Flavonoids regulating NLRP3 inflammasome: a promising approach in alleviating diabetic peripheral neuropathy DOI Creative Commons

Saumya Khanna,

Sachindra Kumar, Priyanka Sharma

и другие.

Inflammopharmacology, Год журнала: 2025, Номер unknown

Опубликована: Апрель 9, 2025

Abstract A common and serious side effect of diabetes is diabetic peripheral neuropathy (DPN), which characterised by gradual nerve damage brought on oxidative stress, chronic inflammation, prolonged hyperglycemia. Studies identify NLRP3 inflammasome as a key mediator in the pathogenesis DPN, connecting neuroinflammation neuronal to metabolic failure. Because their strong anti-inflammatory antioxidant qualities, flavonoids, broad class naturally occurring polyphenols, have drawn interest potential treatments for DPN. The various ways that flavonoids affect treatment DPN are examined this review. It has been demonstrated prevent activation, lowers release pro-inflammatory cytokines including IL-1β IL-18 causes neuroinflammation. Flavonoids work mechanistically reducing altering important signalling pathways, blocking activities NF-κB caspase-1, both essential activation inflammasome. Preclinical research shown neuroprotective benefits, few clinical evidence also points improve function lessen neuropathic pain patients. current review emphasises how may be used strategy target inflammation caused By targeting inflammatory provide new way slow progression debilitating illness. Further investigation into mechanisms, translation, novel drug delivery techniques could enhance therapeutic efficacy neuropathy. Graphical abstract

Язык: Английский

Deciphering the role of metal ion transport-related genes in T2D pathogenesis and immune cell infiltration via scRNA-seq and machine learning DOI Creative Commons
Zuhui Pu,

Tony Bowei Wang,

Ying Lü

и другие.

Frontiers in Immunology, Год журнала: 2025, Номер 15

Опубликована: Янв. 24, 2025

Introduction Type 2 diabetes (T2D) is a complex metabolic disorder with significant global health implications. Understanding the molecular mechanisms underlying T2D crucial for developing effective therapeutic strategies. This study employs single-cell RNA sequencing (scRNA-seq) and machine learning to explore pathogenesis of T2D, particular focus on immune cell infiltration. Methods We analyzed scRNA-seq data from islet cells nondiabetic (ND) patients, identifying differentially expressed genes (DEGs), especially those related metal ion transport (RMITRGs). employed 12 algorithms develop predictive models assessed infiltration using single-sample gene set enrichment analysis (ssGSEA). Correlations between key RMITRGs were investigated, interactions among these explored through protein-protein interaction (PPI) network analysis. Additionally, we performed detailed cell-cell communication identify signaling pathways in T2D. Results Our identified 1953 DEGs ND Stepglm[backward] plus GBM model demonstrating high accuracy 13 hub RMITRGs. Twelve protein structures predicted AlphaFold 3, revealing potential functional conformations. observed strong correlation cells, PPI revealed interactions. Cell-cell highlighted 16 active pathways, CXCL, MIF, COMPLEMENT linked inflammatory responses, WNT, KIT, LIFR, HGF uniquely activated Conclusion emphasizing transport, signaling, These findings suggest enhance management. The provide valuable insights into disease targets intervention.

Язык: Английский

Процитировано

0

Benefits of Liquid Dressings in Post-Operative Wound Dressing of diabetic foot ulcer DOI Creative Commons
Gang Wang, Yingxue Jiang,

Zhen-Nan Liu

и другие.

Current Problems in Surgery, Год журнала: 2025, Номер unknown, С. 101730 - 101730

Опубликована: Фев. 1, 2025

Язык: Английский

Процитировано

0

Effects of traditional Chinese exercise on diabetes with neuropathy: a systematic review and meta-analysis DOI Creative Commons

Junru Mao,

Anni Zhao,

Yiqing Cai

и другие.

Journal of Traditional Chinese Medical Sciences, Год журнала: 2025, Номер unknown

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

What does nerve ultrasound contribute to the evaluation of diabetic polyneuropathy over time? A prospective follow-up observational study of people with type 2 diabetes DOI
Bianka Heiling, Thomas Lehmann, N Müller

и другие.

Diabetes Research and Clinical Practice, Год журнала: 2025, Номер unknown, С. 112115 - 112115

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

Editorial: Understanding and managing diabetic neuropathy: current perspectives and future directions DOI Creative Commons
Paramita Basu, Pranav Prasoon, Keiichiro Susuki

и другие.

