosinophilia and possible role some pro-eosinophilic mediators, like interleukins (IL) IL-5, IL-4/IL-13, IL-33 disease's pathogenesis. Considering that a subgroup patients with chronic obstructive pulmonary disease (COPD) may blood akin to seen asthma, numerous studies last decade suggested eosinophilic COPD is separate entity. While exact eosinophils pathophysiology remains unclear, seems increase effectiveness corticosteroid therapy. Currently, monoclonal antibodies targeting (IL-5, IL-4, IL-13, IL-33) or their receptors are being investigated belonging T2-high endotype. This review focuses on mechanisms COPD, effects outcome, examines most recent data use peripheral treating COPD. Finally, we emphasize current implication context airway inflammation.

Язык: Английский

Outcomes among patients with chronic obstructive pulmonary disease after recovery from COVID-19 infection of different severity DOI Creative Commons
Wang Chun Kwok,

Chi Hung Chau,

Terence Chi Chun Tam

и другие.

Scientific Reports, Год журнала: 2024, Номер 14(1)

Опубликована: Июнь 16, 2024

Abstract While studies have suggested increased risks of severe COVID-19 infection in chronic obstructive pulmonary disease (COPD), the persistent and delayed consequences on patients with COPD upon recovery remain unknown. A prospective clinical study was conducted Hong Kong to investigate outcomes who had different severity (mild-moderate COVID-19), compared those did not. Chinese ≥ 40 years old were recruited from March September 2021. They prospectively followed up for 24.9 ± 5.0 months until 31st August 2023. The primary outcome deterioration control defined as change mMRC dyspnea scale. secondary included exacerbation frequency non-COVID-19 respiratory mortality (including death or bacterial pneumonia). 328 analysis. Patients mild-moderate statistically significant worsening dyspnoea scale by increase 1 score baseline follow-up adjusted odds ratios 4.44 (95% CI = 1.95–10.15, p < 0.001) 6.77 2.08–22.00, respectively. significantly 4.73 1.55–14.41, 0.006) pneumonia hazard ratio 11.25 2.98–42.45, 0.001). After COVID-19, dyspnea, (COPD pneumonia) observed among COVID-19. Mild moderate also associated symptomatic deterioration.

Язык: Английский

Процитировано

3

DElaying disease Progression In COPD with early escalation to Triple therapy – A Modelling Study (DEPICT-2) DOI Creative Commons
Dave Singh,

Diego Litewka,

Joan B. Soriano

и другие.

ERJ Open Research, Год журнала: 2024, Номер unknown, С. 00438 - 2024

Опубликована: Авг. 22, 2024

In patients with COPD, dual bronchodilator (long-acting muscarinic antagonist (LAMA)/long-acting β2-agonist (LABA)) and triple therapy (inhaled corticosteroid/LAMA/LABA) reduce the risk of exacerbations lung function decline in short-mid-term, but their long-term impact is unknown. This modelling study explores these therapies on decline, quality life (QoL) all-cause mortality. approach used a longitudinal nonparametric superposition model using published data regarding exacerbations, QoL (assessed by St George's Respiratory Questionnaire (SGRQ)) The simulated disease progression from 40 to 75 years age assessed initiating at 45 ("LAMA/LABA only" group) escalation 50 ("Escalation triple" forced expiratory volume 1 s (FEV1) Model simulation predicted that age, "LAMA/LABA preserves 159.1 mL FEV1 versus no treatment, while "Escalation an additional 376.5 217.3 pharmacotherapy only", respectively. SGRQ score reduces (-3.2) which further -7.5 triple". mortality 5.4% shows decrease 12.0%. Early initiation could slow preserving improving survival COPD.

Язык: Английский

Процитировано

1

Increased Pneumonia Risk Associated with Concomitant Use of Inhaled Corticosteroids and Benzodiazepines: A Pharmacovigilance Analysis DOI
Junlong Ma, Yaxin Liu, Yuanyuan Sun

и другие.

Lung, Год журнала: 2024, Номер 202(5), С. 673 - 681

Опубликована: Авг. 27, 2024

Язык: Английский

Процитировано

1

Εosinophilic Chronic Obstructive Pulmonary Disease. What Do We Know So Far? DOI Creative Commons
Κonstantinos Bartziokas, Andriana Ι. Papaioannou, Νikoletta Ρovina

и другие.

Pulmonary Therapy, Год журнала: 2024, Номер 11(1), С. 7 - 24

Опубликована: Дек. 11, 2024

The latest advances in asthma treatment have highlighted the significance of eosinophilia and possible role some pro-eosinophilic mediators, like interleukins (IL) IL-5, IL-4/IL-13, IL-33 disease's pathogenesis. Considering that a subgroup patients with chronic obstructive pulmonary disease (COPD) may blood akin to seen asthma, numerous studies last decade suggested eosinophilic COPD is separate entity. While exact eosinophils pathophysiology remains unclear, seems increase effectiveness corticosteroid therapy. Currently, monoclonal antibodies targeting (IL-5, IL-4, IL-13, IL-33) or their receptors are being investigated belonging T2-high endotype. This review focuses on mechanisms COPD, effects outcome, examines most recent data use peripheral treating COPD. Finally, we emphasize current implication context airway inflammation.

Язык: Английский

Процитировано

0