Origin and evolution of mitochondrial inner membrane composition DOI Creative Commons
Kailash Venkatraman, Nicolas-Frédéric Lipp, Itay Budin

и другие.

Journal of Cell Science, Год журнала: 2025, Номер 138(9)

Опубликована: Апрель 23, 2025

ABSTRACT Unique membrane architectures and lipid building blocks underlie the metabolic non-metabolic functions of mitochondria. During eukaryogenesis, mitochondria likely arose from an alphaproteobacterial symbiont Asgard archaea-related host cell. Subsequently, evolved inner folds known as cristae alongside a specialized composition supported by transport machinery. Advancements in phylogenetic methods genomic metagenomic data have suggested potential origins for cristae-shaping protein complexes, such mitochondrial contact site cristae-organizing system (MICOS). MICOS homologs function formation cristae-like intracytoplasmic membranes (ICMs) diverse extant alphaproteobacteria. The machinery responsible synthesizing key phospholipids – which cooperate with proteins to establish architecture could also bacterial ancestor, but its been less explored. In this Review, we examine current understanding evolution, highlighting distinctions between prokaryotic eukaryotic mitochondrial-specific lipids their differing roles shaping ICM architecture, propose model explaining concurrent specialization lipidome structure eukaryogenesis. We discuss how advancements across range disciplines are shedding light on multiple components co-evolved support central

Язык: Английский

GPD1L‐Mediated Glycerophospholipid Metabolism Dysfunction in Women With Diminished Ovarian Reserve: Insights From Pseudotargeted Metabolomic Analysis of Follicular Fluid DOI Creative Commons
Jiaqi Wu,

Xuehan Zhao,

Ying Fang

и другие.

Cell Proliferation, Год журнала: 2025, Номер unknown

Опубликована: Март 20, 2025

ABSTRACT Diminished ovarian reserve (DOR) is a pathological condition characterised by reduced function, which refers to the decreased quality and quantity of oocytes, potentially causing female infertility various health issues. Follicular fluid (FF) serves as microenvironment for follicular development oocyte maturation, gaining an in‐depth understanding metabolic state FF will help us uncover key biological processes involved in aging, while specific underlying pathogenic mechanisms are not fully understood. In this study, we utilised pseudotargeted metabolomic analysis reveal glycerophospholipid metabolism dysfunction mediated GPD1L DOR patients. We also found that was downregulated granulosa cells (GCs) patients, resulting increased cell apoptosis mitochondrial dysfunction. Moreover, our results demonstrated expression could induce atresia impair mouse ovaries. Altogether, research suggested GCs metabolites pathway act novel biomarkers diagnosis, paving way new theoretical basis pathogenesis DOR.

Язык: Английский

Процитировано

0

The Warburg hypothesis and the emergence of the mitochondrial metabolic theory of cancer DOI Creative Commons
Thomas N. Seyfried,

Derek C. Lee,

Tomás Duraj

и другие.

Journal of Bioenergetics and Biomembranes, Год журнала: 2025, Номер unknown

Опубликована: Апрель 8, 2025

Abstract Otto Warburg originally proposed that cancer arose from a two-step process. The first step involved chronic insufficiency of mitochondrial oxidative phosphorylation (OxPhos), while the second protracted compensatory energy synthesis through lactic acid fermentation. His extensive findings showed oxygen consumption was lower lactate production higher in cancerous tissues than non-cancerous tissues. considered both and extracellular as accurate markers for ATP OxPhos glycolysis, respectively. Warburg’s hypothesis challenged showing remained high some cells despite elevated suggesting largely unimpaired. New information indicates neither nor are surrogates quantification cells. also did not know significant amount could come glutamine-driven substrate level glutaminolysis pathway with succinate produced end product, thus confounding linkage to origin within mitochondria. Moreover, new shows cytoplasmic lipid droplets aerobic fermentation biomarkers insufficiency. original can now be linked more complete understanding how underlies dysregulated cell growth. These address several questionable assumptions regarding allowing field advance effective therapeutic strategies less toxic metabolic management prevention cancer.

Язык: Английский

Процитировано

0

Origin and evolution of mitochondrial inner membrane composition DOI Creative Commons
Kailash Venkatraman, Nicolas-Frédéric Lipp, Itay Budin

и другие.

Journal of Cell Science, Год журнала: 2025, Номер 138(9)

Опубликована: Апрель 23, 2025

ABSTRACT Unique membrane architectures and lipid building blocks underlie the metabolic non-metabolic functions of mitochondria. During eukaryogenesis, mitochondria likely arose from an alphaproteobacterial symbiont Asgard archaea-related host cell. Subsequently, evolved inner folds known as cristae alongside a specialized composition supported by transport machinery. Advancements in phylogenetic methods genomic metagenomic data have suggested potential origins for cristae-shaping protein complexes, such mitochondrial contact site cristae-organizing system (MICOS). MICOS homologs function formation cristae-like intracytoplasmic membranes (ICMs) diverse extant alphaproteobacteria. The machinery responsible synthesizing key phospholipids – which cooperate with proteins to establish architecture could also bacterial ancestor, but its been less explored. In this Review, we examine current understanding evolution, highlighting distinctions between prokaryotic eukaryotic mitochondrial-specific lipids their differing roles shaping ICM architecture, propose model explaining concurrent specialization lipidome structure eukaryogenesis. We discuss how advancements across range disciplines are shedding light on multiple components co-evolved support central

Язык: Английский

Процитировано

0