The Function of MicroRNAs in Autoimmune Diseases DOI Open Access

Patricia Mendes Demo

Science Insights, Год журнала: 2022, Номер 40(5), С. 499 - 505

Опубликована: Апрель 30, 2022

MicroRNAs (miRNAs) are a family of non-coding single-stranded RNAs with length around 18-25 nucleotides that play critical role in the control gene expression, cell proliferation, differentiation, and apoptosis, as well biological growth development illness. Autoimmune diseases group disorders characterized by tissue organ damage resulting dysfunction result autoimmune responses. Numerous studies have discovered aberrant expression certain miRNAs may serve biomarkers for early detection these or therapeutic targets. The properties their significance illnesses discussed this review.

Язык: Английский

Glucose metabolism is controlled by non-coding RNAs in autoimmune diseases; a glimpse into immune system dysregulation DOI
Raed Obaid Saleh,

Raed Fanoukh Aboqader Al-Aouadi,

Nujud Almuzaini

и другие.

Human Immunology, Год журнала: 2025, Номер 86(3), С. 111269 - 111269

Опубликована: Фев. 24, 2025

Язык: Английский

Процитировано

1

Neuroprotective and antioxidant effects of docosahexaenoic acid (DHA) in an experimental model of multiple sclerosis DOI
Ana Muñoz-Jurado, Begoña M. Escribano,

Alberto Galván

и другие.

The Journal of Nutritional Biochemistry, Год журнала: 2023, Номер 124, С. 109497 - 109497

Опубликована: Окт. 23, 2023

Язык: Английский

Процитировано

7

Animal model of multiple sclerosis: Experimental autoimmune encephalomyelitis DOI
Ana Muñoz-Jurado, Begoña M. Escribano, Isaac Túnez

и другие.

Methods in cell biology, Год журнала: 2024, Номер unknown, С. 35 - 60

Опубликована: Янв. 1, 2024

Язык: Английский

Процитировано

1

Rs205764 and rs547311 in linc00513 may influence treatment responses in multiple sclerosis patients: A pharmacogenomics Egyptian study DOI Creative Commons
Nada Sherif Amin, Mostafa K. Abd El‐Aziz, Mohamed Hamed

и другие.

Frontiers in Immunology, Год журнала: 2023, Номер 14

Опубликована: Фев. 17, 2023

Multiple sclerosis (MS) is characterized by a complex etiology that reflected in the lack of consistently predictable treatment responses across patients seemingly similar characteristics. Approaches to demystify underlying predictors aberrant have made use genome-wide association studies (GWAS), with imminent progress identifying single nucleotide polymorphisms (SNPs) associated MS risk, disease progression, and response. Ultimately, such pharmacogenomic aim utilize approach personalized medicine maximize patient benefit minimize rate progression.Very limited research available around long intergenic non-coding RNA (linc)00513, recently being reported as novel positive regulator type-1 interferon (IFN) pathway, following its overexpression presence two polymorphisms: rs205764 rs547311 promoter region this gene. We attempt provide data on prevalence genetic variations at Egyptian patients, correlate these patients' disease-modifying treatments.Genomic DNA from 144 RRMS was isolated analyzed for genotypes positions interest linc00513 using RT-qPCR. Genotype groups were compared regards their response treatment; additional secondary clinical parameters including estimated disability status score (EDSS), onset examined relation polymorphisms.Polymorphisms significantly higher fingolimod lower dimethylfumarate. Moreover, average EDSS carrying higher, whereas no correlation appeared exist MS.Understanding interplay factors influencing pivotal MS. One contributing patient's treatment, well disability, may be material, linc00513. Through work, we propose partially drive inconsistent MS; also draw attention towards approaches, screening specific polymorphisms, possibly direct choices disease.

Язык: Английский

Процитировано

2

Non-coding RNAs in the epigenetic landscape of cutaneous T-cell lymphoma DOI

Monaza Adeeb,

Lubna Therachiyil,

Safwan Moton

и другие.

International review of cell and molecular biology, Год журнала: 2023, Номер unknown, С. 149 - 171

Опубликована: Янв. 1, 2023

Язык: Английский

Процитировано

2

The next frontier in multiple sclerosis therapies: Current advances and evolving targets. DOI
K. Trideva Sastri, Neha Gupta,

Anbarasu Kannan

и другие.

European Journal of Pharmacology, Год журнала: 2024, Номер 985, С. 177080 - 177080

Опубликована: Ноя. 2, 2024

Язык: Английский

Процитировано

0

Insight into Early Diagnosis of Multiple Sclerosis by Targeting Prognostic Biomarkers DOI
Nidhi Puranik, Dhananjay Yadav, Minseok Song

и другие.

