Nuclear accumulation of YTHDF1 regulates mRNA splicing in the DNA damage response DOI Creative Commons
Jingyu Hou, Yong-zhong Gao,

Bing Han

и другие.

Science Advances, Год журнала: 2025, Номер 11(16)

Опубликована: Апрель 16, 2025

YTH domain–containing family protein 1 (YTHDF1), a reader of N6-methyladenosine (m 6 A), has been implicated in regulating RNA metabolism the cytosol. Here, we report role YTHDF1 within nucleus response to genotoxic stress. Upon radiation, is phosphorylated at serine-182 an ataxia telangiectasia and Rad3-related–dependent manner. This phosphorylation inhibits exportin 1–mediated nuclear export YTHDF1, resulting its accumulation nucleus. Nuclear enhances binding capacity serine- arginine-rich splicing factor 2 group m A-modified exons, leading increased exon inclusion. Specifically, promotes expression DNA repair genes, such as BRCA1 TP53BP1 , thereby mitigating excessive damage. Depletion sensitizes cancer cells radiation treatment. Together, our study reveals crucial A-mediated messenger damage response, proposing it potential target for therapy.

Язык: Английский

RNA Damage and Its Implications in Genome Stability DOI

Mustapha Olatunji,

Yuan Liu

DNA repair, Год журнала: 2025, Номер 147, С. 103821 - 103821

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

NAT10 and N 4 -acetylcytidine restrain R-loop levels and related inflammatory responses DOI
Turja K. Debnath, Nathan S. Abell, Yi-Ru Li

и другие.

Science Advances, Год журнала: 2025, Номер 11(13)

Опубликована: Март 26, 2025

N 4 -acetylcytidine (ac C) is deposited on diverse RNAs by -acetyltransferase 10 (NAT10), a protein with high biological relevance for aging and cancer. We performed comprehensive survey of ac C using metabolic labeling, sodium cyanoborohydride chemical treatment coupled to next-generation sequencing (NGS), antibody–based cell molecular biology techniques. Our analysis shows that NAT10-dependent C-acetylation robust in rRNA specific tRNAs but low/spurious mRNA. It also revealed an inflammatory signature mutagenesis at transcriptionally active sites NAT10-KO cells. This finding led us explore the role NAT10 R-loops, which were recently linked APOBEC3B-mediated mutagenesis. showed R-loops are C-acetylated manner. Furthermore, restrains levels subset differentially expressed genes catalytic activity–dependent Together cellular data showing C-modified RNA endosomal structures, we propose increased C-unmodified RNAs, likely derived from structures induce responses.

Язык: Английский

Процитировано

0

Sacred codes: preservation, permutation and expression DOI Creative Commons
Aila Akosua Kattner

Biomedical Journal, Год журнала: 2025, Номер unknown, С. 100852 - 100852

Опубликована: Апрель 1, 2025

This issue of the Biomedical Journal features a special section on epigenetics in infection, exploring epitranscriptomic modifications malaria parasite Plasmodium and African sleeping sickness Trypanosoma, with focus how these influence development survival strategies. Additionally, this reviews regulatory mechanisms asymmetric cell division, assessment needs for children developmental coordination disorder along recommendations, advancements 3D chondrocyte culturing tissue engineering. Also examined are three key pathways exacerbating long COVID, increasing impact electromagnetic exposure built environments human health, recent updates liver transplantation criteria hepatocellular carcinoma complicated by portal vein tumor thrombosis. Research bladder cancer investigates role activating transcription factor 3, known its anti-tumor properties, link to metformin. Another study demonstrates efficacy Sanger sequencing streamlining mitochondrial diagnostics, enabling timely identification based clinical evidence. Additional studies present novel intervention approach autoimmune diseases, artificial bone grafts enhance regeneration, benefits public hospital participation oncological trials. Finally, confirms suitability transthoracic echocardiography diagnosing suspected acute aortic syndrome.

Язык: Английский

Процитировано

0

When RNA damage induces DNA breaks DOI
Ivan Mikičić, Petra Beli

Trends in Cell Biology, Год журнала: 2025, Номер unknown

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

Nuclear accumulation of YTHDF1 regulates mRNA splicing in the DNA damage response DOI Creative Commons
Jingyu Hou, Yong-zhong Gao,

Bing Han

и другие.

Science Advances, Год журнала: 2025, Номер 11(16)

Опубликована: Апрель 16, 2025

YTH domain–containing family protein 1 (YTHDF1), a reader of N6-methyladenosine (m 6 A), has been implicated in regulating RNA metabolism the cytosol. Here, we report role YTHDF1 within nucleus response to genotoxic stress. Upon radiation, is phosphorylated at serine-182 an ataxia telangiectasia and Rad3-related–dependent manner. This phosphorylation inhibits exportin 1–mediated nuclear export YTHDF1, resulting its accumulation nucleus. Nuclear enhances binding capacity serine- arginine-rich splicing factor 2 group m A-modified exons, leading increased exon inclusion. Specifically, promotes expression DNA repair genes, such as BRCA1 TP53BP1 , thereby mitigating excessive damage. Depletion sensitizes cancer cells radiation treatment. Together, our study reveals crucial A-mediated messenger damage response, proposing it potential target for therapy.

Язык: Английский

Процитировано

0