SIRT1: A Potential Therapeutic Target for Coronary Heart Disease Combined with Anxiety or Depression DOI
Hongbo Yu, Xinping Li, Bo Ning

и другие.

Journal of drug targeting, Год журнала: 2024, Номер 33(3), С. 328 - 340

Опубликована: Окт. 29, 2024

Coronary heart disease (CHD) combined with anxiety or depression is increasingly receiving attention in the clinical field of cardiology, and exploring comorbidity pathological mechanisms cardiovascular psychological disorders a hot research topic for scholars this field. Current suggests that Silent Information Regulatory Factor 1 (SIRT1) may serve as potential biomarker mechanism treatment CHD depression. SIRT1 considered promising therapeutic target depression, ability to regulate inflammatory cytokine levels, alleviate oxidative stress damage, activate multiple signalling pathways, reduce platelet hyperresponsiveness, exert neuroprotective cardioprotective effects. In comprehensive review, we deeply studied structure, function, SIRT1, discussed its protective effects nervous system. The latest progress SIRT1's role was emphasised, including specific regulating response, alleviating stress, mediating various pathways. addition, article also summarises patients

Язык: Английский

Regulation of cellular senescence in tumor progression and therapeutic targeting: mechanisms and pathways DOI Creative Commons
Bowei Liu, Zhigang Peng, Hao Zhang

и другие.

Molecular Cancer, Год журнала: 2025, Номер 24(1)

Опубликована: Апрель 2, 2025

Cellular senescence, a stable state of cell cycle arrest induced by various stressors or genomic damage, is recognized as hallmark cancer. It exerts context-dependent dual role in cancer initiation and progression, functioning tumor suppressor promoter. The complexity senescence arises from its mechanistic diversity, potential reversibility, heterogeneity. A key mediator these effects the senescence-associated secretory phenotype (SASP), repertoire bioactive molecules that influence microenvironment (TME) remodeling, modulate behavior, contribute to therapeutic resistance. Given intricate biology, presents both challenges opportunities for intervention. Strategies targeting pathways, including senescence-inducing therapies senolytic approaches, offer promising avenues treatment. This review provides comprehensive analysis regulatory mechanisms governing cellular tumors. We also discuss emerging strategies highlighting novel opportunities. deeper understanding processes essential developing precision improving clinical outcomes.

Язык: Английский

Процитировано

3

PGC1-Alpha/Sirt3 Signaling Pathway Mediates the Anti-Pulmonary Fibrosis Effect of Hirudin by Inhibiting Fibroblast Senescence DOI Creative Commons
Bin He, Qian Zeng, Yumei Tian

и другие.

Biomedicines, Год журнала: 2024, Номер 12(7), С. 1436 - 1436

Опубликована: Июнь 27, 2024

Idiopathic pulmonary fibrosis (IPF) is a chronic, progressive fibrotic lung disease for which there lack of effective pharmacological treatments. Hirudin, natural peptide extracted from leeches, has been used broad purposes. In this study, we investigated the therapeutic effects hirudin on IPF and its related mechanism action. By constructing mouse model treating it with in vivo, found that exerted anti-fibrotic, anti-oxidative, anti-fibroblast senescence effects. Moreover, using an vitro stress-induced premature primary fibroblasts hirudin, observed inhibition fibroblast upregulation PGC1-alpha Sirt3 expression. However, specific silencing or suppressed effect hirudin. Thus, PGC1-alpha/Sirt3 pathway mediates potentially serving as molecular underlying anti-fibrosis anti-oxidative stress lungs.

Язык: Английский

Процитировано

3

Microplastics/nanoplastics contribute to aging and age-related diseases: mitochondrial dysfunction as a crucial role DOI
Liang Kong, Shuhao Li, Yu Fu

и другие.

Food and Chemical Toxicology, Год журнала: 2025, Номер 199, С. 115355 - 115355

Опубликована: Фев. 26, 2025

Язык: Английский

Процитировано

0

Mitochondrial malfunction-initiated Leydig cell premature senescence partially participates in 1-nitropyrene-evoked downregulation of steroidogenic synthases in testes DOI

Jian Li,

Xin Dong,

Lan Gao

и другие.

Free Radical Biology and Medicine, Год журнала: 2024, Номер 225, С. 456 - 468

Опубликована: Окт. 18, 2024

Язык: Английский

Процитировано

2

Puerarin alleviates LPS-induced endothelial cells injury via SIRT1-mediated mitochondrial homeostasis signaling DOI Creative Commons

Xing Chang,

Meng Cheng, Ying Li

и другие.

Journal of Holistic Integrative Pharmacy, Год журнала: 2024, Номер 5(3), С. 205 - 214

Опубликована: Сен. 1, 2024

Язык: Английский

Процитировано

0

SIRT1: A Potential Therapeutic Target for Coronary Heart Disease Combined with Anxiety or Depression DOI
Hongbo Yu, Xinping Li, Bo Ning

и другие.

Journal of drug targeting, Год журнала: 2024, Номер 33(3), С. 328 - 340

Опубликована: Окт. 29, 2024

Coronary heart disease (CHD) combined with anxiety or depression is increasingly receiving attention in the clinical field of cardiology, and exploring comorbidity pathological mechanisms cardiovascular psychological disorders a hot research topic for scholars this field. Current suggests that Silent Information Regulatory Factor 1 (SIRT1) may serve as potential biomarker mechanism treatment CHD depression. SIRT1 considered promising therapeutic target depression, ability to regulate inflammatory cytokine levels, alleviate oxidative stress damage, activate multiple signalling pathways, reduce platelet hyperresponsiveness, exert neuroprotective cardioprotective effects. In comprehensive review, we deeply studied structure, function, SIRT1, discussed its protective effects nervous system. The latest progress SIRT1's role was emphasised, including specific regulating response, alleviating stress, mediating various pathways. addition, article also summarises patients

Язык: Английский

Процитировано

0