Ion channel expression in intrinsic cardiac neurons: new players in cardiac channelopathies? DOI Creative Commons

Jocelyn Bescond,

Jean‐François Faivre,

André Jean

и другие.

Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, Год журнала: 2025, Номер 1872(6), С. 119983 - 119983

Опубликована: Май 8, 2025

The autonomic nervous system is an important modulator of electrical disorders observed in cardiac pathologies through changes the balance between sympathetic and parasympathetic tone. final common pathway for neuronal control resides intrinsic (ICNS), composed intracardiac neurons (ICN), which allows sympathetic-parasympathetic efferent interactions at sites. ICNS a complex that plays crucial role regulation physiological parameters has been shown to contribute diseases, particular arrhythmias. It therefore understand molecular determinants, such as ion channels, excitability their potential modulation pathological conditions. This review discusses several channels expressed by ICN, including potassium (e.g., inward rectifier, calcium-dependent, voltage-activated, muscarinic-sensitive), voltage-gated sodium (VGSC), calcium (VGCC), hyperpolarization-activated cyclic nucleotide-gated (HCN) Transient Receptor Potential (TRP) Channels, involvement pathologies. We highlight need further research on ICN particularly under conditions, develop therapies

Язык: Английский

Catecholaminergic Polymorphic Ventricular Tachycardia: Advancing From Molecular Insights to Preclinical Models DOI Creative Commons
Luca Schneider, Merima Begovic, Xiaobo Zhou

и другие.

Journal of the American Heart Association, Год журнала: 2025, Номер unknown

Опубликована: Март 13, 2025

Inherited cardiac channelopathies are linked to a heightened risk of sudden death. Despite evolving knowledge on different genes for these inherited conditions, certain subtypes, such as catecholaminergic polymorphic ventricular tachycardia syndrome, the specific genetic causes remain unidentified. The research pathophysiological mechanisms underlying syndrome has been conducted through in vitro and vivo models, including genetically modified animal cardiac‐specific transgenic pharmacological interventions human‐induced pluripotent stem cell–derived cardiomyocytes 2‐ 3‐dimensional models. Recent predominantly utilizes cardiomyocytes, focusing genotype–phenotype correlations screening. integration cutting‐edge techniques clustered regularly interspaced short palindromic repeats/Cas9 genome editing 3‐dimensional–engineered heart tissues shed new light tachycardia, potentially enhancing drug therapies part personalized medicine approaches. This review emphasizes diverse insights gained from both studies along with application models various contexts.

Язык: Английский

Процитировано

0

Analysis of Factors Relevant to the Severity of Symptoms in Children and Adolescents with Postural Orthostatic Tachycardia Syndrome DOI Creative Commons
Yali Cao, Ping Liu, Bo Li

и другие.

Children, Год журнала: 2025, Номер 12(4), С. 474 - 474

Опубликована: Апрель 7, 2025

Objectives: The current study aims to investigate the factors associated with severity of conditions for pediatric cases postural orthostatic tachycardia syndrome (POTS). Methods: Patients hospitalized and first diagnosed POTS were retrospectively included reviewed. symptoms was evaluated by symptom scores (SSs). Multiple Spearman correlation analyses multiple linear regression used determine independently SS. divided into mild (SS ≤ P25) severe ≥ P75) groups test distinguishing efficiency candidate factors. each correlated factor in indicating condition children assessed receiver operating characteristic (ROC) curve. Results: A series 296 patients aged 5-17 years suffering from included. showed that corrected QT interval dispersion (QTcd) SS (p < 0.05). QTcd can be suggest symptoms, area under curve (AUC) 0.986 (95% CI 0.976-0.997). At a threshold = 45 ms, sensitivity specificity were, respectively, 94.0% 91.8% indication. Conclusions: In POTS, positively their exhibited strong indicative value. ms valid cut-off value severity.

Язык: Английский

Процитировано

0

Ion channel expression in intrinsic cardiac neurons: new players in cardiac channelopathies? DOI Creative Commons

Jocelyn Bescond,

Jean‐François Faivre,

André Jean

и другие.

Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, Год журнала: 2025, Номер 1872(6), С. 119983 - 119983

Опубликована: Май 8, 2025

The autonomic nervous system is an important modulator of electrical disorders observed in cardiac pathologies through changes the balance between sympathetic and parasympathetic tone. final common pathway for neuronal control resides intrinsic (ICNS), composed intracardiac neurons (ICN), which allows sympathetic-parasympathetic efferent interactions at sites. ICNS a complex that plays crucial role regulation physiological parameters has been shown to contribute diseases, particular arrhythmias. It therefore understand molecular determinants, such as ion channels, excitability their potential modulation pathological conditions. This review discusses several channels expressed by ICN, including potassium (e.g., inward rectifier, calcium-dependent, voltage-activated, muscarinic-sensitive), voltage-gated sodium (VGSC), calcium (VGCC), hyperpolarization-activated cyclic nucleotide-gated (HCN) Transient Receptor Potential (TRP) Channels, involvement pathologies. We highlight need further research on ICN particularly under conditions, develop therapies

Язык: Английский

Процитировано

0