Construction of a novel five programmed cell death-related gene signature as a promising prognostic model for triple negative breast cancer DOI Creative Commons
Qing Shao, Haiyan Gao, Ziying Wang

и другие.

PeerJ, Год журнала: 2025, Номер 13, С. e19359 - e19359

Опубликована: Апрель 28, 2025

Triple negative breast cancer (TNBC) is a more aggressive subtype of that usually progresses rapidly, develops drug resistance, metastasis, and relapses, remains challenge for clinicians to treat. Programmed cell death (PCD), conserved mechanism suicide controlled by various pathways, contributed carcinogenesis progression. Nevertheless, the prognostic significance PCD-related genes in TNBC largely unclear, accurate models are urgently needed. Gene expression profiles clinical information patients were obtained from The Cancer Genome Atlas (TCGA) Expression Omnibus (GEO) database. Least absolute shrinkage selection operator (LASSO) multivariate Cox regression analysis used establish gene signature. Kaplan-Meier plotter, receiver operating characteristic curves, nomogram applied validate value set enrichment was carried out investigate pathways molecular functions. Five including SEPTIN3, SCARB1, CHML, SYNM, COL5A3 identified risk score patients. Patients stratified into high-risk or low-risk group showed significantly different survival outcome, immune infiltration, susceptibility. curves good performance prediction cohorts. revealed five-gene signature associated with tumor metabolism, proliferation, invasion microenvironment. Nomogram established. A novel five could be TNBC. present work might offer useful insights digging sensitive effective biomarkers prognosis establishing model management.

Язык: Английский

Elevated POSTN expression predicts poor prognosis and is associated with radioresistance in cervical cancer patients treated with radical radiotherapy DOI Creative Commons
Cuiqin Huang,

Wentao Xiao,

Xiuzhong Yao

и другие.

Scientific Reports, Год журнала: 2025, Номер 15(1)

Опубликована: Фев. 4, 2025

Cervical cancer (CC) is a significant global health issue and remains one of the leading causes cancer-related mortality in women. Radiotherapy crucial treatment modality for CC; however, tumor heterogeneity resistance to radiotherapy often result suboptimal outcomes some patients, including recurrence metastasis. Periostin (POSTN), matricellular protein within microenvironment, has been implicated promotion progression resistance, particularly through mechanisms such as epithelial-mesenchymal transition (EMT). Despite this, role POSTN CC patients underexplored. Therefore, this study, we investigated prognostic significance expression undergoing radical explored potential underlying resistance. We analyzed data from 92 The Cancer Genome Atlas (TCGA) 153 our institution, assessing levels mRNA analysis immunohistochemistry (IHC). Our findings revealed that high was significantly associated with advanced stages, poorer outcomes, worse overall survival (OS). Additionally, multivariate Cox regression identified an independent factor radiotherapy. A nomogram integrating clinicopathological features demonstrated superior predictive accuracy OS. Drug sensitivity suggested may be linked multiple chemotherapeutic agents. Furthermore, weighted correlation network (WGCNA) gene set enrichment (GSEA) EMT top enriched pathway expression, suggesting it play critical Subsequently, vitro experiments confirmed knockdown inhibited HeLa cell proliferation, invasion, enhanced radiosensitivity, while promoting apoptosis. These indicate risk poor prognosis radiotherapy, targeting improve efficacy by reducing proliferation

Язык: Английский

Процитировано

0

Construction of a novel five programmed cell death-related gene signature as a promising prognostic model for triple negative breast cancer DOI Creative Commons
Qing Shao, Haiyan Gao, Ziying Wang

и другие.

PeerJ, Год журнала: 2025, Номер 13, С. e19359 - e19359

Опубликована: Апрель 28, 2025

Triple negative breast cancer (TNBC) is a more aggressive subtype of that usually progresses rapidly, develops drug resistance, metastasis, and relapses, remains challenge for clinicians to treat. Programmed cell death (PCD), conserved mechanism suicide controlled by various pathways, contributed carcinogenesis progression. Nevertheless, the prognostic significance PCD-related genes in TNBC largely unclear, accurate models are urgently needed. Gene expression profiles clinical information patients were obtained from The Cancer Genome Atlas (TCGA) Expression Omnibus (GEO) database. Least absolute shrinkage selection operator (LASSO) multivariate Cox regression analysis used establish gene signature. Kaplan-Meier plotter, receiver operating characteristic curves, nomogram applied validate value set enrichment was carried out investigate pathways molecular functions. Five including SEPTIN3, SCARB1, CHML, SYNM, COL5A3 identified risk score patients. Patients stratified into high-risk or low-risk group showed significantly different survival outcome, immune infiltration, susceptibility. curves good performance prediction cohorts. revealed five-gene signature associated with tumor metabolism, proliferation, invasion microenvironment. Nomogram established. A novel five could be TNBC. present work might offer useful insights digging sensitive effective biomarkers prognosis establishing model management.

Язык: Английский

Процитировано

0