The role of C1orf50 in breast cancer progression and prognosis DOI Creative Commons

Yusuke Otani,

Atsushi Tanaka, Masaki Maekawa

и другие.

Breast Cancer, Год журнала: 2024, Номер unknown

Опубликована: Ноя. 28, 2024

Abstract Although the prognosis of breast cancer has significantly improved compared to other types cancer, there are still some patients who expire due recurrence or metastasis. Therefore, it is necessary develop a method identify with poor at early stages cancer. In process discovering new prognostic markers from genes unknown function, we found that expression C1orf50 determines patients, especially for those Luminal A This study aims elucidate molecular role in progression. Bioinformatic analyses dataset TCGA, and vitro analyses, reveal pathways influenced by expression. knockdown suppressed cell cycle cells weakened their ability maintain undifferentiated state self-renewal capacity. Interestingly, upregulation increased sensitivity CDK4/6 inhibition. addition, was be more abundant than normal epithelium, suggesting C1orf50’s involvement pathogenesis. Furthermore, mRNA level positively correlated PD-L1 its related factors. These results suggest promotes progression through upregulation, maintenance stemness, immune evasion mechanisms. Our uncovers biological functions progression, finding not previously reported any type

Язык: Английский

Unraveling the peripheral nervous System's role in tumor: A Double-edged Sword DOI
Yanhao Wang, Yang Xuan,

Cuicui Liu

и другие.

Cancer Letters, Год журнала: 2025, Номер 611, С. 217451 - 217451

Опубликована: Янв. 8, 2025

Язык: Английский

Процитировано

2

Adrenergic microenvironment driven by cancer‐associated Schwann cells contributes to chemoresistance in patients with lung cancer DOI Creative Commons

Yusuke Otani,

Haruyoshi Katayama,

Yidan Zhu

и другие.

Cancer Science, Год журнала: 2024, Номер 115(7), С. 2333 - 2345

Опубликована: Апрель 27, 2024

Doublecortin (DCX)-positive neural progenitor-like cells are purported components of the cancer microenvironment. The number DCX-positive in tissues reportedly correlates with progression; however, little is known about mechanism by which these affect progression. Here we demonstrated that cells, found all major histological subtypes lung cancer, cancer-associated Schwann (CAS) and contribute to chemoresistance establishing an adrenergic Mechanistically, activation Hippo transducer YAP/TAZ was involved acquisition new traits CAS DCX positivity. We further revealed express catecholamine-synthesizing enzymes synthesize adrenaline, potentiates through YAP/TAZ. Our findings shed light on CAS, drive formation microenvironment reciprocal regulation tissues.

Язык: Английский

Процитировано

10

Sympathetic nerve signaling rewires the tumor microenvironment: a shift in “microenvironmental-ity” DOI Creative Commons
Ariana Sattler, Tetiana Korzun, Kasmira Gupta

и другие.

Cancer and Metastasis Reviews, Год журнала: 2025, Номер 44(1)

Опубликована: Янв. 20, 2025

Nerve signaling within the tumor microenvironment (TME) plays a critical role in initiation, progression, and metastasis of solid tumors. Due to their highly responsive behavior activation upon injury cancer onset, this review specifically focuses on how sympathetic nerves rewire TME. Within tumors, closely interact with various TME components, combined often shifts tumor-intrinsic physiology toward tumor-supportive phenotypes. In turn, such as myeloid cells, lymphoid extracellular matrix (ECM), endothelial associated fibroblasts (CAFs), Schwann secrete neurotrophic axon guidance factors that influence both outgrowth cell behavior, further exacerbating progression metastasis. Here, we current evidence multidirectional impacts immune non-immune nature these communication processes, exploring interactions may inform future therapeutics impair

Язык: Английский

Процитировано

0

Changes in Adrenoceptor Expression Level Contribute to the Cellular Plasticity of Glioblastoma Cells DOI Creative Commons

Youko Asaka,

Toshio Masumoto, Atsuhito Uneda

и другие.

The Journal of Physiological Sciences, Год журнала: 2025, Номер unknown, С. 100016 - 100016

Опубликована: Март 1, 2025

Glioblastoma cells are known to regulate their cellular plasticity in response surrounding microenvironment, but it is not fully understood what factors contribute the cells' changing plasticity. Here, we found that glioblastoma alter expression level of adrenoreceptors depending on differentiation stage. Catecholamines abundant central nervous system, and noradrenaline, particular, enhances stemness promotes dedifferentiation potential already differentiated cells. Antagonist RNAi experiments revealed signaling through α1D-adrenoreceptor important for noradrenaline action We also high was associated with poor prognosis patients gliomas. These data suggest increase own tumor microenvironment favorably survival. believe our findings will development new therapeutic strategies glioblastoma.

Язык: Английский

Процитировано

0

Intersection between lung cancer and neuroscience: opportunities and challenges DOI
Xiang Xiao, Lingli Huang, Ming Li

и другие.

