SARS-CoV-2 Infection and Alpha-Synucleinopathies: Potential Links and Underlying Mechanisms DOI Open Access
Joanna Motyl, Grażyna Gromadzka, Grzegorz A. Czapski

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(22), С. 12079 - 12079

Опубликована: Ноя. 10, 2024

Alpha-synuclein (α-syn) is a 140-amino-acid, intrinsically disordered, soluble protein that abundantly present in the brain. It plays crucial role maintaining cellular structures and organelle functions, particularly supporting synaptic plasticity regulating neurotransmitter turnover. However, for reasons not yet fully understood, α-syn can lose its physiological begin to aggregate. This altered disrupts dopaminergic transmission causes both presynaptic postsynaptic dysfunction, ultimately leading cell death. A group of neurodegenerative diseases known as α-synucleinopathies characterized by intracellular accumulation deposits specific neuronal glial cells within certain brain regions. In addition Parkinson's disease (PD), these conditions include dementia with Lewy bodies (DLBs), multiple system atrophy (MSA), pure autonomic failure (PAF), REM sleep behavior disorder (RBD). Given disorders are associated α-syn-related neuroinflammation-and considering SARS-CoV-2 infection has been shown affect nervous system, COVID-19 patients experiencing neurological symptoms-it proposed may contribute neurodegeneration PD other promoting misfolding aggregation. this review, we focus on whether could act an environmental trigger facilitates onset or progression α-synucleinopathies. Specifically, new evidence potential modulating function discuss causal relationship between development parkinsonism-like symptoms.

Язык: Английский

Advances of the MAPK pathway in the treatment of spinal cord injury DOI Creative Commons

Shixue Huang,

Yinuo Zhang, Haoming Shu

и другие.

CNS Neuroscience & Therapeutics, Год журнала: 2024, Номер 30(6)

Опубликована: Июнь 1, 2024

Spinal cord injury (SCI) represents a complex pathology within the central nervous system (CNS), leading to severe sensory and motor impairments. It activates various signaling pathways, notably mitogen-activated protein kinase (MAPK) pathway. Present treatment approaches primarily focus on symptomatic relief, lacking efficacy in addressing underlying pathophysiological mechanisms. Emerging research underscores significance of MAPK pathway neuronal differentiation, growth, survival, axonal regeneration, inflammatory responses post-SCI. Modulating this post-injury has shown promise attenuating inflammation, minimizing apoptosis, alleviating neuropathic pain, fostering neural regeneration. Given its pivotal role, emerges as potential therapeutic target SCI management. This review synthesizes current knowledge pathology, delineates pathway's characteristics, explores dual roles interventions. Furthermore, it addresses existing challenges context SCI, proposing solutions overcome these hurdles. Our aim is offer comprehensive reference for future laying groundwork targeted strategies.

Язык: Английский

Процитировано

10

A Comprehensive Review of the Global Epidemiology, Clinical Management, Socio-Economic Impacts, and National Responses to Long COVID with Future Research Directions DOI Creative Commons

Xiufang Song,

Weiwei Song, Lizhen Cui

и другие.

Diagnostics, Год журнала: 2024, Номер 14(11), С. 1168 - 1168

Опубликована: Май 31, 2024

Long COVID, characterized by a persistent symptom spectrum following SARS-CoV-2 infection, poses significant health, social, and economic challenges. This review aims to consolidate knowledge on its epidemiology, clinical features, underlying mechanisms guide global responses;

Язык: Английский

Процитировано

3

Loganic Acid Alleviates the Olfactory-Brain NLRP3 Inflammasome Activation and Rescues Dopaminergic Neurons in Experimental Models of Parkinson’s Disease DOI
Samir Ranjan Panda,

Pallabi Panja,

Meenakshi Singh

и другие.

Journal of Neuroimmune Pharmacology, Год журнала: 2025, Номер 20(1)

Опубликована: Фев. 12, 2025

Язык: Английский

Процитировано

0

Neurodegenerative diseases and neuroinflammation-induced apoptosis DOI Creative Commons

Shi Huang,

Yaxin Lu,

Wenjun Fang

и другие.

Open Life Sciences, Год журнала: 2025, Номер 20(1)

Опубликована: Янв. 1, 2025

Abstract Neuroinflammation represents a critical pathway in the brain for clearance of foreign bodies and maintenance homeostasis. When neuroinflammatory process is dysregulate, such as over-activation microglia, which results excessive accumulation free oxygen inflammatory factors brain, among other factors, it can lead to an imbalance homeostasis development various diseases. Recent research has indicated that numerous neurodegenerative diseases closely associated with neuroinflammation. The pathogenesis neuroinflammation intricate, involving alterations genes proteins, well activation inhibition signaling pathways. Furthermore, inflammation result neuronal cell apoptosis, further exacerbate extent disease. This article presents summary recent studies on relationship between apoptosis caused by aim identify link two provide new ideas targets exploring pathogenesis, prevention treatment

Язык: Английский

Процитировано

0

OLFACTORY DYSFUNCTION: AN EARLY INDICATOR OF PARKINSON'S DISEASE DOI Creative Commons
Dhuly Chowdhury, Ashmita Das,

Mrityunjay Mishra

и другие.

Brain Disorders, Год журнала: 2025, Номер unknown, С. 100209 - 100209

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

Integrated Analysis of Single-Cell and Transcriptome Data Reveals the Role and Regulatory Mechanisms of Neuroinflammation in Parkinson's Disease DOI Creative Commons

Yao Geng,

Ruiyu Wang,

Manyu Dong

и другие.

