Deleted Journal, Год журнала: 2025, Номер unknown

Опубликована: Фев. 24, 2025

Abstract Current scientific endeavours in the field of geroscience have begun to reveal how factors such as exercise could modulate brain aging process. In this review, we ask potentially aging, and by extension, development Alzheimer’s Disease (AD). Furthermore, discuss mitigate cascade debilitating manifestations AD. Mechanistically, biomolecules brain-derived neurotrophic factor (BDNF) its cognate receptor, tropomyosin receptor kinase B (TrkB) change during life course signalling pathways be altered with (acute sessions or chronic training). Tackling these questions help medical community age-related decline terms neurological functions.

Язык: Английский