The mechanisms of resistance, epidemiological characteristics, and molecular evolution of carbapenem-resistant hypervirulent Klebsiella pneumoniae DOI
Qun Wang,

M M Ye,

Chi Hong

и другие.

Laboratory Medicine, Год журнала: 2024, Номер unknown

Опубликована: Дек. 23, 2024

Carbapenem-resistant hypervirulent Klebsiella pneumoniae (CR-hvKP) is a highly pathogenic, drug-resistant, and transmissible "superbug" that causes infections in hospitals communities. Because of the lack effective antimicrobial treatment options, morbidity mortality from CR-hvKP have increased dramatically, outbreaks rapid spread become major global public health challenge. The mechanisms molecular evolution include acquisition plasmid encoding virulence gene by carbapenemase-producing K pneumoniae, horizontal transfer plasmids carrying carbapenem resistance genes to hvKP, fusion both classic pneumoniae. In addition, hvKP can develop phenotype under antibiotic pressure. arises through plasmid-mediated convergence factors. Its multidrug lethal pathogenicity fuel hospital outbreaks, requiring urgent action block transmission strengthen surveillance contain this evolving superbug. article, we summarized carbapenemase mechanism, factors, epidemiology CR-hvKP. Our aim was elucidate evolutionary mechanism provide reference for curbing

Язык: Английский

Mobile genetic elements encoding antibiotic resistance genes and virulence genes in Klebsiella pneumoniae: important pathways for the acquisition of virulence and resistance DOI Creative Commons

Bin Han,

Chunlin Feng,

Jiang Yuan

и другие.

Frontiers in Microbiology, Год журнала: 2025, Номер 16

Опубликована: Фев. 24, 2025

Klebsiella pneumoniae is an opportunistic pathogen primarily associated with nosocomial infections, characterized by a propensity for multi-drug resistance and the potential evolution into hypervirulent strains. Based on its phenotypic genotypic characteristics, K. can be classified two types: classical (cKP) (hvKP). The spread of mobile genetic elements (MGEs) in has led to emergence carbapenem-resistant (CRKP) (CR-hvKP). CR-hvKP particularly concerning due multidrug resistance, high pathogenicity, increased transmissibility. This review summarizes types MGEs present pneumoniae, mechanisms horizontal gene transfer (HGT) mediated these elements, their roles dissemination antibiotic genes (ARGs) virulence genes, relationships among that resemble Russian dolls or exhibit hybrid characteristics. Additionally, clinical treatment epidemiological characteristics are discussed. Given variability transmissibility MGEs, continuous monitoring control variation transmission such material should prioritized.

Язык: Английский

Процитировано

0

High-risk clones of carbapenem resistant Klebsiella pneumoniae recovered from pediatric patients in Southern Brazil DOI
Damaris Krul, Luiza Souza Rodrigues, Adriele Celine Siqueira

и другие.

Brazilian Journal of Microbiology, Год журнала: 2024, Номер 55(2), С. 1437 - 1443

Опубликована: Март 19, 2024

Язык: Английский

Процитировано

2

Carbapenem-resistant Klebsiella pneumoniae among hospitalized patients in Cape Town, South Africa: molecular epidemiology and characterization DOI Creative Commons
Gert Marais, Clinton Moodley, Shantelle Claassen-Weitz

и другие.

JAC-Antimicrobial Resistance, Год журнала: 2024, Номер 6(2)

Опубликована: Март 5, 2024

Abstract Background The molecular epidemiology of carbapenem-resistant Enterobacterales in Cape Town remains largely unknown. Objectives This study aimed to describe the epidemiology, resistome, virulome and mobilome Klebsiella pneumoniae (CRKP) within guide therapy, antimicrobial stewardship infection prevention control practices. Methods Eighty-five CRKP isolates from hospitalized patients underwent WGS as part a prospective, multicentre, cross-sectional study, conducted between 1 November 2020 30 2022, across public-sector private-sector hospitals Town, South Africa. Results MLST revealed three novel types, ST6785, ST6786 ST6787, while most common were ST219, ST307, ST17, ST13 ST2497. Different predominant clones noted each hospital. carbapenemase gene was blaOXA-48-like, detected 71% isolates, with blaNDM 5%. Notably, co-detection two genes (blaOXA-48-like blaNDM) occurred 13% isolates. yersiniabactin siderophore 73% commonly associated ICEKp5 mobile element. All carbapenemases located on plasmids. blaOXA-181 blaOXA-232 colocalized ColKP3 replicon type assembled contigs 83% 100% cases, respectively. Conclusions reflects institutionally dominant, rather than regional, clones. prevalent keeping Africa general. Emerging harbouring both blaOXA-48-like blaNDM, such ST2497 are concern due limited availability appropriate agents

Язык: Английский

Процитировано

1

Computational Systems and Network Biology Perspective: Understanding Klebsiella pneumoniae Infection Mechanisms DOI Creative Commons
Maulida Mazaya, Novaria Sari Dewi Panjaitan,

Anis Kamilah Hayati

и другие.

