Nature Reviews Endocrinology, Год журнала: 2024, Номер unknown
Опубликована: Окт. 8, 2024
Язык: Английский
Nature Reviews Endocrinology, Год журнала: 2024, Номер unknown
Опубликована: Окт. 8, 2024
Язык: Английский
Cell stem cell, Год журнала: 2024, Номер unknown
Опубликована: Сен. 1, 2024
Язык: Английский
Процитировано
7Investigative Ophthalmology & Visual Science, Год журнала: 2011, Номер 52(8), С. 5786 - 5786
Опубликована: Июнь 21, 2011
Purpose.: The aim of the present study is to evaluate roles TGFs β1 and β2, glial cell line–derived neurotrophic factor (GDNF), nerve growth (NGF) in pathogenesis idiopathic epiretinal membrane (ERM). Methods.: Eight patients, six males two females, with an average age 60.25 ± 17.16 years (range, 33–75 years) who were affected by ERM enrolled study. All patients underwent standard pars plana vitrectomy surgery removal specific ELISA was performed TGFβ1, TGFβ2, GDNF, NGF vitreous samples. This repeated after acidification samples hydrochloric acid. Results.: Before acidification, analysis revealed a significant increase TGFβ2 (327.98 99.58 pg/mL; range, 206.864–466.235 pg/mL) compared control group (187.17 58.20 132.758–271.707 t = 3.4; P < 0.05). A statistically difference also obtained (618.15 201.43 409.795–866.215 pg/mL 265.04 98.15 152.478–352.101 4.5; Notably, before differences between groups not (t 0.79; 0.46), while levels found comparison (723.41 235.4 vs. 242.84 104.61; 3; Conclusions.: reveals that are associated ERMs, suggesting novel compensatory mechanism so far never proposed.
Язык: Английский
Процитировано
64Biomaterials Advances, Год журнала: 2023, Номер 154, С. 213624 - 213624
Опубликована: Сен. 11, 2023
Язык: Английский
Процитировано
15Bone Research, Год журнала: 2024, Номер 12(1)
Опубликована: Сен. 29, 2024
Язык: Английский
Процитировано
6Nature Reviews Endocrinology, Год журнала: 2024, Номер unknown
Опубликована: Окт. 8, 2024
Язык: Английский
Процитировано
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