Hemoglobin Affinity to Oxygen during Coronavirus Infection: New Faces of a Known Problem DOI
В. В. Зинчук, N. V. Hlutkina

Российский физиологический журнал им  И  М  Сеченова, Год журнала: 2023, Номер 109(12), С. 1780 - 1798

Опубликована: Дек. 1, 2023

The development of coronavirus infection prevents the transport oxygen to tissues through mechanism impaired hemoglobin saturation in damaged lung. change affinity for oxygen, which is most important factor compensating deficiency various pathological conditions, underlies processes adaptation hypoxia. question significance shift oxyhemoglobin dissociation curve this pathology actively discussed, its left arterial blood and right venous are noted, conditions hypoxic syndrome reflects a-ctivation body’s mechanisms compensate deficiency. position not specific pathology, but determined by severity imbalance cardiorespiratory system, as well peculiarities status patients analyzed sample.

Язык: Английский

Red Blood Cell Adenylate Energetics Is Related to Endothelial and Microvascular Function in Long COVID DOI Creative Commons

Marzena Romanowska‐Kocejko,

Agata Jędrzejewska, Alicja Braczko

и другие.

Biomedicines, Год журнала: 2024, Номер 12(3), С. 554 - 554

Опубликована: Март 1, 2024

Adenine nucleotides play a critical role in maintaining essential functions of red blood cells (RBCs), including energy metabolism, redox status, shape fluctuations and RBC-dependent endothelial microvascular functions. Recently, it has been shown that infection with the severe acute respiratory syndrome coronavirus-2 (SARS-CoV-2) might lead to morphological metabolic alterations erythrocytes both mild cases coronavirus disease (COVID-19). However, little is known about effects COVID-19 on nucleotide energetics RBCs nor potential contribution metabolism long COVID syndrome. This study aimed analyze levels adenine isolated from patients 12 weeks after SARS-CoV-2 who suffered symptoms relate them function parameters as well rate peripheral tissue oxygen supply. Although absolute quantities were rather slightly changed individuals, many related microcirculatory showed significant correlations RBC adenosine triphosphate (ATP) total (TAN) concentration. A particularly strong relationship was observed between ATP serum ratio arginine asymmetric dimethylarginine—an indicator function. Consistently, positive correlation also ATP/ADP diminished reactive hyperemic response patients, assessed by flow-mediated skin fluorescence (FMSF) technique, which reflected decreased vascular nitric oxide bioavailability. In addition, we have significantly impaired ischemic (IR max IR index), examined FMSF, revealed residual bioavailability epidermal keratinocytes brachial artery occlusion. These ratio, confirming key bioenergetics Taken together, outcomes this indicate dysregulation processes co-occurring dysfunction associated intracellular delivery, may partly explain COVID-specific such physical impairment fatigue.

Язык: Английский

Процитировано

5

Pathology of Red Blood Cells in Patients with SARS-CoV-2 DOI Creative Commons
Sona Hakobyan,

Lina Hakobyan,

Liana Abroyan

и другие.

Biomedicines, Год журнала: 2025, Номер 13(1), С. 191 - 191

Опубликована: Янв. 14, 2025

Background: Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) infection has been associated with various hematological disorders. Understanding the pathology of erythrocytes (red blood cells) in may provide insights into disease severity and progression. Objective: To review analyze general patients infected SARS-CoV-2, focusing on clinical laboratory findings across different groups. Methods: Patients were classified four groups based criteria: Group 1: Regular group (fever, symptoms, radiographic evidence pneumonia). 2: (shortness breath >30 breaths/min, peripheral oxygen saturation <92% at rest, extensive pneumonia, failure requiring mechanical ventilation, and/or organ necessitating intensive care). 3: Low (peripheral <85% rest). 4: Erythroblastosis (erythroblast count >0.5% among total nucleated cells). Clinical investigations included major routine studies scanning microspectrophotometry to measure hemoglobin (Hb) spectra unstained erythrocytes. Results: Erythroblasts detected approximately 30% SARS-CoV-2 patients, predominantly severe group. Serum ferritin, C-reactive protein (CRP), anisocytosis strongly correlated severity. Microspectrophotometric revealed significant changes adsorption spectra, an increase Hb absorbance 420 nm cases compared normal controls. Conclusions: Elevated serum CRP levels, anisocytosis, altered absorption wavelength are adverse outcomes infection. These highlight potential utility parameters as markers for prognosis viral infections.

