The DNA damage response and RNA Polymerase II regulator Def1 has posttranscriptional functions in the cytoplasm DOI

Oluwasegun T. Akinniyi,

Smita Kulkarni,

M.M. Hribal

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Сен. 16, 2024

Abstract Yeast Def1 mediates RNA polymerase II degradation and transcription elongation during stress. is predominantly cytoplasmic, DNA damage signals cause its proteolytic processing, liberating N-terminus to enter the nucleus. Cytoplasmic functions for this abundant protein have not been identified. Proximity-labeling (BioID) experiments indicate that binds an array of proteins involved in posttranscriptional control translation mRNAs. Deleting DEF1 reduces both mRNA synthesis decay rates, indicating transcript buffering mutant. Directly tethering a reporter suppressed expression, suggesting directly regulates Surprisingly, we found interacts with polyribosomes, which requires ubiquitin-binding domain located N-terminus. The binding ribosomes ubiquitylation eS7a (Rsp7A) small subunit by Not4 Ccr4-Not complex. ribosome quality co-translational decay. polyglutamine-rich unstructured C-terminus required interaction factors, acts as ubiquitin-dependent scaffold link status Thus, identified novel function response factor regulation cytoplasm establish master regulator gene functioning transcription, decay, translation.

Язык: Английский

RNA Polymerase II Activity Control of Gene Expression and Involvement in Disease DOI Creative Commons

James C. Kuldell,

Craig D. Kaplan

Journal of Molecular Biology, Год журнала: 2024, Номер 437(1), С. 168770 - 168770

Опубликована: Авг. 28, 2024

Gene expression is dependent on RNA Polymerase II (Pol II) activity in eukaryotes. In addition to determining the rate of synthesis for all protein coding genes, Pol serves as a platform recruitment factors and regulation co-transcriptional events, from processing chromatin modification remodeling. The transcriptome can be shaped by changes kinetics affecting itself or because alterations events that are responsive coupled with transcription. Genetic, biochemical, structural approaches model organisms have revealed critical insights into how works types regulate it. complexity generally increases organismal complexity. this review, we describe fundamental aspects shape gene expression, discuss recent advances elongation regulated altered function linked human disease aging.

Язык: Английский

Процитировано

4

Next-Generation Mapping of the ACINUS-Mediated Alternative Splicing Machinery and Its Regulation by O-glycosylation inArabidopsis DOI Creative Commons
Ruben Shrestha, Andres V. Reyes, Shane Carey

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2025, Номер unknown

Опубликована: Янв. 4, 2025

Alternative splicing (AS) is a key mechanism of gene regulation, but the full repertoire proteins involved and regulatory mechanisms governing this process remain poorly understood. Using TurboID-based proximity labeling coupled with mass spectrometry (PL-MS), we comprehensively mapped Arabidopsis AS machinery, focusing on evolutionarily conserved factor ACINUS, its paralog PININ, stable interactor SR45. We identified 298 high-confidence components, including both established novel interactors, providing strong evidence that alternative to transcription multiple RNA processing steps occur simultaneously in plants. Bioinformatic analysis reveals high redundancy, mechanisms, unique plant-specific features. Selected known interactors were validated by readouts phenotypic analysis, which also revealed coordinated influence splicing. Furthermore, systematic evaluation O-glycosylation double mutants SECRET AGENT (O-GlcNAc transferase) SPINDLY (O-fucose modulate through ACINUS-dependent -independent pathways. Our results reveal as well network highlight global role sugar modification processing.

Язык: Английский

Процитировано

0

PAF1C-mediated activation of CDK12/13 kinase activity is critical for CTD phosphorylation and transcript elongation DOI Creative Commons
David López Martínez, Izabela Todorovski, Melvin Noé González

и другие.

Molecular Cell, Год журнала: 2025, Номер unknown

Опубликована: Май 1, 2025

The transcription cycle is regulated by dynamic changes in RNA polymerase II (RNAPII) C-terminal domain (CTD) phosphorylation, which are crucial for gene expression. However, the mechanisms regulating transcription-specific cyclin-dependent kinases (CDKs) during remain poorly understood. Here, we show that human CDK12 co-phosphorylates CTD Serine2 and Serine5. This di-phosphorylated Serine2-Serine5 mark may then act as a precursor mono-phosphorylated through Serine5 de-phosphorylation. Notably, specifically association with elongation-specific factor PAF1 complex (PAF1C), CDC73 subunit contains metazoan-specific peptide motif, capable of allosteric CDK12/cyclin K activation. motif essential cell proliferation required normal levels phosphorylation chromatin, transcript elongation, particularly across long genes. Together, these findings provide insight into governing RNAPII phospho-CTD dynamics ensure progression cycle.

Язык: Английский

Процитировано

0

High-resolution sequencing reveals that the Paf1 complex may be a conserved transcription elongation factor for eukaryotic RNA polymerase I DOI

Abigail K. Huffines,

Naiheng J. Yang,

David A. Schneider

и другие.

Journal of Molecular Biology, Год журнала: 2025, Номер unknown, С. 169220 - 169220

Опубликована: Май 1, 2025

Язык: Английский

Процитировано

0

A direct interaction between the Chd1 CHCT domain and Rtf1 controls Chd1 distribution and nucleosome positioning on active genes DOI Creative Commons
Sarah A. Tripplehorn, Margaret K. Shirra, Santana M. Lardo

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Дек. 6, 2024

ABSTRACT The nucleosome remodeler Chd1 is required for the re-establishment of positioning in wake transcription elongation by RNA Polymerase II. Previously, we found that occupancy on gene bodies depends Rtf1 subunit Paf1 complex yeast. Here, identify an N-terminal region and CHCT domain as sufficient their interaction demonstrate this direct. Mutations disrupt Rtf1-Chd1 result accumulation at 5’ ends Chd1-occupied genes, increased cryptic transcription, altered positioning, concordant shifts histone modification profiles. We show a homologous within mouse RTF1 interacts with domains CHD1 CHD2. This work supports conserved mechanism coupling family proteins to identifies cellular function about which little known.

Язык: Английский

Процитировано

2

The DNA damage response and RNA Polymerase II regulator Def1 has posttranscriptional functions in the cytoplasm DOI

Oluwasegun T. Akinniyi,

Smita Kulkarni,

M.M. Hribal

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Сен. 16, 2024

Abstract Yeast Def1 mediates RNA polymerase II degradation and transcription elongation during stress. is predominantly cytoplasmic, DNA damage signals cause its proteolytic processing, liberating N-terminus to enter the nucleus. Cytoplasmic functions for this abundant protein have not been identified. Proximity-labeling (BioID) experiments indicate that binds an array of proteins involved in posttranscriptional control translation mRNAs. Deleting DEF1 reduces both mRNA synthesis decay rates, indicating transcript buffering mutant. Directly tethering a reporter suppressed expression, suggesting directly regulates Surprisingly, we found interacts with polyribosomes, which requires ubiquitin-binding domain located N-terminus. The binding ribosomes ubiquitylation eS7a (Rsp7A) small subunit by Not4 Ccr4-Not complex. ribosome quality co-translational decay. polyglutamine-rich unstructured C-terminus required interaction factors, acts as ubiquitin-dependent scaffold link status Thus, identified novel function response factor regulation cytoplasm establish master regulator gene functioning transcription, decay, translation.

Язык: Английский

Процитировано

0