Role of DNA methylation transferase in urinary system diseases: From basic to clinical perspectives (Review) DOI Creative Commons
Yipin Yang,

Yafen Wang,

Xiying Fan

и другие.

International Journal of Molecular Medicine, Год журнала: 2024, Номер 55(2)

Опубликована: Ноя. 19, 2024

DNA methylation is one of the earliest discovered and most extensively studied epigenetic regulatory mechanisms. Broadly, refers to transfer a methyl group on S‑adenosine‑L‑methionine (SAM) C5 site cytosine, reaction catalysed by methyltransferase (DNMT). This process can either up‑ or down‑regulate gene expression due promoter methylation, leading occurrence certain diseases. Urinary system diseases, known for their high prevalence complex pathogenesis, significantly affect lives health patients. Urological tumours, in particular, represent non‑negligible disease burden worldwide. With development epigenetics, an increasing number studies have demonstrated that DNMT plays important role urinary disease. The present review aims explore relationship between diseases potential clinical management these

Язык: Английский

Exploring viral mimicry combined with epigenetics and tumor immunity: new perspectives in cancer therapy DOI Creative Commons
Ruirui Wang, Xin Dong,

Xiongjian Zhang

и другие.

International Journal of Biological Sciences, Год журнала: 2025, Номер 21(3), С. 958 - 973

Опубликована: Янв. 6, 2025

Viral mimicry refers to an active antiviral response triggered by the activation of endogenous retroviruses (ERVs), usually manifested formation double-stranded RNA (dsRNA) and cellular interferon response, which activates immune system produces anti-tumor effects. Epigenetic studies have shown that epigenetic modifications (e.g. DNA methylation, histone modifications, etc.) play a crucial role in tumorigenesis, progression, treatment resistance. Particularly, alterations methylation may be closely associated with suppression ERVs expression, demethylation restore activity thus strengthen tumor response. Therefore, we propose viral can induce responses microenvironment activating expression ERVs, key regulatory this process. In paper, review intersection mimicry, epigenetics immunotherapy, explore possible interactions synergistic effects among three, aiming provide new theoretical basis potential strategies for cancer immunotherapy.

Язык: Английский

Процитировано

0

Research progress of DNA methylation in the diagnosis and treatment of thyroid carcinoma DOI Creative Commons

Xin Zhang,

Bing Luo, Minjie Sun

и другие.

International Immunopharmacology, Год журнала: 2025, Номер 152, С. 114426 - 114426

Опубликована: Март 9, 2025

Thyroid cancer is the most prevalent endocrine malignancy, and its timely accurate diagnostic prognostic assessments are crucial for enhancing patient survival rates. As an important epigenetic modification, DNA methylation plays a key role in regulation of gene expression tumorigenesis. Recent studies increasingly indicate that abnormal patterns closely associated with onset progression thyroid cancer. This review discusses diagnosing adenocarcinoma, impact on prognosis, potential utility immunotherapy. Additionally, it explores prospect using as biomarker highlights significant personalized treatment article aims to serve resource future research clinical applications advance diagnosis

Язык: Английский

Процитировано

0

Insight into the role of DNA methylation in prognosis and treatment response prediction of gastrointestinal cancers DOI Creative Commons

Abudurousuli Reyila,

Xianchun Gao, Jun Yu

и другие.

Epigenomics, Год журнала: 2025, Номер unknown, С. 1 - 14

Опубликована: Март 14, 2025

Gastrointestinal (GI) cancers impose a significant disease burden, underscoring the critical importance of accurate prognosis prediction and treatment response evaluation. DNA methylation, one most extensively studied epigenetic modifications, has gained prominence due to its reliable measurement across various sample types. Numerous studies have reported that methylation was linked diagnosis, in malignancies, including GI cancers. While diagnostic role been comprehensively reviewed. Recent research increasingly highlighted potential However, no existing reviews exclusively focused on these two aspects. In this review, we retrieved relevant included 230 them our discussion, thereby providing an overview clinical applicability aberrant fields among patients with esophageal, gastric, colorectal, pancreatic cancers, hepatocellular carcinomas. Additionally, discuss limitations current literature propose directions for future research. Specifically, emphasize need standardized methodologies advocate integration gene panels, rather than single genes, address tumor heterogeneity more effectively.

Язык: Английский

Процитировано

0

Unleashing the Potential of Metal Ions in cGAS-STING Activation: Advancing Nanomaterial-Based Tumor Immunotherapy DOI Creative Commons

Xingyin Li,

Shaojie Xu,

Ziliang Su

и другие.

ACS Omega, Год журнала: 2025, Номер unknown

Опубликована: Март 17, 2025

Immunotherapy is a critical modality in cancer treatment with diverse activation pathways. In recent years, the stimulator of interferon genes (STING) signaling pathway has exhibited significant potential tumor immunotherapy. This exerts notable antitumor effects by activating innate and adaptive immunity regulating immune microenvironment. Various metal ions have been identified as effective activators STING and, through design synthesis nanodelivery platforms, applied immunotherapy well combination therapies, such chemotherapy, chemodynamic therapy, photodynamic vaccines. Metal nanomaterials showcase unique advantages immunotherapy; however, there are still aspects that require optimization. review systematically examines existing metal-based nanomaterials, elaborates on mechanisms which different activate pathway, discusses their application models therapies. We also provide comparative analysis over other methods. Our exploration highlights broad prospects treatment, offering new insights directions for advancement

Язык: Английский

Процитировано

0

Uncovering the Effects and Molecular Mechanisms of Shaoyao Decoction Against Colorectal Cancer Using Network Pharmacology Analysis Coupled With Experimental Validation and Gut Microbiota Analysis DOI Creative Commons
Yuan Rong, Guiyu Zhang, Wenhao Ye

и другие.

