From Inflammation to Infertility: How Oxidative Stress and Infections Disrupt Male Reproductive Health DOI Creative Commons
Anastasios Potiris, Efthalia Moustakli,

Eleni Trismpioti

и другие.

Metabolites, Год журнала: 2025, Номер 15(4), С. 267 - 267

Опубликована: Апрель 11, 2025

Background/Objectives: Inflammation, infections, and oxidative stress (OS) all have an impact on male infertility, which is a complicated, multifaceted illness. OS affects motility fertilization capability. It accomplishes this through damaging sperm DNA, oxidizing proteins, triggering lipid peroxidation. These effects occur due to imbalance between reactive oxygen species (ROS) antioxidant defenses. Methods: This review aims evaluate the of inflammation infertility by assessing recent literature. Results: Pro-inflammatory cytokines, like TNF-α IL-6, interfere with spermatogenesis promote damage. Additionally, infections caused pathogens Escherichia coli Chlamydia trachomatis alter reproductive microenvironment, leading dysfunction inflammation. Conclusions: Early detection targeted treatment are essential intricate interactions among these elements. Microbiota-modulating techniques, antimicrobial therapies, anti-inflammatory drugs, antioxidants therapeutic approaches that may help reduce damage enhance fertility.

Язык: Английский

Transferrin Receptor 2 in Canine Testicular Tumors: An Emerging Key Role in Seminomas DOI Creative Commons
Rebecca Leandri, S. Buonocore, Karen A. Power

и другие.

Animals, Год журнала: 2025, Номер 15(2), С. 264 - 264

Опубликована: Янв. 18, 2025

Transferrin Receptor 2 (TfR2) is a homolog of 1 (TfR1), involved in regulating intra and extracellular iron levels. Altered pathways have been associated with cancer onset progression; however, their role canine tumors remains poorly explored. This study investigated TfR2 immunohistochemical expression non-neoplastic testis for the first time most common types testicular tumors: intratubular seminomas (ITSEMs), diffuse (DSEMs), Leydig cell (LCTs), Sertoli (SCTs). Immunohistochemical analysis revealed differential pattern according to tumor type, high observed ITSEMs DSEMs, occasional LCTs, absence SCTs. These results suggest that may play relevant seminoma development. Furthermore, specific highlights its potential as therapeutic target, where regulation possible compensatory mechanisms warrant further investigation.

Язык: Английский

Процитировано

0

Ferritinophagy: a possible new iron-related metabolic target in canine osteoblastic osteosarcoma DOI Creative Commons
Karen A. Power, Rebecca Leandri, Giorgia Federico

и другие.

Frontiers in Veterinary Science, Год журнала: 2025, Номер 12

Опубликована: Март 24, 2025

Canine osteosarcomas (COS) are the most common bone tumors in dogs, characterized by high metastatic rates, poor prognosis, and responsiveness to routine therapies, which highlights need for new treatment targets. In this context, metabolism of neoplastic cells represents an increasingly studied element, as cancer depend on particular metabolic pathways that also elements vulnerability. Among these, tumor (TCs) show higher iron requirements sustain proliferation (so-called addiction), achieved increasing uptake and/or activating ferritinophagy, a process mediated Nuclear receptor Co-Activator 4 (NCOA4) leading mobilization from ferritin (Ft) deposits. Previous studies have shown COS overexpress Transferrin Receptor 1 (TfR1) increase uptake. study we evaluated immunohistochemical expression ferritinophagy-related proteins, namely Ferritin Heavy chain (FTH1) NCOA4, proliferating cell nuclear antigen (PCNA) canine normal osteoblastic osteosarcoma (COOS) samples. Normal samples revealed negative/weak immunoreactivity FTH1, NCOA4 PCNA <10% osteocytes. COOS majority showed PCNA. Our data suggest activation ferritinophagy responds feed their “iron addiction.” These data, though preliminary, further targeting potential strategy worthy be transferred into clinical practice.

Язык: Английский

Процитировано

0

From Inflammation to Infertility: How Oxidative Stress and Infections Disrupt Male Reproductive Health DOI Creative Commons
Anastasios Potiris, Efthalia Moustakli,

Eleni Trismpioti

и другие.

Metabolites, Год журнала: 2025, Номер 15(4), С. 267 - 267

Опубликована: Апрель 11, 2025

Background/Objectives: Inflammation, infections, and oxidative stress (OS) all have an impact on male infertility, which is a complicated, multifaceted illness. OS affects motility fertilization capability. It accomplishes this through damaging sperm DNA, oxidizing proteins, triggering lipid peroxidation. These effects occur due to imbalance between reactive oxygen species (ROS) antioxidant defenses. Methods: This review aims evaluate the of inflammation infertility by assessing recent literature. Results: Pro-inflammatory cytokines, like TNF-α IL-6, interfere with spermatogenesis promote damage. Additionally, infections caused pathogens Escherichia coli Chlamydia trachomatis alter reproductive microenvironment, leading dysfunction inflammation. Conclusions: Early detection targeted treatment are essential intricate interactions among these elements. Microbiota-modulating techniques, antimicrobial therapies, anti-inflammatory drugs, antioxidants therapeutic approaches that may help reduce damage enhance fertility.

Язык: Английский

Процитировано

0