The impact of diabetes mellitus on tendon pathology: a review DOI Creative Commons
Jian Xu, Jinbo Wang,

Yuncong Ji

и другие.

Frontiers in Pharmacology, Год журнала: 2024, Номер 15

Опубликована: Ноя. 5, 2024

Diabetes is one of the most common metabolic diseases worldwide, leading to complications, mortality, and significant healthcare expenditures, which impose a substantial social financial burden globally. A diabetic environment can induce changes, negatively affecting tendon homeostasis, alterations in biomechanical properties histopathology. Numerous studies have investigated mechanisms through diabetes exerts pathological effects on tendons, including increased free radical production, oxidative stress, inflammatory responses, deposition advanced glycation end products (AGEs), microvascular changes. These changes damages structure, biomechanics, repair processes. The proliferation stem cells decreases, apoptosis increases, abnormal differentiation, along with expression myofibroblasts, ultimately lead insufficient repair, fibrosis, remodeling. Although researches unveiling tendinopathy, fibrosis or contracture, injury healing are growing, systematic understanding still lacking. Therefore, this review summarizes current research status provides comprehensive overview, offering theoretical guidance for future in-depth exploration impact tendons development treatments diabetes-related diseases.

Язык: Английский

Exercise-augmented THSD7B exhibited a positive prognostic implication and tumor-suppressed functionality in pan-cancer DOI Creative Commons
Zhiwen Luo, Jin-guo Zhu,

Zhengyuan Fang

и другие.

Frontiers in Immunology, Год журнала: 2024, Номер 15

Опубликована: Авг. 5, 2024

Breast cancer, one of the most prevalent malignancies among women worldwide, has rising incidence rates. Physical activity, particularly exercise, emerged as a significant modifier cancer prognosis, influencing both tumor biology and patient outcomes.

Язык: Английский

Процитировано

4

Novel therapeutic insights into pathological cardiac hypertrophy: tRF-16-R29P4PE regulates PACE4 and metabolic pathways DOI

Feng Wang,

Ping Li, Xinxin Yan

и другие.

Biochimica et Biophysica Acta (BBA) - Molecular Cell Research, Год журнала: 2025, Номер unknown, С. 119920 - 119920

Опубликована: Фев. 1, 2025

Язык: Английский

Процитировано

0

Zn‐DHM Nanozymes Enhance Muscle Regeneration Through ROS Scavenging and Macrophage Polarization in Volumetric Muscle Loss Revealed by Single‐Cell Profiling DOI

Xinting Feng,

Zhiwen Luo,

Wei Zhang

и другие.

Advanced Functional Materials, Год журнала: 2025, Номер unknown

Опубликована: Апрель 4, 2025

Abstract Volumetric muscle loss (VML) is a severe condition in which the of skeletal surpasses body's intrinsic repair capabilities, leading to irreversible functional deficits and potential disability, with persistent inflammation impaired myogenic differentiation. To address these challenges, novel zinc‐dihydromyricetin (Zn‐DHM) nanozyme superoxide dismutase (SOD)‐like activity developed, designed neutralize excessive reactive oxygen species (ROS) restore oxidative balance. Zn‐DHM mitigates stress promotes polarization macrophages from proinflammatory M1 phenotype anti‐inflammatory M2 phenotype, thereby reducing chronic creating conducive environment for repair. Further, significantly enhances differentiation C 2 12 cells, accelerating wound healing processes. These studies confirm biosafety low toxicity Zn‐DHM. As per murine tibialis anterior VML model, effectively suppresses markedly improves outcomes. Single‐cell RNA sequencing reveals that treatment increases expression macrophage markers proliferation capacity stem cells (MuSCs). In addition, intercellular communication analysis interactions between MuSCs group, suggesting may drive tissue regeneration through activation GAS Notch signaling pathways.

Язык: Английский

Процитировано

0

Platelet-rich plasma Inhibits Ferroptosis and Inflammation to Alleviate Frozen Shoulder via Activating the CST1/GPX4 Signaling Pathway DOI

Zihao Ren,

Zhen Hu, Yun Zhou

и другие.

Archives of Biochemistry and Biophysics, Год журнала: 2025, Номер unknown, С. 110429 - 110429

Опубликована: Апрель 1, 2025

Язык: Английский

Процитировано

0

The impact of diabetes mellitus on tendon pathology: a review DOI Creative Commons
Jian Xu, Jinbo Wang,

Yuncong Ji

и другие.

Frontiers in Pharmacology, Год журнала: 2024, Номер 15

Опубликована: Ноя. 5, 2024

Diabetes is one of the most common metabolic diseases worldwide, leading to complications, mortality, and significant healthcare expenditures, which impose a substantial social financial burden globally. A diabetic environment can induce changes, negatively affecting tendon homeostasis, alterations in biomechanical properties histopathology. Numerous studies have investigated mechanisms through diabetes exerts pathological effects on tendons, including increased free radical production, oxidative stress, inflammatory responses, deposition advanced glycation end products (AGEs), microvascular changes. These changes damages structure, biomechanics, repair processes. The proliferation stem cells decreases, apoptosis increases, abnormal differentiation, along with expression myofibroblasts, ultimately lead insufficient repair, fibrosis, remodeling. Although researches unveiling tendinopathy, fibrosis or contracture, injury healing are growing, systematic understanding still lacking. Therefore, this review summarizes current research status provides comprehensive overview, offering theoretical guidance for future in-depth exploration impact tendons development treatments diabetes-related diseases.

Язык: Английский

Процитировано

0