Reactive Oxygen Species in Cystic Kidney Disease DOI Creative Commons
Sanat Subhash,

Sonya Vijayvargiya,

Aetan Parmar

и другие.

Antioxidants, Год журнала: 2024, Номер 13(10), С. 1186 - 1186

Опубликована: Сен. 30, 2024

Polycystic kidney disease (PKD) is a rare but significant renal condition with major implications for global acute and chronic patient care. Oxidative stress reactive oxygen species (ROS) can significantly alter its pathophysiology, clinical outcomes, treatment, contributing to negative including hypertension, disease, failure. Inflammation from ROS existing cysts propagate the generation accumulation of ROS, exacerbating injury, pro-fibrotic signaling cascades, interstitial fibrosis. Early identification prevention oxidative contribute reduced cystic progression improved longitudinal outcomes. Increased research regarding biomarkers, pathophysiology stress, novel therapeutic interventions alongside creation comprehensive guidelines establishing methods assessment, monitoring, intervention in patients imperative standardize practice improve The integration artificial intelligence (AI), genetic editing, genome sequencing could further early detection management mitigate adverse In this review, we aim comprehensively assess multifactorial role analyzing treatment interventions, trials, animal models, future directions

Язык: Английский

Case Report: Nephrotic syndrome as the primary manifestation of Alport syndrome in a Chinese pediatric patient DOI Creative Commons
Yue Song, Yifei Li, Liqun Lu

и другие.

Frontiers in Pediatrics, Год журнала: 2025, Номер 12

Опубликована: Янв. 8, 2025

Alport syndrome (AS) is a genetically heterogeneous disorder resulting from variants in genes coding for the alpha-3/4/5 chains of Collagen IV, leading to defective basement membranes kidney, cochlea, and eye. The clinical manifestations AS vary patients. Cases childhood caused by COL4A3 presenting primarily with nephrotic (NS) are rarely reported. Here, we report pediatric case initially NS attributed COL4A3. An 11-year-old boy presented hematuria range proteinuria. After excluding secondary causes, primary was considered. He administered prednisone (60 mg/day). patient had not responded treatment end 4 weeks, so he diagnosed steroid-resistant NS. A renal biopsy showed granular vacuolar degeneration tubular epithelial cells, multifocal foam cell infiltration interstitium, immunofluorescence indicated absence α3, α4, α5 expression glomerular membrane, while Bowman's capsule normal. Electron microscopy ultrastructural suggested variable membrane thickness, partial tearing web-like structures. Genetic testing revealed heterozygous missense mutation c.3210 (exon 37)G>A(NM:000091). These findings consistent diagnosis AS. Prednisone gradually tapered enalapril maleate initiated. We have described featuring as its manifestation. It important consider be or differential patients who steroid resistance.

Язык: Английский

Процитировано

0

Stipulations of cell and gene therapy and the ties to biomanufacturing DOI Open Access

Justin Allisha,

J.J. Das,

Thomas Dunnigan

и другие.

Biotechnology Progress, Год журнала: 2025, Номер unknown

Опубликована: Янв. 23, 2025

Abstract Cell and gene therapy (CGT) products are emerging innovative biopharmaceuticals that hold promise for treating diseases otherwise beyond the scope of conventional medicines. The evolution CGT from a research idea to promising therapeutic product is due complementary advancements across various scientific disciplines. First, innovations in editing delivery technology have provided fundamental tools manipulate genes cells pursuits. Second, applied translational research, including how clinical trials designed, performed, evaluated, analyzed, transformed into potential product. Third, scaling up production been critical delivering preclinical studies, trials, approved treatments. In parallel, regulatory requirements continuously evolved, with lessons learned studies biomanufacturing. These combined efforts concept reality treat wide range diseases. However, continued R&D oversight crucial further improve safety, efficacy, accessibility products.

