An IL-17-DUOX2 axis controls gastrointestinal colonization byCandida albicans DOI Open Access
Pallavi Kakade, Juan F. Burgueño, Shabnam Sircaik

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Авг. 19, 2024

Candida albicans is a ubiquitous fungus in the human gut microbiome as well prevalent cause of opportunistic mucosal and systemic disease. There currently little understanding, however, to how crosstalk between C. host regulates colonization this key niche. Here, we performed expression profiling on ileal colonic tissues germ-free mice colonized with define global response fungus. We reveal that Duox2 Duoxa2 , encoding dual NADPH oxidase activity, are upregulated both ileum colon, induction requires yeast-hyphal transition hyphal-specific toxin candidalysin. Hosts lacking IL-17 receptor failed upregulate Duox2/Duoxa2 while addition IL-17A colonoids induced these genes together concomitant production hydrogen peroxide. To directly role fungal colonization, antibiotic-treated intestinal DUOX2 activity were evaluated for responses. Surprisingly, loss function reduced at extended time points (>17 days colonization) increased proportion hyphal cells gut. levels also elevated -colonized functional highlighting cross-regulation cytokine DUOX2. Together, experiments novel links cells, candidalysin IL-17-DUOX2 axis, complex interplay factors filamentation

Язык: Английский

Research Progress of FIBCD1 Mediated Host-Related Biological Functions by Recognizing Chitin DOI Open Access

佳佳 于

Journal of Clinical Personalized Medicine, Год журнала: 2025, Номер 04(02), С. 972 - 980

Опубликована: Янв. 1, 2025

Язык: Английский

Процитировано

0

Human immunity to fungal infections DOI
Donald C. Vinh

The Journal of Experimental Medicine, Год журнала: 2025, Номер 222(6)

Опубликована: Апрель 15, 2025

Fungi increasingly threaten health globally. Mycoses range from life-threatening, often iatrogenic conditions, to enigmatic syndromes occurring without apparent immunosuppression. Despite some recent advances in antifungal drug development, complementary therapeutic strategies are essential for addressing these opportunistic pathogens. One promising avenue is leveraging host immunity combat fungal infections; this necessitates deeper understanding of the molecular immunology human susceptibility differentiate beneficial versus harmful immunopathological responses. Investigating models diseases natural settings, particularly through genetic immunodeficiencies and ethnographic-specific vulnerabilities, reveals crucial immune pathways fighting various yeasts molds. This review highlights diversity intrinsic across individuals populations, genetic- autoantibody-mediated processes, complementing previous principles learned animal studies contexts. Improved will facilitate development host-directed immunotherapies targeted public interventions, paving way precision medicine disease management.

Язык: Английский

Процитировано

0

Full Circle: When HSFs bring the heat—mapping the transcriptional circuitries of HSF-type regulators in Candida albicans DOI Creative Commons
Sadri Znaidi

mSphere, Год журнала: 2024, Номер unknown

Опубликована: Дек. 20, 2024

ABSTRACT Heat shock factor (HSF)-type regulators are stress-responsive transcription factors widely distributed among eukaryotes, including fungi. They carry a four-stranded winged helix-turn-helix DNA-binding domain considered as the signature for HSFs. The genome of opportunistic yeast Candida albicans encodes four HSF members, namely, Sfl1, Sfl2, Skn7, and essential regulator, Hsf1. C. HSFs do not only respond to heat and/or temperature variation but also CO 2 levels, oxidative stress, quorum sensing, acting this way central decision makers. In minireview, I follow on heels my mSphere Influence commentary (2020) provide an overview repertoire in Saccharomyces cerevisiae describe how their genetic perturbation , coupled with genome-wide expression location analyses, allow map transcriptional circuitry. highlight they can regulate, common, crucial developmental program: filamentous growth.

Язык: Английский

Процитировано

1

Efficacy of Limosilactobacillus fermentum in the management of vulvovaginal candidiasis: comparative analysis with topical miconazole in a single-blind randomized clinical trial DOI Creative Commons
Marco Pane, Emanuele Chisari

Frontiers in Microbiology, Год журнала: 2024, Номер 15

Опубликована: Авг. 1, 2024

Vulvovaginal candidiasis (VVC) significantly impacts women's quality of life and often shows a high recurrence rate despite conventional antifungal therapies. This study evaluates the efficacy

Язык: Английский

Процитировано

0

An IL-17-DUOX2 axis controls gastrointestinal colonization byCandida albicans DOI Open Access
Pallavi Kakade, Juan F. Burgueño, Shabnam Sircaik

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Авг. 19, 2024

Candida albicans is a ubiquitous fungus in the human gut microbiome as well prevalent cause of opportunistic mucosal and systemic disease. There currently little understanding, however, to how crosstalk between C. host regulates colonization this key niche. Here, we performed expression profiling on ileal colonic tissues germ-free mice colonized with define global response fungus. We reveal that Duox2 Duoxa2 , encoding dual NADPH oxidase activity, are upregulated both ileum colon, induction requires yeast-hyphal transition hyphal-specific toxin candidalysin. Hosts lacking IL-17 receptor failed upregulate Duox2/Duoxa2 while addition IL-17A colonoids induced these genes together concomitant production hydrogen peroxide. To directly role fungal colonization, antibiotic-treated intestinal DUOX2 activity were evaluated for responses. Surprisingly, loss function reduced at extended time points (>17 days colonization) increased proportion hyphal cells gut. levels also elevated -colonized functional highlighting cross-regulation cytokine DUOX2. Together, experiments novel links cells, candidalysin IL-17-DUOX2 axis, complex interplay factors filamentation

Язык: Английский

Процитировано

0