Frontiers in Neuroscience, Год журнала: 2025, Номер 19

Опубликована: Март 18, 2025

Diabetic neuropathy is a heterogeneous condition that encompasses spectrum of clinical and subclinical syndromes with varying anatomical distributions, courses, underlying pathogenetic mechanisms (Savelieff et al., 2024). The prevalence diabetic high, estimates suggesting up to 50% individuals diabetes may develop the impact on quality life, morbidity, healthcare costs significant, underscoring importance early detection effective management. This research topic, six original articles two review articles, aims provide an overview current understanding epidemiology, pathogenesis, diagnosis, management this condition, particular emphasis insights gained from studies.Understanding features crucial for better management, treatment condition. In institutional-based retrospective follow-up study design, Tilahun al. examined incidence predictors among type 2 mellitus (T2DM) patients in Addis Ababa hospitals over 10 years (Tilahun Of 414 T2DM participants, 23.4% developed neuropathy. Key included hypertension, anemia, age, low HDL, high creatinine, complications such as retinopathy nephropathy. average onset was 5 years, mean survival 7 post-diagnosis. associated wide variety complications. Zhang investigated changes brain functional connectivity (FC) patients, comparing those neuropathy, without, healthy control (Zhang 2024a). Statistical analyses identified group differences linked FC measures, showing reduced regions essential motion motor These findings suggest impair movement-related cognitive function, highlighting need comprehensive assessments personalized treatments enhance patient life. Fang explored relationship between sarcopenia older (Fang Mendelian randomization analysis suggested partial causality traits sarcopenia. addition, cross-sectional appendicular skeletal muscle mass index 5-time chair stand test were data highlight screening neuropathy.Understanding genetic, immunological, or metabolic factors development diagnostic therapeutic strategies. using bioinformatic identify differentially expressed genes (DEGs) human mouse samples 2024b). Researchers found several hub immune dysregulation revealing shared DEGs species. emphasized significance cell infiltration suggests these could new targets neuropathy.Chen circulating adiponectin leptin levels risk (Chen Logistic regression models used analyze 198 205 controls, we lower higher significantly increased adipokines serve potential biomarkers identifying at-risk individuals, emphasizing further into mechanisms. article, Sango use immortalized Schwann lines pathogenesis therapy (Sango highlights how hyperglycemia-related abnormalities cells contribute progression focusing stress responses. also discusses various pathogenic strategies aimed at alleviating effects neuropathy.Early diagnosis are mitigating adverse patient's well-being Liu nomogram model predict (Liu By analyzing 1185 researchers seven significant factors, including hip circumference, fasting plasma glucose, C-peptide, postprandial albumin, blood urea nitrogen.The demonstrated good predictive ability, providing practical tool clinicians high-risk early. outcomes settings, particularly cases where medical resources limited. Finally, Atmaca highlighted detection, accurate (Atmaca It greater physician awareness, first-contact clinicians, ensure timely intervention. A structured algorithm recommend evaluating prediabetes by incorporating laboratory testing neurological referrals atypical cases. advocated individualized approaches targeting specific rather than universal strategy. Alpha-lipoic acid promising first-line antioxidant its dual might help alleviate neuropathy-related conditions. However, gaps persist disease efficacy calls robust multicenter trials biomarker discovery enable more therapies emphasizes urgent improve diagnostics, targeted manage effectively.We hope reader will find Research Topic valuable reference which has long been subject intense focus. conclusion, offer innovative

Язык: Английский

Процитировано

0

Diabetic neuropathy in adolescents and young adults: The need for early detection and improved understanding DOI
Prashanth Vas,

Viviana Maggio,

Manfredi Rizzo

и другие.

Journal of Diabetes and its Complications, Год журнала: 2025, Номер unknown, С. 109018 - 109018

Опубликована: Март 1, 2025

Язык: Английский

Процитировано

0

The Therapeutic Potential of Dipeptidyl Peptidase 4 Inhibitors and Glucagon-Like Peptide-1 Receptor Agonists in Diabetic Peripheral Neuropathy DOI Creative Commons
Theodoros Panou, Evanthia Gouveri,

Djordje S Popovic

и другие.

Diabetes Therapy, Год журнала: 2025, Номер unknown

Опубликована: Март 28, 2025

Язык: Английский

Процитировано

0

Flavonoids regulating NLRP3 inflammasome: a promising approach in alleviating diabetic peripheral neuropathy DOI Creative Commons

Saumya Khanna,

Sachindra Kumar, Priyanka Sharma

и другие.

Inflammopharmacology, Год журнала: 2025, Номер unknown

Опубликована: Апрель 9, 2025

Abstract A common and serious side effect of diabetes is diabetic peripheral neuropathy (DPN), which characterised by gradual nerve damage brought on oxidative stress, chronic inflammation, prolonged hyperglycemia. Studies identify NLRP3 inflammasome as a key mediator in the pathogenesis DPN, connecting neuroinflammation neuronal to metabolic failure. Because their strong anti-inflammatory antioxidant qualities, flavonoids, broad class naturally occurring polyphenols, have drawn interest potential treatments for DPN. The various ways that flavonoids affect treatment DPN are examined this review. It has been demonstrated prevent activation, lowers release pro-inflammatory cytokines including IL-1β IL-18 causes neuroinflammation. Flavonoids work mechanistically reducing altering important signalling pathways, blocking activities NF-κB caspase-1, both essential activation inflammasome. Preclinical research shown neuroprotective benefits, few clinical evidence also points improve function lessen neuropathic pain patients. current review emphasises how may be used strategy target inflammation caused By targeting inflammatory provide new way slow progression debilitating illness. Further investigation into mechanisms, translation, novel drug delivery techniques could enhance therapeutic efficacy neuropathy. Graphical abstract

Язык: Английский

Процитировано

0