Modulating Autophagy in Osteoarthritis: Exploring Emerging Therapeutic Drug Targets DOI Open Access
Corina Andrei, Dragoș Paul Mihai, George Mihai Nițulescu

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(24), С. 13695 - 13695

Опубликована: Дек. 21, 2024

Osteoarthritis (OA) is a degenerative joint disease characterized by the breakdown of cartilage and subsequent inflammation tissues, leading to pain reduced mobility. Despite advancements in symptomatic treatments, disease-modifying therapies for OA remain limited. This narrative review examines dual role autophagy OA, emphasizing its protective functions during early stages potential contribute degeneration later stages. By delving into molecular pathways that regulate autophagy, this highlights intricate interplay with oxidative stress inflammation, key drivers progression. Emerging therapeutic strategies aimed at modulating are explored, including pharmacological agents such as AMP kinase activators, microRNA-based therapies. Preclinical studies reveal encouraging results, demonstrating enhancing can reduce decelerate degradation. However, benefits modulation depend on precise, stage-specific approaches. Excessive or dysregulated advanced may lead chondrocyte apoptosis, exacerbating damage. underscores promise autophagy-based interventions bridging gap between experimental research clinical application. advancing our understanding autophagy’s these findings pave way innovative effective Nonetheless, further essential optimize strategies, address off-target effects, develop safe, targeted treatments improve outcomes patients.

Язык: Английский

Non-apoptotic cell death in osteoarthritis: Recent advances and future DOI Open Access
Pengfei Ji, Ziyu Zhou, Jinyuan Zhang

и другие.

Biomedicine & Pharmacotherapy, Год журнала: 2024, Номер 179, С. 117344 - 117344

Опубликована: Авг. 27, 2024

Osteoarthritis (OA) is the most common degenerative joint disease. Multiple tissues are altered during development of OA, resulting in pain and permanent damage to osteoarticular joints. Current research has demonstrated that non-apoptotic cell death plays a crucial role OA. With continuous targeted therapies, shown great potential prevention treatment We systematically reviewed progress on pathogenesis, development, outcome including autophagy, pyroptosis, ferroptosis, necroptosis, immunogenic death, parthanatos. This article reviews mechanism OA provides theoretical basis for identification new targets treatment.

Язык: Английский

Процитировано

1

Self-healing hydrogels loaded with Spatholobi Caulis alleviate disc degeneration by promoting autophagy in nucelus pulposus DOI Creative Commons
Sanjun Cai, Rui Ding, Hongjun Zhang

и другие.

Materials Today Bio, Год журнала: 2024, Номер 29, С. 101323 - 101323

Опубликована: Ноя. 8, 2024

Intervertebral disc degeneration (IDD) is a common degenerative disease of the spine that has significant impact on both society and human health. Many studies have confirmed there close relationship between IDD senescence apoptosis, autophagy can combat apoptosis senescence. Spatholobi caulis (SC) an herb contains various active compounds are effective in tissue repair regeneration, but it not been explored field IDD. In this study, was first found SC boost reduce Nucleus pulposus cell (NPCs). However, our animal revealed limited absorption SC. To improve bioavailability efficacy SC, we developed hydrogel incorporating quaternary ammonium chitosan (QCS) oxidized starch (OST) as carriers for The QCS-OST/SC exhibits excellent compatibility with cells, be easily injected, release durably. At cellular level, enhances viability, initiates extracellular matrix (ECM), inhibits apoptosis. injection via microneedles (MNs) into discs had successfully diminished rats, which shows broad potential treatment

Язык: Английский

Процитировано

0

Modulating Autophagy in Osteoarthritis: Exploring Emerging Therapeutic Drug Targets DOI Open Access
Corina Andrei, Dragoș Paul Mihai, George Mihai Nițulescu

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(24), С. 13695 - 13695

Опубликована: Дек. 21, 2024

Osteoarthritis (OA) is a degenerative joint disease characterized by the breakdown of cartilage and subsequent inflammation tissues, leading to pain reduced mobility. Despite advancements in symptomatic treatments, disease-modifying therapies for OA remain limited. This narrative review examines dual role autophagy OA, emphasizing its protective functions during early stages potential contribute degeneration later stages. By delving into molecular pathways that regulate autophagy, this highlights intricate interplay with oxidative stress inflammation, key drivers progression. Emerging therapeutic strategies aimed at modulating are explored, including pharmacological agents such as AMP kinase activators, microRNA-based therapies. Preclinical studies reveal encouraging results, demonstrating enhancing can reduce decelerate degradation. However, benefits modulation depend on precise, stage-specific approaches. Excessive or dysregulated advanced may lead chondrocyte apoptosis, exacerbating damage. underscores promise autophagy-based interventions bridging gap between experimental research clinical application. advancing our understanding autophagy’s these findings pave way innovative effective Nonetheless, further essential optimize strategies, address off-target effects, develop safe, targeted treatments improve outcomes patients.

Язык: Английский

Процитировано

0