Maternal immune activation and neuropsychiatric disease in offspring: Pathogen's perspective DOI Creative Commons

Zhiyang Yin,

Catherine Gordon,

Zikai Zhou

и другие.

Decoding Infection and Transmission, Год журнала: 2024, Номер 2, С. 100029 - 100029

Опубликована: Янв. 1, 2024

Язык: Английский

Maternal Immune-Mediated Conditions and ADHD Risk in Offspring DOI
Kjersti Mæhlum Walle, Kristin Gustavson, Siri Mjaaland

и другие.

Опубликована: Апрель 14, 2025

Abstract BACKGROUND Maternal immune-mediated conditions during pregnancy have been linked with increased risk of attention-deficit/hyperactivity disorder (ADHD) in offspring. However, we do not know the extent to which these associations are influenced by shared genetic predispositions, as opposed maternal inflammatory/immune responses pregnancy. This study contributes using paternal a negative control explore underlying factors, investigate between and offspring ADHD. METHODS Prospective data from Norwegian Mother, Father, Child Cohort Study (MoBa) was Medical Birth Registry Norway (MBRN) Patient (NPR) assess prenatal exposure ADHD up age 18. Nationwide recruitment 1999 2008 resulted 104,270 eligible mother-child pairs. Among these, 21,340 children were exposed allergic (asthma, allergies, atopic conditions) 7,478 other immune (autoimmune, inflammatory). Paternal self-reported served controls for confounding. Data mostly collected through MoBa, additional condition cases sourced MBRN, children’s diagnoses obtained NPR. Cox proportional hazard models estimated Hazard ratios diagnoses. RESULTS Both overall categories associated (allergic HR 1.23 95% CI, 1.14–1.34; 1.36 1.21–1.53). Specifically, found asthma (HR 1.47 1.30–1.67); allergies 1.20 1.10–1.31); rheumatologic/musculoskeletal 1.64 1.28–2.10), Crohn’s disease/ulcerative colitis (adjusted 1.95 1.23–3.09), endocrine 1.42 1.15–1.77), specifically, type 1 diabetes 2.50 1.66–3.75). Although some (psoriasis, ulcerative colitis, disease) showed similar trends offspring, only significantly 1.26 1.10–1.45). CONCLUSIONS Several Observations higher, more consistent estimates most versus ones indicate that unmeasured confounding does fully explain associations. These results suggest direct effects on fetal development events at maternal-fetal interface may alter potentially lead greater Asthma be possible exception this mechanism, also

Язык: Английский

Процитировано

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Maternal immune activation and neuropsychiatric disease in offspring: Pathogen's perspective DOI Creative Commons

Zhiyang Yin,

Catherine Gordon,

Zikai Zhou

и другие.

Decoding Infection and Transmission, Год журнала: 2024, Номер 2, С. 100029 - 100029

Опубликована: Янв. 1, 2024

Язык: Английский

Процитировано

0