Current Pharmaceutical Design, Год журнала: 2023, Номер 29(32), С. 2534 - 2544

Опубликована: Сен. 1, 2023

Multiple sclerosis (MS) is a central nervous system (CNS) immune-mediated disease that mainly strikes young adults and leaves them disabled. MS an autoimmune illness causes the immune to attack brain spinal cord. The myelin sheaths, which insulate nerve fibers, are harmed by our own cells, this interferes with signal transmission. Numbness, tingling, mood swings, memory problems, exhaustion, agony, vision and/or paralysis just few of symptoms. Despite technological advancements significant research efforts in recent years, diagnosing can still be difficult. Each patient's distinct due heterogeneous complex pathophysiology diverse types courses. There pressing need identify markers will allow for more rapid accurate diagnosis prognosis assessments choose best course treatment each patient. cerebrospinal fluid (CSF) excellent source particular indicators associated pathology. CSF contains molecules represent pathological processes such as inflammation, cellular damage, loss blood-brain barrier integrity. Oligoclonal bands, neurofilaments, MS-specific miRNA, lncRNA, IgG-index, anti-aquaporin 4 antibodies all clinically utilised diagnosis. In slew new possible biomarkers have been presented. review, we look at what know about molecular how they aid differentiation different forms options, monitoring predicting progression, therapy response, consequences during opportunistic infections.

Язык: Английский

Процитировано

1

Ataxin‐1 controls the expression of specific noncoding RNAs in B cells upon autoimmune demyelination DOI Creative Commons
Qin Ma, Alessandro Didonna

Immunology and Cell Biology, Год журнала: 2023, Номер 101(4), С. 358 - 367

Опубликована: Янв. 22, 2023

B cells play a key mechanistic role in the pathogenesis of multiple sclerosis (MS), chronic neurological disease central nervous system with an autoimmune etiology. contribute to initiation and progression by acting as professional antigen-presenting well via secreting autoantibodies proinflammatory cytokines. We have recently shown that polyglutamine protein ataxin-1, which was first linked movement disorder spinocerebellar ataxia type 1, also acts master regulator B-cell functions context autoimmunity. In fact, ataxin-1-deficient mice display aggravated manifestation MS model experimental encephalomyelitis along aberrant functions. Consistent this scenario, transcriptomic analysis Atxn1-null highlighted distinct genetic signatures involved cell activation, proliferation antigen presentation. To further characterize we profiled noncoding transcriptome controlled ataxin-1 compartment upon encephalitogenic challenge. show two specific classes RNAs, namely, processed pseudogenes intergenic long are differentially regulated disease. Furthermore, pathway network analyses on their putative protein-coding gene targets found significant enrichment ontologies related mitosis, together molecular processes relevant such chitin metabolism. Altogether, these findings shed light possible contribution RNAs biology pathogenesis, establish immunomodulatory demyelination.

Язык: Английский

Процитировано

0

Transcriptome driven discovery of novel candidate genes for human neurological disorders in the telomer-to-telomer genome assembly era DOI Creative Commons
Clemens Falker‐Gieske

Human Genomics, Год журнала: 2023, Номер 17(1)

Опубликована: Окт. 23, 2023

Abstract Background With the first complete draft of a human genome, Telomere-to-Telomere Consortium unlocked previously concealed genomic regions for genetic analyses. These harbour nearly 2000 potential novel genes with unknown function. In order to uncover candidate associated neurological pathologies, comparative transcriptome study using T2T-CHM13 and GRCh38 genome assemblies was conducted on published datasets eight distinct disorders. Results The analysis differential expression in RNA sequencing data led identification 336 linked Additionally, it revealed that, average, 3.6% differentially expressed detected assembly may represent false positives. Among noteworthy findings, two were discovered, one encoding pore-structured protein other highly ordered β-strand-rich protein. exhibited upregulation multiple epilepsy hold promise as potentially modulating progression disease. Furthermore, an derived from white matter lesions sclerosis patients indicated significant 26 rRNA genes. putative pathology related identified Alzheimer’s disease, amyotrophic lateral sclerosis, glioblastoma, glioma, conditions resulting m.3242 A > G mtDNA mutation. Conclusion results presented here underline facilitating discovery context Moreover, demonstrate value remapping superior assembly. Numerous genes, either causative factors or elements, have been unveiled, warranting further experimental validation.

Язык: Английский

Процитировано

0

The Function of MicroRNAs in Autoimmune Diseases DOI Open Access

Patricia Mendes Demo

Science Insights, Год журнала: 2022, Номер 40(5), С. 499 - 505

Опубликована: Апрель 30, 2022

MicroRNAs (miRNAs) are a family of non-coding single-stranded RNAs with length around 18-25 nucleotides that play critical role in the control gene expression, cell proliferation, differentiation, and apoptosis, as well biological growth development illness. Autoimmune diseases group disorders characterized by tissue organ damage resulting dysfunction result autoimmune responses. Numerous studies have discovered aberrant expression certain miRNAs may serve biomarkers for early detection these or therapeutic targets. The properties their significance illnesses discussed this review.

Язык: Английский

Процитировано

0