TRIM8 as a predictor for prognosis in childhood acute lymphoblastic leukemia based on a signature of neutrophil extracellular traps DOI Creative Commons

Waihin Tin,

Cuilan Xiao,

Kexin Sun

и другие.

Frontiers in Oncology, Год журнала: 2024, Номер 14

Опубликована: Авг. 19, 2024

Background Neutrophil extracellular traps (NETs) can be attributed to the metastasis, occurrence, and immune evasion of cancer cells. We investigated prognostic value NET-related genes in childhood acute lymphoblastic leukemia (cALL) patients. Methods Differential gene expression analysis was conducted on samples collected from public databases. Grouping them based level genes, we assessed correlation between cell types risk score for having a poor prognosis cALL, with an evaluation sensitivity drugs used cALL. further divided groups, integrating survival data. Subsequently, methods including multivariable Cox algorithms, least absolute shrinkage selection operator (LASSO), univariable were utilized create model predicting prognosis. Experiments lines animals performed explore functions TRIM8, selected by model. To validate role TRIM8 development, lentivirus-mediated overexpression or knockdown employed mice T-ALL B-ALL. Results Kaplan–Meier (KM) underscored importance differentially expressed identified groups participated NETs, enrichment showing mechanism. Correlation revealed significant associations B cells, NK mast T plasma dendritic monocytes. The IC 50 values such as all-trans-retinoic acid (ATRA), axitinib, doxorubicin, methotrexate, sorafenib, vinblastine increased, while dasatinib exhibited lower . A total 13 constructing In training, testing, merged cohorts, KM demonstrated significantly improved low-risk cALL patients compared high-risk ( p < 0.001). area under curve (AUC) indicated strong predictive performance. Jurkat SUP-B15 that decreased proliferation lines. Further experiments more favorable TRIM8-knockdown showed better outcomes when knocked down. Conclusion novelty could aid development personalized treatments Furthermore, it is contributing factor cells worsens

Язык: Английский

Single-cell RNA-sequencing and genome-wide Mendelian randomisation along with abundant machine learning methods identify a novel B cells signature in gastric cancer DOI Creative Commons

Qi Ma,

Jie Gao, Yuan Hui

и другие.

Discover Oncology, Год журнала: 2025, Номер 16(1)

Опубликована: Янв. 6, 2025

Gastric cancer (GC) has a poor prognosis, considerable cellular heterogeneity, and ranks fifth among malignant tumours. Understanding the tumour microenvironment (TME) intra-tumor heterogeneity (ITH) may lead to development of novel GC treatments. The single-cell RNA sequencing (scRNA-seq) dataset was obtained from Gene Expression Omnibus (GEO) database, where diverse immune cells were isolated re-annotated based on cell markers established in original study ascertain their individual characteristics. We conducted weighted gene co-expression network analysis (WGCNA) identify genes with significant correlation GC. Utilising bulk data, we employed machine learning integration methods train specific biomarkers for diagnostic combinations. A two-sample Mendelian randomisation performed investigate causal effect gastric (GC). Ultimately, utilised DSigDB database acquire associations between signature pharmaceuticals. 18 that made up as follows: ZFAND2A, PBX4, RAMP2, NNMT, RNASE1, CD93, CDH5, NFKBIE, VWF, DAB2, FAAH2, VAT1, MRAS, TSPAN4, EPAS1, AFAP1L1, DNM3. Patients categorised into high-risk low-risk groups according risk scores. Individuals cohort exhibited dismal outlook. demonstrated individuals genetic predisposition elevated NFKBIE levels heightened likelihood acquiring Molecular docking indicates gemcitabine chloropyramine serve effective therapeutics against NFKBIE. developed validated utilising scRNA-seq data patients. function biomarker therapeutic target

Язык: Английский

Процитировано

0

Construction of a TAN-associated risk score model with integrated multi-omics data analysis and clinical validation in gastric cancer DOI

Zhangdi Xu,

Lan Zhang, Xiaping Wang

и другие.

Life Sciences, Год журнала: 2024, Номер 349, С. 122731 - 122731

Опубликована: Май 21, 2024

Язык: Английский

Процитировано

2

A prognostic model based on CLEC6A predicts clinical outcome of breast cancer patients DOI
Li Chen, Deenraj Kush Dhoomun, Qiang Liu

и другие.

International Immunopharmacology, Год журнала: 2024, Номер 137, С. 112411 - 112411

Опубликована: Июнь 12, 2024

Язык: Английский

Процитировано

2

A novel platelets-related gene signature for predicting prognosis, immune features and drug sensitivity in gastric cancer DOI Creative Commons
Qun Li, Cheng Zhang,

Yulin Ren

и другие.

Frontiers in Immunology, Год журнала: 2024, Номер 15

Опубликована: Ноя. 13, 2024

Background Platelets can dynamically regulate tumor development and progression. Nevertheless, research on the predictive value specific roles of platelets in gastric cancer (GC) is limited. This aims to establish a platelets-related gene signature GC with prognostic therapeutic implications. Methods We downloaded transcriptome data clinical materials patients (n=378) from The Cancer Genome Atlas (TCGA) database. Prognostic genes screened by univariate Cox regression were included Least Absolute Shrinkage Selection Operator (LASSO) analysis construct risk model. Kaplan-Meier curves receiver operating characteristic (ROCs) performed TCGA cohort three independent validation cohorts. A nomogram integrating score clinicopathological features was constructed. Functional enrichment microenvironment (TME) analyses performed. Drug sensitivity prediction conducted through Therapeutics Response Portal (CTRP) Finally, expression ten validated quantitative real-time PCR (qRT-PCR). Results ten-gene ( SERPINE1 , ANXA5 DGKQ PTPN6 F5 DGKB PCDH7 GNG11 APOA1 TF ) model finally Patients categorized as high- or low-risk using median threshold. area under ROC curve (AUC) values for 1-, 2-, 3-year overall survival (OS) training 0.670, 0.695, 0.707, respectively. Survival showed better OS AUCs predicting 0.708, 0.763, 0.742, TME revealed higher M2 macrophage infiltration an immunosuppressive high-risk group. Furthermore, High-risk tended be more sensitive thalidomide, MK-0752, BRD-K17060750. Conclusion novel we identified could used prognosis treatment GC.

