To explore the protective mechanism of promethazine against hippocampal neuron injury based on network pharmacology and experimental verification DOI Creative Commons
Bai Li, Fang Li

Medicine, Год журнала: 2024, Номер 103(49), С. e40550 - e40550

Опубликована: Дек. 6, 2024

This study aims to investigate the effect of promethazine (PMZ) on hippocampal neuronal injury through network pharmacology and in vivo experiments. Network pharmacology: The intersection genes PMZ Alzheimer Disease (AD) were obtained, core AD screened. enriched by Gene Ontology Kyoto Encyclopedia Genes Genomes (KEGG) pathway enrichment analyses. In vitro experiment, mouse neurons (HT22) divided into control, glutamate (GLU) model, GLU + treatment groups. control group was given a complete culture medium, model for 24 hours, pretreatment 3 then administered hours. Cell viability determined, cell morphology observed microscopy, reactive oxygen species levels glutathione content detected, protein expression P53, PTGS2, SLC7A11, GPX4 detected western blotting. A total 317 targets, 1934 genes, 125 18 including P53 PTGS2. analysis showed that mainly related proliferation, inflammation, hypoxia, synaptic structure, plasma membrane, oxidoreductase activity. results neuroactive ligand-receptor interaction, senescence, cancer pathway, PI3K-AKT signal neurodegeneration, HIF-1 pathway. experiments: improved GLU-induced decrease morphological changes neurons. inhibited increased injured Up-regulated SLC7A11 expression, regulates SLC7A11-GPX4 antioxidant system protect from oxidative stress injury.

Язык: Английский

Application of carbon-based nanomaterials in Alzheimer's disease DOI
Mengyao Bai, Xu Shao, Chao Wang

и другие.

Materials Horizons, Год журнала: 2024, Номер unknown

Опубликована: Янв. 1, 2024

Alzheimer's disease (AD) is a chronic, progressive neurodegenerative disorder marked by permanent impairment of brain function across the whole brain. This condition results in deterioration cognitive patients and frequently associated with psychological symptoms such as agitation anxiety, imposing significant burden on both their families. Nanomaterials possess numerous distinctive physical chemical features that render them extensively utilized. In biomedical domain, nanomaterials can be utilized for prevention therapy, including medication delivery systems, biosensors, tissue engineering. article explores etiology potential molecular processes AD, well application carbon-based diagnosis treatment AD. Some are carbon quantum dots, nanotubes, graphene, among others. These materials physicochemical highly promising applications biosensing, drug delivery, neuroprotection, photothermal treatment. addition, this review explored various therapeutic approaches AD terms reducing inflammation, preventing oxidative damage, inhibiting Aβ aggregation. The advent nanotechnology has facilitated development novel disease. strategies provide early diagnosis, effective intervention neuroprotection

Язык: Английский

Процитировано

4

Dual‐Carbon Dots Composite: A Multifunctional Photo‐Propelled Nanomotor Against Alzheimer's β‐Amyloid DOI Open Access
Xiaoding Lin, Xiaoyan Dong, Yan Sun

и другие.

Small, Год журнала: 2024, Номер unknown

Опубликована: Окт. 11, 2024

Abstract The abnormal accumulation of β ‐amyloid protein (A ) is considered as the main pathological hallmark Alzheimer's disease (AD). design potent multifunctional theranostic agents targeting A one effective strategies for AD prevention and treatment. Nanomotors intelligent, advanced, nanoplatforms can perform many complex tasks, but their application in theranostics rare. Herein, sub‐10nm dual‐carbon dots composites (ERCD) with photo‐propelled nanomotor behavior are fabricated by conjugating near‐infrared (NIR) carbon (RCD) thermogenic photodynamic capability previously reported epigallocatechin gallate‐derived carbonized polymer (ECD). ERCD‐1 (ECD:RCD = 1:2.5) showed inhibitory similar to ECD absence NIR light, exhibited photooxygenation activity powered “self‐thermophoretic force” under irradiation, significantly enhancing inhibition, disaggregation, capabilities. suppressed fibrillization rapidly disaggregated mature fibrils at very low concentrations (0.5 µg mL −1 ). Moreover, NIR‐activated imaged plaques vivo prolonged nematode lifespan 6 d 2 . As a proof‐of‐concept, this work opened new avenue nanomotors theranostics.

Язык: Английский

Процитировано

3

Phototherapy via Modulation of β‐Amyloid in Combating Alzheimer's Disease DOI Creative Commons
Yunhua Zhang,

Chengyuan Qian,

Yuncong Chen

и другие.

Aggregate, Год журнала: 2025, Номер unknown

Опубликована: Март 4, 2025

ABSTRACT Alzheimer's disease (AD) is one of the most prevalent forms neurodegenerative disease. Although some controversy exists, β‐amyloid peptide (Aβ) recognized to play an essential role in pathophysiology AD. The Aβ species are known exist various forms, including soluble monomers, oligomers, and insoluble aggregates. Despite extensive efforts regulate aggregation, no successful medications have been developed date. Among strategies for AD treatment, phototherapy, photodynamic therapy (PDT), photothermal (PTT), photopharmacology, photobiomodulation (PBM) attracted increased attention because spatiotemporal controllability. Representative examples PDT, PTT, PBM discussed terms inhibitory mechanism, unique properties materials, design phototherapy modulators. major challenges against addressed promising prospects proposed. It concluded that noninvasive light‐assisted approaches will become a strategy intensifying modulation aggregation or promoting clearance thus facilitating treatment.

