Identifying microglia-derived NFKBIA as a potential contributor to the pathogenesis of Alzheimer’s disease and age-related macular degeneration DOI
Shizhen Lei,

Yani Liu

Journal of Alzheimer s Disease, Год журнала: 2025, Номер unknown

Опубликована: Март 19, 2025

Background Alzheimer's disease (AD) and age-related macular degeneration (AMD) place considerable health burden on affected individuals significant economic society. Objective This study aims to explore the shared cellular molecular mechanisms underlying pathogenesis of AD AMD. Methods The investigation in this is conducted via single-cell bulk tissue transcriptomic analysis. Transcriptomic datasets AMD were obtained from GEO database. differentially expressed genes (DEGs) control AD- AMD-affected samples identified. Functional enrichment analysis for DEGs was subsequently performed. Then, protein-protein interaction (PPI) network these established STRING database hub identified by Cytoscape software. Single-cell performed using Seurat R package their expression different cell types. Results Differential 127 two diseases, including 71 upregulated 56 downregulated genes. Upregulated enriched inflammation, gliogenesis, apoptosis, response bacterial viral infection mitochondrial function energy production. PPI determined 10 genes, which NFKBIA gene associated with severity both Moreover, showed that highly microglia disease-affected tissues. Conclusions findings indicated high important contributors progression Microglia-derived might serve as a potential therapeutic target

Язык: Английский

Enhanced activation of retinal progenitor cells by zinc silicate bioceramics for effective functional restoration in retinal degenerative diseases DOI Creative Commons
Na Sun, Zhaowenbin Zhang, Junling Wang

и другие.

View, Год журнала: 2025, Номер unknown

Опубликована: Янв. 29, 2025

Abstract Retinal degenerative diseases, primarily owing to the gradual loss of neuronal cells, could lead blindness. Transplantation retinal progenitor cells (RPCs), which had unique ability differentiate into offered promising therapeutic potential for these diseases. However, a critical challenge in stem cell therapy was efficient promotion directed proliferation and differentiation cells. To overcome this limitation, we formulated zinc silicate bioceramic (ZS) composite hydrogel. The experimental outcomes demonstrated that novel material possessed substantial advantages immense possibilities mimicking extracellular matrix environment, thereby effectively modulating differentiation. Notably, it enhanced RPCs neurons while suppressing glial In particular, ZS an exceptional capacity induce photoreceptor specialized type cell. It important highlight hydrogel predominantly facilitated through activation Tiam2‐Rac signaling pathway. Concurrently, stimulated via AKT ERK pathways. Zn ions promoted RPCs, synergistic effect Si contributed reducing inflammatory factor expression promoting RPCs. conclusion, meticulous modulation RPC mediated by bioceramics held promise strategies diseases neurodegenerative disorders.

Язык: Английский

Процитировано

1

Identifying microglia-derived NFKBIA as a potential contributor to the pathogenesis of Alzheimer’s disease and age-related macular degeneration DOI
Shizhen Lei,

Yani Liu

Journal of Alzheimer s Disease, Год журнала: 2025, Номер unknown

Опубликована: Март 19, 2025

Background Alzheimer's disease (AD) and age-related macular degeneration (AMD) place considerable health burden on affected individuals significant economic society. Objective This study aims to explore the shared cellular molecular mechanisms underlying pathogenesis of AD AMD. Methods The investigation in this is conducted via single-cell bulk tissue transcriptomic analysis. Transcriptomic datasets AMD were obtained from GEO database. differentially expressed genes (DEGs) control AD- AMD-affected samples identified. Functional enrichment analysis for DEGs was subsequently performed. Then, protein-protein interaction (PPI) network these established STRING database hub identified by Cytoscape software. Single-cell performed using Seurat R package their expression different cell types. Results Differential 127 two diseases, including 71 upregulated 56 downregulated genes. Upregulated enriched inflammation, gliogenesis, apoptosis, response bacterial viral infection mitochondrial function energy production. PPI determined 10 genes, which NFKBIA gene associated with severity both Moreover, showed that highly microglia disease-affected tissues. Conclusions findings indicated high important contributors progression Microglia-derived might serve as a potential therapeutic target

Язык: Английский

Процитировано

0