Язык: Английский

Процитировано

0

“ Advancing Nephrogenetics: Utilizing Single-Cell RNA Sequencing and Organoid Models for Precision Kidney Disease Treatment” DOI

Mohanapriya C.D.,

Praveen Kumar Kochuthakidiyel Suresh

Current Pediatrics Reports, Год журнала: 2025, Номер 13(1)

Опубликована: Март 17, 2025

Язык: Английский

Процитировано

0

Advances in CRISPR-Cas systems for kidney diseases DOI

Bhupendra Puri,

Yogesh A. Kulkarni, Anil Bhanudas Gaikwad

и другие.

Progress in molecular biology and translational science, Год журнала: 2024, Номер unknown, С. 149 - 162

Опубликована: Авг. 17, 2024

Язык: Английский

Процитировано

2

Gene Therapy and kidney diseases DOI Creative Commons

Nadia Tavakolidakhrabadi,

Wen Y. Ding, Moin A. Saleem

и другие.

Molecular Therapy — Methods & Clinical Development, Год журнала: 2024, Номер 32(4), С. 101333 - 101333

Опубликована: Сен. 6, 2024

Язык: Английский

Процитировано

2

Alpha-adducin 1 (rs4961) gene and its expression associated with sodium sensitivity in hypertensive patients: a cohort study in the western Ukrainian population DOI Creative Commons
Larysa Sydorchuk,

Bogdan Lytvyn,

Andrii Sydorchuk

и другие.

Endocrine Regulations, Год журнала: 2024, Номер 58(1), С. 195 - 205

Опубликована: Янв. 1, 2024

Abstract Objective. The aim of this study was to evaluate the association α-adducin-1 gene ( ADD1 ) (Gly460Trp [rs4961]) polymorphism and its expression in with renal dysfunction sodium sensitivity hypertensive patients western Ukrainian population. Methods. One-hundred essential arterial hypertension (EAH) hypertensive-mediated target organ damage (stage 2), moderate, high, very high cardiovascular risk were enrolled case-control study. Sixty healthy individuals assigned as controls. Sodium resistance determined by salt load reaction. (rs4961) genotyping performed RT-PCR. Results. quantitative trait loci (eQTL) (chr4:2906707 [hg19]) confirmed 37 tissues organs 23 phenotypic traits. Two hundred eQTL associations revealed - all cis-variants (cis-QTL); 73 methylation QTL (mQTL), 34 splicing (sQTL), 14 histone modification (hQTL), 2 protein (pQTL), transcript utilization (tuQTL), 4 incorporated long noncoding areas RNA (lncRNA). GG-genotype unreliably enhances EAH (OR=1.92; 95%CI: 0.90–4.10; p=0.066). observed 54.0% 20.0% controls (c2=17.89; p<0.001). T-allele carriers (1378G>T; rs4961) dominated 12-fold general (OR 2.24–64.29; p=0.001), women – 4.71 times 1.92–11.56; p<0.001), men 4.09 1.03–16.28; p=0.041). elevated likelihood severe twice (OR=2.19; OR 1.00–5.05; p=0.049). Conclusion. associates increases regardless gender. probability

Язык: Английский

Процитировано

1

Reactive Oxygen Species in Cystic Kidney Disease DOI Creative Commons
Sanat Subhash,

Sonya Vijayvargiya,

Aetan Parmar

и другие.

Antioxidants, Год журнала: 2024, Номер 13(10), С. 1186 - 1186

Опубликована: Сен. 30, 2024

Polycystic kidney disease (PKD) is a rare but significant renal condition with major implications for global acute and chronic patient care. Oxidative stress reactive oxygen species (ROS) can significantly alter its pathophysiology, clinical outcomes, treatment, contributing to negative including hypertension, disease, failure. Inflammation from ROS existing cysts propagate the generation accumulation of ROS, exacerbating injury, pro-fibrotic signaling cascades, interstitial fibrosis. Early identification prevention oxidative contribute reduced cystic progression improved longitudinal outcomes. Increased research regarding biomarkers, pathophysiology stress, novel therapeutic interventions alongside creation comprehensive guidelines establishing methods assessment, monitoring, intervention in patients imperative standardize practice improve The integration artificial intelligence (AI), genetic editing, genome sequencing could further early detection management mitigate adverse In this review, we aim comprehensively assess multifactorial role analyzing treatment interventions, trials, animal models, future directions

Язык: Английский

Процитировано

0