Cancer Medicine, Год журнала: 2025, Номер 14(6)

Опубликована: Март 1, 2025

ABSTRACT Background Chronic gut inflammation and dysbiosis contribute significantly to colorectal cancer (CRC) development. Shaoyao decoction (SYD) is a well‐established Chinese medicine prescription. Besides ameliorating CRC via anti‐inflammatory effects, SYD modulates microbiota (GM) improve inflammatory responses in ulcerative colitis (UC). However, whether how suppresses by regulating GM remains largely unknown. Methods SD rats were orally administered for 7 days obtain medicated serum. We utilized liquid chromatography–mass spectrometry (LC–MS) analysis, GeneCards, DisGeNET, SwissTargetPrediction databases analyze blank SYD‐medicated rat serum, comparing the findings with those of aqueous extract previous studies identify circulating compounds/components predictable target genes. Using network pharmacology, potential active compounds corresponding hub genes associated modulating suppress selected molecular docking. In vivo experiments, transplantation tumor model was established BALB/c mice using CT26 cells, gavage 14 days. To investigate mechanism SYD‐regulated against CRC, HE IHC staining, Western blotting, 16S rRNA sequencing employed. Results LC–MS identified 26 computationally predicted Network pharmacology prioritized 13 targeting 8 inflammation/immunity‐related (IL‐17/TNF pathways), validated dose‐dependently suppressed growth ( p < 0.05, medium/high doses), as confirmed staining analysis Ki‐67. Notably, potentially delayed liver metastasis alleviated hepatic injury tumor‐bearing mice. blotting demonstrated SYD's inhibition IL‐17/TNF/NF‐κB axis, aligning computational predictions. revealed SYD‐enriched Akkermansia structural shifts, mechanistically linking remodeling anti‐tumor efficacy. Conclusions combats dual modulation signaling ecosystems (e.g., enrichment). This microbiota‐immune crosstalk positions adjunct conventional therapies, particularly patients dysbiosis.

Язык: Английский

Процитировано

0

YEATS2: a novel cancer epigenetic reader and potential therapeutic target DOI Creative Commons

Kangkang Ji,

Guoping Chen, Bin Wang

и другие.

Cancer Cell International, Год журнала: 2025, Номер 25(1)

Опубликована: Апрель 26, 2025

YEATS2, an evolutionarily conserved reader of histone acylation marks (H3K27ac, H3K27cr, H3K27bz), functions as a central oncogenic driver in diverse cancers, including non-small cell lung cancer (NSCLC), pancreatic ductal adenocarcinoma (PDAC), and hepatocellular carcinoma (HCC). Its structurally plastic YEATS domain bridges acyl-CoA metabolism to chromatin remodeling, amplifying transcription survival genes such MYC, BCL2, PD-L1. YEATS2 orchestrates malignancy-specific programs-sustaining ribosome biogenesis NSCLC through ATAC complex recruitment, enhancing NF-κB-dependent immune evasion PDAC, activating PI3K/AKT-driven metabolic rewiring HCC. Structural studies demonstrate unique aromatic cage architecture that selectively engages acylated histones. Although pyrazolopyridine-based inhibitors targeting the show preclinical efficacy, developing isoform-selective agents remains challenging. Clinically, overexpression correlates with therapy resistance may synergize checkpoint blockade. This review integrates mechanistic insights into role epigenetic regulation, evaluates its therapeutic potential, proposes future directions: elucidating full-length topologies, mapping synthetic lethal interactors, optimizing selective inhibitors. Disrupting YEATS2-mediated adaptation presents novel opportunities for precision therapy.

Язык: Английский

Процитировано

0

Role of DNA methylation transferase in urinary system diseases: From basic to clinical perspectives (Review) DOI Creative Commons
Yipin Yang,

Yafen Wang,

Xiying Fan

и другие.

International Journal of Molecular Medicine, Год журнала: 2024, Номер 55(2)

Опубликована: Ноя. 19, 2024

DNA methylation is one of the earliest discovered and most extensively studied epigenetic regulatory mechanisms. Broadly, refers to transfer a methyl group on S‑adenosine‑L‑methionine (SAM) C5 site cytosine, reaction catalysed by methyltransferase (DNMT). This process can either up‑ or down‑regulate gene expression due promoter methylation, leading occurrence certain diseases. Urinary system diseases, known for their high prevalence complex pathogenesis, significantly affect lives health patients. Urological tumours, in particular, represent non‑negligible disease burden worldwide. With development epigenetics, an increasing number studies have demonstrated that DNMT plays important role urinary disease. The present review aims explore relationship between diseases potential clinical management these

Язык: Английский

Процитировано

0