Metabolomics reveals the metabolic disturbance caused by arsenic in the mouse model of inflammatory bowel disease DOI Creative Commons

Yanmei Wu,

Yin Wang, Yu‐Ning Lin

и другие.

Ecotoxicology and Environmental Safety, Год журнала: 2024, Номер 287, С. 117305 - 117305

Опубликована: Ноя. 1, 2024

Arsenic exposure has long been a significant global health concern due to its association with various human diseases. The adverse effects of arsenic can be influenced by multiple factors, resulting in considerable individual variability. Individuals inflammatory bowel disease (IBD) are particularly vulnerable the toxin exposure, yet specific impact context IBD remains unclear. In this study, we employed non-targeted metabolomics approach investigate how affects metabolic homeostasis an model using Helicobacter trogontum-infected interleukin-10 deficient mice. Our results demonstrated that disrupted balance metabolites, including tryptophan, polyunsaturated fatty acids, purine and pyrimidine branched-chain amino mice colitis but not those without colitis. Notably, several crucial metabolites involved anti-inflammatory responses, oxidative stress, energy metabolism were significantly altered These indicate lead extensive disturbances, potentially exacerbating severity impacting overall health. This study underscores necessity evaluating toxicity relation better understand mitigate associated risks.

Язык: Английский

Reverse engineering the Gut-Brain Axis and microbiome-metabolomics for symbiotic/pathogenic balance in neurodegenerative diseases DOI Creative Commons
Muhammad Usman Munir, Syed Aoun Ali, Ka Hang Karen Chung

и другие.

Gut Microbes, Год журнала: 2024, Номер 16(1)

Опубликована: Ноя. 10, 2024

Deciphering the molecular communications along gut-brain axis can help in understanding pathophysiology of neurodegenerative diseases and exploiting gut microbiome for therapeutics. However, microbes their metabolites have a multifaceted role mediating both brain physiology pathology. There is lack how when this tipped what are those contributing factors, at local (gut) distal (neuronal) levels, that drive imbalance. Here we reviewed its context summarized different factors such as gut-microbial diversity, metabolites, native immune system integrity epithelial blood-brain barriers interconnected collectively define involvement gut-microbiome pathologies. It also underlines need multidisciplinary tools animal models to simultaneously reflect on many these better correlate with clinical observations data obtained from human biopsies fecal samples. Harnessing will herald paradigm shift medicine aging, emphasizing significance broader spectrum health disease.

Язык: Английский

Процитировано

4

Exercise-induced adaptations in the kynurenine pathway: implications for health and disease management DOI Creative Commons
Marcus Vinícius dos Santos Rangel, Karynne Grutter Lopes, Xuebin Qin

и другие.

Frontiers in Sports and Active Living, Год журнала: 2025, Номер 7

Опубликована: Март 6, 2025

Background Tryptophan (TRP) metabolism through the kynurenine (KYN) pathway is influenced by inflammatory mediators, generating metabolites that regulate immune and responses. Exercise has been proposed as a modulator of this pathway, but its role in health benefits chronic disease management remains unclear. Objective This systematic review examines exercise-induced adaptations KYN their potential implications for management. Additionally, we identify key methodological considerations future research. Methods A structured search PubMed/Medline, Web Science, Scopus was conducted up to October 2024 clinical trials investigating effects exercise training on pathway. Results Of 2,795 articles initially found, 13 involving 592 participants met inclusion criteria. Most studies reported particularly cancer survivors. These appeared be intensity duration. However, several limitations were noted, no included patients with metabolic or cardiovascular diseases. Conclusions Here, show modulates both healthy diseased populations, highlighting prevention further randomized-controlled are needed clarify mechanisms applications, Systematic Review Registration https://www.crd.york.ac.uk/PROSPERO/view/CRD42022351481 , PROSPERO (CRD42022351481).

Язык: Английский

Процитировано

0

Unraveling lipopolysaccharide-induced behavioral and molecular effects in Lymnaea stagnalis, an emerging model organism for translational neuroscience DOI Creative Commons
Veronica Rivi, Giovanna Rigillo,

Silvia Alboni

и другие.