Язык: Английский

Процитировано

0

Prognostic impact and immunotherapeutic implications of NETosis-related prognostic model in clear cell renal cell carcinoma DOI Creative Commons

Xingjun Mao,

Wen Huang,

Qing Xue

и другие.

Research Square (Research Square), Год журнала: 2024, Номер unknown

Опубликована: Март 15, 2024

Abstract Background The ramifications of necroptosis on the prognostication clear cell renal carcinoma (ccRCC) remain inadequately expounded. Methods A prognostic model delineating facets in ccRCC was constructed, employing a compendium algorithms. External validation effectuated using E-MTAB-1980 dataset. exploration immune infiltration scores undertaken through exploitation multiple Single-gene RNA sequencing data were procured from GSE171306 Real-time quantitative PCR (RT-qPCR) engaged to scrutinize differential expression SLC25A37 across cancer and paracancer tissues, as well diverse lines. Assessments proliferative metastatic alterations 769-P 786-O cells accomplished Cell Counting Kit-8 (CCK8) wound healing assays. Results necroptosis-related signature (NRS) emerges discerning metric, patients' attributes, tumor mutation burden, immunotherapy response, drug susceptibility. analysis unveils marked enrichment cells. Concurrently, RT-qPCR discloses overexpression both tissues knockdown mitigates propensities cells, evidenced by CCK8 Conclusion NRS assumes pivotal role ascertaining prognosis, susceptibility, features patients. putative player immunosuppressive microenvironments, thereby providing prospective avenue for design innovative immunotherapeutic targets ccRCC.

Язык: Английский

Процитировано

0

Prognostic impact and immunotherapeutic implications of NETosis-related prognostic model in clear cell renal cell carcinoma DOI Creative Commons

Xingjun Mao,

Wen Huang,

Qing Xue

и другие.

Journal of Cancer Research and Clinical Oncology, Год журнала: 2024, Номер 150(5)

Опубликована: Май 27, 2024

Abstract Background The ramifications of necroptosis on the prognostication clear cell renal carcinoma (ccRCC) remain inadequately expounded. Methods A prognostic model delineating facets in ccRCC was constructed, employing a compendium algorithms. External validation effectuated using E-MTAB-1980 dataset. exploration immune infiltration scores undertaken through exploitation multiple Single-cell RNA sequencing data were procured from GSE171306 Real-time quantitative PCR (RT-qPCR) engaged to scrutinize differential expression SLC25A37 across cancer and paracancer tissues, as well diverse lines. Assessments proliferative metastatic alterations 769-P 786-O cells accomplished Cell Counting Kit-8 (CCK8) wound healing assays. Results necroptosis-related signature (NRS) emerges discerning metric, patients’ attributes, tumor mutation burden, immunotherapy response, drug susceptibility. analysis unveils marked enrichment cells. Concurrently, RT-qPCR discloses overexpression both tissues knockdown mitigates propensities cells, evidenced by CCK8 Conclusion NRS assumes pivotal role ascertaining prognosis, susceptibility, features patients. putative player immunosuppressive microenvironments, thereby providing prospective avenue for design innovative immunotherapeutic targets ccRCC.

Язык: Английский

Процитировано

0

TRIM8 as a predictor for prognosis in childhood acute lymphoblastic leukemia based on a signature of neutrophil extracellular traps DOI Creative Commons

Waihin Tin,

Cuilan Xiao,

Kexin Sun

и другие.

Frontiers in Oncology, Год журнала: 2024, Номер 14

Опубликована: Авг. 19, 2024

Background Neutrophil extracellular traps (NETs) can be attributed to the metastasis, occurrence, and immune evasion of cancer cells. We investigated prognostic value NET-related genes in childhood acute lymphoblastic leukemia (cALL) patients. Methods Differential gene expression analysis was conducted on samples collected from public databases. Grouping them based level genes, we assessed correlation between cell types risk score for having a poor prognosis cALL, with an evaluation sensitivity drugs used cALL. further divided groups, integrating survival data. Subsequently, methods including multivariable Cox algorithms, least absolute shrinkage selection operator (LASSO), univariable were utilized create model predicting prognosis. Experiments lines animals performed explore functions TRIM8, selected by model. To validate role TRIM8 development, lentivirus-mediated overexpression or knockdown employed mice T-ALL B-ALL. Results Kaplan–Meier (KM) underscored importance differentially expressed identified groups participated NETs, enrichment showing mechanism. Correlation revealed significant associations B cells, NK mast T plasma dendritic monocytes. The IC 50 values such as all-trans-retinoic acid (ATRA), axitinib, doxorubicin, methotrexate, sorafenib, vinblastine increased, while dasatinib exhibited lower . A total 13 constructing In training, testing, merged cohorts, KM demonstrated significantly improved low-risk cALL patients compared high-risk ( p < 0.001). area under curve (AUC) indicated strong predictive performance. Jurkat SUP-B15 that decreased proliferation lines. Further experiments more favorable TRIM8-knockdown showed better outcomes when knocked down. Conclusion novelty could aid development personalized treatments Furthermore, it is contributing factor cells worsens

Язык: Английский

Процитировано

0