Cancer Letters, Год журнала: 2025, Номер unknown, С. 217701 - 217701

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

Tumor-associated Schwann cells as new therapeutic target in non-neurological cancers DOI
Leyi Huang, Qi Ge, Guangyao Chen

и другие.

Cancer Letters, Год журнала: 2025, Номер unknown, С. 217748 - 217748

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

Interplay Between Schwann Cells and Peripheral Cancers: Mechanisms and Therapeutic Targets in Cancer Progression DOI
Zhini He, Fei‐Fei Liu, Lin Lin

и другие.

Glia, Год журнала: 2025, Номер unknown

Опубликована: Май 10, 2025

ABSTRACT Cancer, a leading global health concern, is characterized by uncontrolled proliferation of cells, high invasion into surrounding tissues, and eventual metastasis to distant organs. The complexity cancer further amplified diverse cellular components within the tumor microenvironment (TME), encompassing both cancerous non‐cancerous cells that fuel tumorigenesis progression. Schwann (SCs), main glial peripheral nervous system, have emerged as crucial TME in development. Here, we summarize multifaceted roles SCs growth, epithelial‐mesenchymal transition, perineural invasion, chemotherapy resistance. This review focuses on effects eight distinct types, particularly pancreatic, lung, colorectal cancers, along with cancer‐related pain, one most common symptoms affect quality life prognosis patients. Furthermore, emphasize therapeutic potential delving advanced technologies clinical strategies related SCs, which make us advocate for research elucidate events molecular mechanisms underlying SC‐cancer relationship. Translating these insights applications may offer new hope improved management patient outcomes.

Язык: Английский

Процитировано

0

The Role of C1orf50 in Breast Cancer Progression and Prognosis DOI Creative Commons

Yusuke Otani,

Atsushi Tanaka, Masaki Maekawa

и другие.

Research Square (Research Square), Год журнала: 2024, Номер unknown

Опубликована: Авг. 9, 2024

Abstract Although the prognosis of breast cancer has significantly improved compared to other types cancer, there are still some patients who expire due recurrence or metastasis. Therefore, it is necessary develop a method identify with poor at early stages cancer. In process discovering new prognostic markers from genes unknown function, we found that expression C1orf50 determines patients, especially for those Luminal A This study aims elucidate molecular role in progression. Bioinformatic analyses dataset TCGA, and vitro analyses, reveal pathways influenced by expression. knockdown suppressed cell cycle cells weakened their ability maintain undifferentiated state self-renewal capacity. Interestingly, upregulation increased sensitivity CDK4/6 inhibition. addition, was be more abundant than normal epithelium, suggesting involvement pathogenesis. Furthermore, mRNA level positively correlated PD-L1 its related factors. These results suggest promotes progression through upregulation, maintenance stemness immune evasion mechanisms. Our uncovers biological functions progression, finding not previously reported any type

Язык: Английский

Процитировано

0

What a wonderful world! DOI Open Access
Claire Magnon

Genes & Development, Год журнала: 2024, Номер 38(17-20), С. 802 - 804

Опубликована: Сен. 1, 2024

The world of cancer science is moving toward a paradigm shift in making connections with neuroscience. After decades research on genetic instability and mutations or the tumor microenvironment, emerging evidence suggests that malignant able to hijack use brain its network peripheral central neurons as disrupters homeostasis body. Whole-body requires brain-body circuits maintain survival health via processes interoception, immunoception, nociception. It now likely disturbs physiological communication bidirectional connections.

Язык: Английский

Процитировано

0

The role of C1orf50 in breast cancer progression and prognosis DOI Creative Commons

Yusuke Otani,

Atsushi Tanaka, Masaki Maekawa

и другие.

Breast Cancer, Год журнала: 2024, Номер unknown

Опубликована: Ноя. 28, 2024

Abstract Although the prognosis of breast cancer has significantly improved compared to other types cancer, there are still some patients who expire due recurrence or metastasis. Therefore, it is necessary develop a method identify with poor at early stages cancer. In process discovering new prognostic markers from genes unknown function, we found that expression C1orf50 determines patients, especially for those Luminal A This study aims elucidate molecular role in progression. Bioinformatic analyses dataset TCGA, and vitro analyses, reveal pathways influenced by expression. knockdown suppressed cell cycle cells weakened their ability maintain undifferentiated state self-renewal capacity. Interestingly, upregulation increased sensitivity CDK4/6 inhibition. addition, was be more abundant than normal epithelium, suggesting C1orf50’s involvement pathogenesis. Furthermore, mRNA level positively correlated PD-L1 its related factors. These results suggest promotes progression through upregulation, maintenance stemness, immune evasion mechanisms. Our uncovers biological functions progression, finding not previously reported any type

Язык: Английский

Процитировано

0