Inflammation, Год журнала: 2025, Номер unknown

Опубликована: Апрель 26, 2025

There is increasing interest in developing therapeutic interventions aimed at preventing neuroinflammation Parkinson's disease (PD). However, the specific characteristics of inflammation across different cell types and underlying mechanisms PD-related remain inadequately understood. In this study, we conducted an analysis single-cell RNA sequencing (scRNA-seq) microarray data derived from human PD midbrain tissue, specifically focusing on substantia nigra compacta (SNc). These datasets were sourced (GEO) database. We utilized GSVA, GSEA, as well KEGG GO analyses to explore transcriptional variations associated with PD. Furthermore, trajectory SCENIC uncover progression. Subsequent animal cellular experiments validated role regulon regulating neuroinflammation. Results: Our revealed that microglia displayed highest levels inflammatory activity, characterized by increased abundance proinflammatory activated state within SNc patients. This finding was further a mouse model induced 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP). The transcription factor STAT3 demonstrated significant upregulation implicated promoting response activating context. 1-methyl-4-phenylpyridine (MPP +)-induced BV2 model, inhibition led reduced inflammation, hindered phosphorylation, decreased production factors. downregulation P-STAT3 alleviated harmful effects SH-SY5Y cells cocultured conditioned medium. Conclusions: study underscored pivotal central regulator activation findings offer fresh insights into pathogenesis suggest potential avenues for development novel strategies.

Язык: Английский

Процитировано

0

IL‐6 and Olfactory Dysfunction: Focus on Changes, Effects, and Mechanisms DOI Creative Commons
Xiao-Yu Song, Yakui Mou, Hanrui Wang

и другие.

Mediators of Inflammation, Год журнала: 2025, Номер 2025(1)

Опубликована: Янв. 1, 2025

The sense of smell is vital for human life and risk identification. Many diseases can cause olfactory disorders, early identification intervention disorders are crucial. Currently, the diagnosis in clinical practice mostly relies on subjective visual analog scale (VAS) evaluations, expensive complex imaging, neurophysiological examinations, which lead to poor patient compliance low completion rates. Therefore, there an urgent need identify novel, objective, easily detectable biological indicators. Interleukin‐6 (IL‐6) inflammatory factor that closely associated with dysfunction various diseases. However, role IL‐6 occurrence development not yet clear, limits its application. This article reviews changes possible mechanisms aim providing a reference application as biomarker promoting in‐depth exploration mechanism disorders.

Язык: Английский

Процитировано

0

A Comprehensive Global Review of Epidemiology, Clinical Management, Socio-Economic Impacts and National Responses of Long COVID with Future Research Directions DOI Open Access

Xiufang Song,

Weiwei Song, Lizhen Cui

и другие.

Опубликована: Апрель 26, 2024

Background: Long COVID, characterized by a persistent symptom spectrum following SARS-CoV-2 infection, poses significant health, social, and economic challenges. This review aims to consolidate knowledge on its epidemiology, clinical features, underlying mechanisms guide global responses; Methods: We conducted systematic literature review, analyzing peer-reviewed articles reports gather comprehensive data long COVID’s symptomatology, management approaches; Results: Our analysis revealed wide array of COVID symptoms risk factors, with notable demographic variability. The current understanding pathophysiology suggests multifactorial origin yet remains partially understood. Emerging diagnostic criteria potential therapeutic strategies were identified, highlighting advancements in management.; Conclusion: highlights the multifaceted nature revealing broad symptoms, diverse complex interplay physiological underpinning condition. disorders will continue weigh healthcare systems years come. Ad-dressing requires holistic strategy that integrates care, social support, policy initiatives. findings underscore need for increased international cooperation research health planning address challenges COVID. There is call continued refinement treatment modalities, emphasizing multidisciplinary approach manage ongoing evolving impacts

Язык: Английский

Процитировано

2

SARS-CoV-2 Infection and Alpha-Synucleinopathies: Potential Links and Underlying Mechanisms DOI Open Access
Joanna Motyl, Grażyna Gromadzka, Grzegorz A. Czapski

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(22), С. 12079 - 12079

Опубликована: Ноя. 10, 2024

Alpha-synuclein (α-syn) is a 140-amino-acid, intrinsically disordered, soluble protein that abundantly present in the brain. It plays crucial role maintaining cellular structures and organelle functions, particularly supporting synaptic plasticity regulating neurotransmitter turnover. However, for reasons not yet fully understood, α-syn can lose its physiological begin to aggregate. This altered disrupts dopaminergic transmission causes both presynaptic postsynaptic dysfunction, ultimately leading cell death. A group of neurodegenerative diseases known as α-synucleinopathies characterized by intracellular accumulation deposits specific neuronal glial cells within certain brain regions. In addition Parkinson's disease (PD), these conditions include dementia with Lewy bodies (DLBs), multiple system atrophy (MSA), pure autonomic failure (PAF), REM sleep behavior disorder (RBD). Given disorders are associated α-syn-related neuroinflammation-and considering SARS-CoV-2 infection has been shown affect nervous system, COVID-19 patients experiencing neurological symptoms-it proposed may contribute neurodegeneration PD other promoting misfolding aggregation. this review, we focus on whether could act an environmental trigger facilitates onset or progression α-synucleinopathies. Specifically, new evidence potential modulating function discuss causal relationship between development parkinsonism-like symptoms.

Язык: Английский

Процитировано

1