The Microbe, Год журнала: 2024, Номер unknown, С. 100175 - 100175

Опубликована: Сен. 1, 2024

Язык: Английский

Процитировано

1

Klebsiella pneumoniae species complex: From wastewater to the environment DOI Creative Commons

Ilse Verburg,

Lucía Hernández Leal,

K. Waar

и другие.

One Health, Год журнала: 2024, Номер 19, С. 100880 - 100880

Опубликована: Авг. 17, 2024

Язык: Английский

Процитировано

0

Emergence of mcr-8.1 -bearing MDR-hypervirulent Klebsiella pneumoniae ST307 DOI Creative Commons
Jie Sheng, Rory Cave,

Mary M. Ter-Stepanyan

и другие.

Microbiology Spectrum, Год журнала: 2024, Номер unknown

Опубликована: Дек. 13, 2024

ABSTRACT We report for the first time whole-genome sequencing of four multidrug-resistant sequence type (ST) 307 Klebsiella pneumoniae recovered from patients in two hospitals Armenia. Comparative genomic analysis revealed that isolates were closely related, with a maximum 39 single nucleotide polymorphism (SNP) differences core genome. All Armenian carried integrative and conjugative element ICE Kp 4, which bears yersiniabactin locus, shared common evolutionary origin, diverging around 2005 (95% CI: 1999 to 2011). Antibiotic susceptibility testing showed resistance several antibiotics, including ampicillin, amoxicillin-clavulanic acid, cefepime, ceftazidime, norfloxacin, levofloxacin, chloramphenicol. Specifically, designated as ARM03 ARM06 resistant piperacillin-tazobactam, ARM04 ARM05 had intermediate both piperacillin-tazobactam imipenem, amikacin. further identified antimicrobial (AMR) genes isolates, bla OXA-1 , TEM-1D SHV-28 dfrA14 tet(A ), sul2 qnrB1 aac(6´)-Ib-cr strA strB extended-spectrum β-lactamase gene CTX-M-15 . Additionally, also obtained dfrA5 sul1 sul3 cmlA1 mphA aph3-Ia unique colistin mcr-8.1 was absent all other publicly available ST307 isolates. These acquired aerobactin siderophore-encoding clusters ( iucABCD-iutA ) hypermucoidy locus rmpADC (ARM06 rmpA fragment). ARM05, well ARM06, nearly identical AMR virulence genes, along similar plasmid replicon profiles, respectively. Our findings suggest transmission event occurred between Armenia, likely facilitated by or community members, during K. plasmids carrying genes. IMPORTANCE Multidrug-resistant (MDR) has emerged high-risk clone associated hospital- community-acquired infections, posing major threat global public health. in-depth comparative genomics analyses The data sets. possessed incomplete Armenia either through members. In addition, event. study emphasises importance surveillance this emerging MDR-hypervirulent pathogen provide early interventions.

Язык: Английский

Процитировано

0

The mechanisms of resistance, epidemiological characteristics, and molecular evolution of carbapenem-resistant hypervirulent Klebsiella pneumoniae DOI
Qun Wang,

M M Ye,

Chi Hong

и другие.

Laboratory Medicine, Год журнала: 2024, Номер unknown

Опубликована: Дек. 23, 2024

Carbapenem-resistant hypervirulent Klebsiella pneumoniae (CR-hvKP) is a highly pathogenic, drug-resistant, and transmissible "superbug" that causes infections in hospitals communities. Because of the lack effective antimicrobial treatment options, morbidity mortality from CR-hvKP have increased dramatically, outbreaks rapid spread become major global public health challenge. The mechanisms molecular evolution include acquisition plasmid encoding virulence gene by carbapenemase-producing K pneumoniae, horizontal transfer plasmids carrying carbapenem resistance genes to hvKP, fusion both classic pneumoniae. In addition, hvKP can develop phenotype under antibiotic pressure. arises through plasmid-mediated convergence factors. Its multidrug lethal pathogenicity fuel hospital outbreaks, requiring urgent action block transmission strengthen surveillance contain this evolving superbug. article, we summarized carbapenemase mechanism, factors, epidemiology CR-hvKP. Our aim was elucidate evolutionary mechanism provide reference for curbing

Язык: Английский

Процитировано

0