Blueprints for healing: central nervous system regeneration in zebrafish and neonatal mice DOI Creative Commons
Brianna R. Cellini,

Sreeparvathy Vayankara Edachola,

Timothy D. Faw

и другие.

BMC Biology, Год журнала: 2025, Номер 23(1)

Опубликована: Апрель 30, 2025

In adult mammals, including humans, neurons, and axons in the brain spinal cord are inherently incapable of regenerating after injury. Studies animals with innate capacity for regeneration providing valuable insights into mechanisms driving tissue healing. The aim this review is to summarize recent data on zebrafish neonatal mice. We infer that elucidating these understanding how why they lost mammals will contribute development strategies promote central nervous system regeneration.

Язык: Английский

Hallmarks of regeneration DOI
Kenneth D. Poss, Elly M. Tanaka

Cell stem cell, Год журнала: 2024, Номер 31(9), С. 1244 - 1261

Опубликована: Авг. 19, 2024

Язык: Английский

Процитировано

10

Histone H4 lysine 20 monomethylation is not a mark of transcriptional silencers DOI Creative Commons

Julian A. Segert,

Martha L. Bulyk

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2025, Номер unknown

Опубликована: Янв. 13, 2025

Abstract Transcriptional silencers are cis -regulatory elements that downregulate the expression of target genes. Although thousands have been identified experimentally, a predictive chromatin signature has not found. H4K20me1 previously was reported to be highly enriched among human silencers, but our reanalysis those data using an appropriate background revealed enrichment is only marginal. We generated ChIP-seq profiles in Drosophila S2 cells, which similarly showed does mark and instead associated with active transcription. Silencers remain poorly annotated, difficult predict class whose specific features identified.

Язык: Английский

Процитировано

0

Common and specific gene regulatory programs in zebrafish caudal fin regeneration at single-cell resolution DOI Creative Commons
Yujie Chen, Yiran Hou, Qi Zeng

и другие.

Genome Research, Год журнала: 2025, Номер unknown

Опубликована: Янв. 14, 2025

Following amputation, zebrafish regenerate their injured caudal fin through lineage-restricted reprogramming. Although previous studies have charted various genetic and epigenetic dimensions of this process, the intricate gene regulatory programs shared by, or unique to, different regenerating cell types remain underinvestigated. Here, we mapped landscape regeneration by applying paired snRNA-seq snATAC-seq on uninjured fins. This map delineates dynamics predominant populations at multiple stages regeneration. We observe a marked increase in accessibility chromatin regions associated with regenerative developmental processes 1 dpa, followed gradual closure across major later stages. pattern is distinct from that transcriptomic dynamics, which characterized several waves upregulation downregulation. identified vivo validated cell-type-specific position-specific regeneration-responsive enhancers constructed networks type stage. Our single-cell resolution provides new insights into mechanisms serves as valuable resource for community.

Язык: Английский

Процитировано

0

Cardiac enhancers: Gateway to the regulatory mechanisms of heart regeneration DOI

Ian J. Begeman,

Megan E Guyer,

Junsu Kang

и другие.

Seminars in Cell and Developmental Biology, Год журнала: 2025, Номер 170, С. 103610 - 103610

Опубликована: Апрель 10, 2025

Язык: Английский

Процитировано

0

Blueprints for healing: central nervous system regeneration in zebrafish and neonatal mice DOI Creative Commons
Brianna R. Cellini,

Sreeparvathy Vayankara Edachola,

Timothy D. Faw

и другие.

BMC Biology, Год журнала: 2025, Номер 23(1)

Опубликована: Апрель 30, 2025

In adult mammals, including humans, neurons, and axons in the brain spinal cord are inherently incapable of regenerating after injury. Studies animals with innate capacity for regeneration providing valuable insights into mechanisms driving tissue healing. The aim this review is to summarize recent data on zebrafish neonatal mice. We infer that elucidating these understanding how why they lost mammals will contribute development strategies promote central nervous system regeneration.

Язык: Английский

Процитировано

0