Identification of key genes with abnormal RNA methylation modification and selected m6A regulators in ankylosing spondylitis DOI Creative Commons

Fengqing Wu,

Hongbin Huang, Deyang Sun

и другие.

Immunity Inflammation and Disease, Год журнала: 2024, Номер 12(8)

Опубликована: Авг. 1, 2024

Abstract Background N6‐methyladenosine (m6A) has been identified as the most abundant modification of RNA molecules and aberrant m6A modifications have associated with development autoimmune diseases. However, role in ankylosing spondylitis (AS) not adequately investigated. Therefore, we aimed to explore significance regulator‐mediated methylation AS. Methods The methylated immunoprecipitation sequencing (meRIP‐seq) digital (Digital RNA‐seq) were conducted using peripheral blood mononuclear cells from three AS cases healthy controls, identify genes affected by abnormal methylation. different peaks cross‐referenced AS‐related obtained GeneCards Suite. Subsequently, expression levels shared differentially expressed (DEGs) key regulators evaluated data 68 36 controls two sets (GSE25101 GSE73754). In addition, results validated through quantitative polymerase chain reaction (qPCR). Results meRIP‐seq Digital RNA‐seq analyses 28 upregulated but downregulated expression, 52 expression. By intersecting 2184 Suite, a total five DEGs: BCL11B , KAT6B IL1R1 TRIB1 ALDH2 . Through analysis qPCR, found that Moreover, regulators, WTAP heterogeneous nuclear ribonucleoprotein C, identified. Conclusions conclusion, current study revealed plays crucial might hence provide new treatment strategy for disease.

Язык: Английский

RNA methylation patterns, immune characteristics, and autophagy-related mechanisms mediated by N6-methyladenosine (m6A) regulatory factors in venous thromboembolism DOI Creative Commons
Deshuai Zhang,

Wenxia Fu,

Shiwei Zhu

и другие.

BMC Genomics, Год журнала: 2024, Номер 25(1)

Опубликована: Апрель 24, 2024

Abstract Recent studies have found a link between deep vein thrombosis and inflammatory reactions. N6-methyladenosine (m6A), crucial element in immunological regulation, is believed to contribute the pathophysiology of venous thromboembolism (VTE). However, how m6A-modified immune microenvironment involved VTE remains unclear. In present study, we identified relationship expression several m6A regulatory elements by analyzing peripheral blood samples from 177 patients with 88 healthy controls public GEO databases GSE19151 GSE48000. We used machine learning identify essential genes constructed diagnostic model for using multivariate logistic regression. Unsupervised cluster analysis revealed marked difference modification patterns terms cell infiltration, reactivity, autophagy. two m6A-related autophagy (i.e., CHMP2B SIRT1) regulator YTHDF3 bioinformatics. also examined potential mechanisms through which may affect VTE. modification, immunity, are closely linked VTE, offering novel mechanistic therapeutic insights.

Язык: Английский

Процитировано

2

Exploring hub pyroptosis-related genes, molecular subtypes, and potential drugs in ankylosing spondylitis by comprehensive bioinformatics analysis and molecular docking DOI Creative Commons
Xin Li, Xiangying Li, Hongqiang Wang

и другие.

BMC Musculoskeletal Disorders, Год журнала: 2023, Номер 24(1)

Опубликована: Июнь 29, 2023

Ankylosing spondylitis (AS) is a chronic inflammatory autoimmune disease, and the diagnosis treatment of AS have been limited because its pathogenesis still unclear. Pyroptosis proinflammatory type cell death that plays an important role in immune system. However, relationship between pyroptosis genes has never elucidated.GSE73754, GSE25101, GSE221786 datasets were collected from Gene Expression Omnibus (GEO) database. Differentially expressed pyroptosis-related (DE-PRGs) identified by R software. Machine learning PPI networks used to screen key construct diagnostic model AS. patients clustered into different subtypes according DE-PRGs using consensus cluster analysis validated principal component (PCA). WGCNA was for screening hub gene modules two subtypes. Ontology (GO) terms Kyoto Encyclopedia Genes Genomes (KEGG) pathways enrichment elucidate underlying mechanisms. The ESTIMATE CIBERSORT algorithms reveal signatures. connectivity map (CMAP) database predict potential drugs Molecular docking calculate binding affinity gene.Sixteen detected compared healthy controls, some these showed significant correlation with cells such as neutrophils, CD8 + T cells, resting NK cells. Enrichment mainly related pyroptosis, IL-1β, TNF signaling pathways. (TNF, NLRC4, GZMB) screened machine protein-protein interaction (PPI) network establish ROC had good properties GSE73754 (AUC: 0.881), GSE25101 0.797), 0.713). Using 16 DE-PRGs, divided C1 C2 subtypes, differences infiltration. A module WGCNA, suggested function. Three drugs, including ascorbic acid, RO 90-7501, celastrol, selected based on CMAP analysis. Cytoscape GZMB highest-scoring gene. Finally, molecular results acid formed three hydrogen bonds, ARG-41, LYS-40, HIS-57 (affinity: -5.3 kcal/mol). RO-90-7501 one bond, CYS-136 -8.8 celastrol TYR-94, HIS-57, LYS-40 -9.4 kcal/mol).Our research systematically analyzed may play essential microenvironment Our findings will contribute further understanding

Язык: Английский

Процитировано

6

Identification of key genes with abnormal RNA methylation modification and selected m6A regulators in ankylosing spondylitis DOI Creative Commons

Fengqing Wu,

Hongbin Huang, Deyang Sun

и другие.

Immunity Inflammation and Disease, Год журнала: 2024, Номер 12(8)

Опубликована: Авг. 1, 2024

Abstract Background N6‐methyladenosine (m6A) has been identified as the most abundant modification of RNA molecules and aberrant m6A modifications have associated with development autoimmune diseases. However, role in ankylosing spondylitis (AS) not adequately investigated. Therefore, we aimed to explore significance regulator‐mediated methylation AS. Methods The methylated immunoprecipitation sequencing (meRIP‐seq) digital (Digital RNA‐seq) were conducted using peripheral blood mononuclear cells from three AS cases healthy controls, identify genes affected by abnormal methylation. different peaks cross‐referenced AS‐related obtained GeneCards Suite. Subsequently, expression levels shared differentially expressed (DEGs) key regulators evaluated data 68 36 controls two sets (GSE25101 GSE73754). In addition, results validated through quantitative polymerase chain reaction (qPCR). Results meRIP‐seq Digital RNA‐seq analyses 28 upregulated but downregulated expression, 52 expression. By intersecting 2184 Suite, a total five DEGs: BCL11B , KAT6B IL1R1 TRIB1 ALDH2 . Through analysis qPCR, found that Moreover, regulators, WTAP heterogeneous nuclear ribonucleoprotein C, identified. Conclusions conclusion, current study revealed plays crucial might hence provide new treatment strategy for disease.

Язык: Английский

Процитировано

1