Язык: Английский

Процитировано

0

Harnessing Photo‐Energy Conversion in Nanomaterials for Precision Theranostics DOI Creative Commons
Jingyu Shi, Yadi Fan, Qin Zhang

и другие.

Advanced Materials, Год журнала: 2025, Номер unknown

Опубликована: Май 16, 2025

Abstract The rapidly advancing field of theranostics aims to integrate therapeutic and diagnostic functionalities into a single platform for precision medicine, enabling the simultaneous treatment monitoring diseases. Photo‐energy conversion‐based nanomaterials have emerged as versatile that utilizes unique properties light activate with high spatial temporal precision. This review provides comprehensive overview recent developments in photo‐energy conversion using nanomaterials, highlighting their applications disease theranostics. discussion begins by exploring fundamental principles including types materials used various light‐triggered mechanisms, such photoluminescence, photothermal, photoelectric, photoacoustic, photo‐triggered SERS, photodynamic processes. Following this, delves broad spectrum emphasizing role diagnosis major diseases, cancer, neurodegenerative disorders, retinal degeneration, osteoarthritis. Finally, challenges opportunities technologies are discussed, aiming advance personalized medicine.

Язык: Английский

Процитировано

0

Deep eutectic solvent-assisted preparation of resveratrol-based carbon dots with high biocompatibility for in situ screening inhibitors on amyloid protein aggregation and scavenging reactive oxygen species DOI

Luo Wan-chun,

Ming Jiang, Li Li

и другие.

Sensors and Actuators B Chemical, Год журнала: 2023, Номер 400, С. 134901 - 134901

Опубликована: Ноя. 3, 2023

Язык: Английский

Процитировано

8

Multifunctional selenium-doped carbon dots for modulating Alzheimer's disease related toxic ions, inhibiting amyloid aggregation and scavenging reactive oxygen species DOI
Xu Shao,

Tiange Fan,

Chaoren Yan

и другие.

International Journal of Biological Macromolecules, Год журнала: 2024, Номер unknown, С. 139333 - 139333

Опубликована: Дек. 1, 2024

Язык: Английский

Процитировано

2

An in situ-activated and chemi-excited photooxygenation system based on G-poly(thioacetal) for Aβ1-42 aggregates DOI
Shasha Liu, Yan-Ping Li,

Jinrong Yang

и другие.

Journal of Materials Chemistry B, Год журнала: 2024, Номер 12(42), С. 10850 - 10860

Опубликована: Янв. 1, 2024

BD-6-QM/NPs are chemically activated by H 2 O to produce 1 , inhibit Aβ 1–42 aggregation, promote BV2 phagocytosis of and alleviate -induced neurotoxicity.

Язык: Английский

Процитировано

1

Application of carbon dots-based nanomaterials in amyloid aggregation disease DOI
Chaoren Yan, Xu Shao,

Yixuan Wang

и другие.

Carbon, Год журнала: 2024, Номер unknown, С. 119971 - 119971

Опубликована: Дек. 1, 2024

Язык: Английский

Процитировано

1

Multifunctional carbonized polymer dots: Synthesis and applications in retarding protein amyloid aggregation, capturing free radicals and cell imaging DOI
Jie Li, Sujuan Wang, Jiawei Dong

и другие.

Journal of Molecular Structure, Год журнала: 2024, Номер 1319, С. 139419 - 139419

Опубликована: Июль 26, 2024

Язык: Английский

Процитировано

0

To explore the protective mechanism of promethazine against hippocampal neuron injury based on network pharmacology and experimental verification DOI Creative Commons
Bai Li, Fang Li

Medicine, Год журнала: 2024, Номер 103(49), С. e40550 - e40550

Опубликована: Дек. 6, 2024

This study aims to investigate the effect of promethazine (PMZ) on hippocampal neuronal injury through network pharmacology and in vivo experiments. Network pharmacology: The intersection genes PMZ Alzheimer Disease (AD) were obtained, core AD screened. enriched by Gene Ontology Kyoto Encyclopedia Genes Genomes (KEGG) pathway enrichment analyses. In vitro experiment, mouse neurons (HT22) divided into control, glutamate (GLU) model, GLU + treatment groups. control group was given a complete culture medium, model for 24 hours, pretreatment 3 then administered hours. Cell viability determined, cell morphology observed microscopy, reactive oxygen species levels glutathione content detected, protein expression P53, PTGS2, SLC7A11, GPX4 detected western blotting. A total 317 targets, 1934 genes, 125 18 including P53 PTGS2. analysis showed that mainly related proliferation, inflammation, hypoxia, synaptic structure, plasma membrane, oxidoreductase activity. results neuroactive ligand-receptor interaction, senescence, cancer pathway, PI3K-AKT signal neurodegeneration, HIF-1 pathway. experiments: improved GLU-induced decrease morphological changes neurons. inhibited increased injured Up-regulated SLC7A11 expression, regulates SLC7A11-GPX4 antioxidant system protect from oxidative stress injury.

Язык: Английский

Процитировано

0