International Immunopharmacology, Год журнала: 2025, Номер 152, С. 114418 - 114418

Опубликована: Март 15, 2025

Язык: Английский

Процитировано

0

Physiological and pathophysiological effects of L-tryptophan’s metabolites on the brain and immunity – a challenge for drug development DOI Creative Commons
Petar Iliev,

Samuil Ivanov,

S Lazarov

и другие.

Pharmacia, Год журнала: 2025, Номер 72, С. 1 - 7

Опубликована: Март 19, 2025

The dysregulation of the kynurenine branch from tryptophan (TRP) metabolism can cause an imbalance between neuroprotective kynurenic acid and neurotoxic metabolites in some psychiatric neurodegenerative disorders. Therefore, modulation TRP may contribute to novel therapeutic strategies for several neuropsychiatric diseases. Targeting L-TRP-kynurenine pathway enzymes, particularly involving indoleamine 2,3-dioxygenase-1, 2,3-dioxygenase, 3-monooxygenase, aminotransferase II, is a promising approach. future development potent selective inhibitors these enzymes with good safety profile would be potential new avenue treatment many essential amino L-Tryptophan, together its metabolites, plays key role diverse physiological pathophysiological processes brain, especially disorders autoimmunity. Also, vital biochemical precursor functional neuroactive molecules, including serotonin melatonin, which are great importance memory learning, emotional regulation, circadian cycle, hunger, pain, etc. This article aims underscore links tryptophan’s certain diseases central nervous system.

Язык: Английский

Процитировано

0

Mitigating effect of dietary tryptophan on intra-cohort cannibalism in tiger puffer (Takifugu rubripes) with clipped or intact teeth DOI
Yuliang Wei, Zhibin Zhou, Liu Yang

и другие.

Aquaculture, Год журнала: 2025, Номер unknown, С. 742598 - 742598

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

Role of Kynurenine and Its Derivatives in the Neuroimmune System DOI Open Access
Makoto Fujikawa,

Masashi Ueda,

Kenta Maruyama

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(13), С. 7144 - 7144

Опубликована: Июнь 28, 2024

In recent years, there has been a growing realization of intricate interactions between the nervous and immune systems, characterized by shared humoral factors receptors. This interplay forms basis neuroimmune system, understanding which will provide insights into pathogenesis neurological diseases, in involvement system overlooked. Kynurenine its derivatives derived from tryptophan have long implicated various diseases. Recent studies revealed their close association not only with disorders but also sepsis-related deaths. review provides an overview biochemistry kynurenine derivatives, followed discussion role via modulation

Язык: Английский

Процитировано

3

Novel machine learning investigation for Buongiorno fluidic model with Sutterby nanomaterial DOI

Maryam Pervaiz Khan,

Chuan‐Yu Chang, Muhammad Asif Zahoor Raja

и другие.

Tribology International, Год журнала: 2024, Номер 199, С. 110009 - 110009

Опубликована: Июль 19, 2024

Язык: Английский

Процитировано

1

Metabolomics reveals the metabolic disturbance caused by arsenic in the mouse model of inflammatory bowel disease DOI Creative Commons

Yanmei Wu,

Yin Wang, Yu‐Ning Lin

и другие.

Ecotoxicology and Environmental Safety, Год журнала: 2024, Номер 287, С. 117305 - 117305

Опубликована: Ноя. 1, 2024

Arsenic exposure has long been a significant global health concern due to its association with various human diseases. The adverse effects of arsenic can be influenced by multiple factors, resulting in considerable individual variability. Individuals inflammatory bowel disease (IBD) are particularly vulnerable the toxin exposure, yet specific impact context IBD remains unclear. In this study, we employed non-targeted metabolomics approach investigate how affects metabolic homeostasis an model using Helicobacter trogontum-infected interleukin-10 deficient mice. Our results demonstrated that disrupted balance metabolites, including tryptophan, polyunsaturated fatty acids, purine and pyrimidine branched-chain amino mice colitis but not those without colitis. Notably, several crucial metabolites involved anti-inflammatory responses, oxidative stress, energy metabolism were significantly altered These indicate lead extensive disturbances, potentially exacerbating severity impacting overall health. This study underscores necessity evaluating toxicity relation better understand mitigate associated risks.

Язык: Английский

Процитировано

0