Mechanistic Insights into 3-Isopropylphenol-Induced Neurotoxicity in Zebrafish: A Network Toxicology and Molecular Docking Approach DOI Creative Commons
Jie Gu,

Huilin Jin,

Jun Hu

и другие.

Toxics, Год журнала: 2025, Номер 13(4), С. 274 - 274

Опубликована: Апрель 3, 2025

Endocrine-disrupting chemicals (EDCs) are exogenous substances discharged into the environment through human activities. 3-Isopropylphenol, a typical alkylphenol-based EDC, has been extensively studied due to its broad application and potential ecological impacts. However, mechanism of neurotoxicity remains unclear. In this study, neurotoxic effects 3-isopropylphenol were examined using zebrafish model. We predicted toxic mechanisms action targets network toxicology molecular docking verified them via RT-qPCR. Results showed that exposure inhibits cAMP/PKA signaling pathway in larvae, promoting apoptosis, impairing neural development, suppressing locomotor behavior. These findings enhance our understanding on larval development aid evaluating hazards.

Язык: Английский

Computational insights into the inhibitory effects of PFAS 14 on colorectal cancer targeting GSTA1 through competitive binding DOI Creative Commons
Jinxiao Li, Yanran Wu, Pian Ye

и другие.

Ecotoxicology and Environmental Safety, Год журнала: 2025, Номер 292, С. 117925 - 117925

Опубликована: Март 1, 2025

This study employed computational biology approaches to investigate the interactions between per- and polyfluoroalkyl substances (PFAS) key colorectal cancer (CRC) proteins. The results indicate that PFAS may influence CRC progression by modulating multiple proteins, particularly glutathione S-transferase A1 (GSTA1). Computational analysis revealed 14 exhibits high binding affinity for GSTA1, occupying its glutathione-binding site. Further simulations confirmed stable of across different environments, forming persistent hydrogen bonds water bridges, suggesting a potential inhibitory effect on GSTA1.GSTA1, member family, plays critical role in detoxification catalyzing conjugation electrophilic compounds. Dysregulation GSTA1 has been implicated chemoresistance. In CRC, altered expression affect tumor metabolism drug response, making it therapeutic target.This identifies as target interactions, environmental exposure interfering with mechanisms. competitive inhibition impact cell survival progression. Future research should integrate experimental validation assess phenotypic effects evaluate inhibitor.

Язык: Английский

Процитировано

0

Mechanistic Insights into 3-Isopropylphenol-Induced Neurotoxicity in Zebrafish: A Network Toxicology and Molecular Docking Approach DOI Creative Commons
Jie Gu,

Huilin Jin,

Jun Hu

и другие.

Toxics, Год журнала: 2025, Номер 13(4), С. 274 - 274

Опубликована: Апрель 3, 2025

Endocrine-disrupting chemicals (EDCs) are exogenous substances discharged into the environment through human activities. 3-Isopropylphenol, a typical alkylphenol-based EDC, has been extensively studied due to its broad application and potential ecological impacts. However, mechanism of neurotoxicity remains unclear. In this study, neurotoxic effects 3-isopropylphenol were examined using zebrafish model. We predicted toxic mechanisms action targets network toxicology molecular docking verified them via RT-qPCR. Results showed that exposure inhibits cAMP/PKA signaling pathway in larvae, promoting apoptosis, impairing neural development, suppressing locomotor behavior. These findings enhance our understanding on larval development aid evaluating hazards.

Язык: Английский

Процитировано

0