Язык: Английский

Процитировано

0

COVID‐19 and cognitive impairment: From evidence to SARS‐CoV‐2 mechanism DOI Creative Commons

Haodong Pan,

Jing-Yan Niu,

Feng Lin

и другие.

Brain‐X, Год журнала: 2024, Номер 2(2)

Опубликована: Апрель 16, 2024

Abstract Caused by severe acute respiratory syndrome coronavirus 2 (SARS‐CoV‐2), disease 2019 (COVID‐19) primarily manifests as dysfunction. However, emerging evidence suggests SARS‐CoV‐2 can invade the brain, leading to cognitive impairment (CI). It may spread other brain regions through transsynaptic neurons, including olfactory, optic, and vagus nerves. Moreover, it central nervous system blood transmission or lymphatic system. This review summarizes neuroimaging from clinical imaging studies of COVID‐19‐associated CIs, magnetic resonance 18F‐fluorodeoxyglucose positron emission tomography‐computed tomography. The mechanisms underlying CIs are currently being actively investigated. They include nonimmune effects, such viral proteins, tissue hypoxia, hypercoagulability, pathological changes in neuronal cells, immune microglia astrocyte activation, peripheral cell infiltration, blood‐brain barrier impairment, cytokine network dysregulation, intestinal microbiota. Inflammation is feature. Both systemic inflammation cause persistent neurological changes, existing indicates that underlies elevated risk Alzheimer's disease. Finally, potential therapeutic options for discussed. In‐depth research into still needed help develop new therapies.

Язык: Английский

Процитировано

2

The Ways of the Virus: Interactions of Platelets and Red Blood Cells with SARS-CoV-2, and Their Potential Pathophysiological Significance in COVID-19 DOI Open Access
Mikhail A. Panteleev, А. Н. Свешникова,

Soslan S. Shakhidzhanov

и другие.

International Journal of Molecular Sciences, Год журнала: 2023, Номер 24(24), С. 17291 - 17291

Опубликована: Дек. 9, 2023

The hematological effects of severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) are important in COVID-19 pathophysiology. However, the interactions SARS-CoV-2 with platelets and red blood cells still poorly understood. There conflicting data regarding mechanisms significance these interactions. aim this review is to put together available discuss hypotheses, known suspected virus on cells, their pathophysiological diagnostic significance, potential role virus's transport, propagation, clearance by immune system. We pay particular attention mutual activation platelets, system, endothelium, coagulation how changes evolution SARS-CoV-2. now convincing evidence that along platelet erythroid precursors (but not mature erythrocytes), frequently infected functionally changed. infection yet entirely clear. Still, significantly associated disease severity likely have prognostic development thrombotic pulmonary complications.

Язык: Английский

Процитировано

6

Hemoglobin Affinity to Oxygen during Coronavirus Infection: New Faces of a Known Problem DOI
В. В. Зинчук, N. V. Hlutkina

Journal of Evolutionary Biochemistry and Physiology, Год журнала: 2023, Номер 59(6), С. 2242 - 2257

Опубликована: Ноя. 1, 2023

Язык: Английский

Процитировано

0

Hemoglobin Affinity to Oxygen during Coronavirus Infection: New Faces of a Known Problem DOI
В. В. Зинчук, N. V. Hlutkina

Российский физиологический журнал им  И  М  Сеченова, Год журнала: 2023, Номер 109(12), С. 1780 - 1798

Опубликована: Дек. 1, 2023

The development of coronavirus infection prevents the transport oxygen to tissues through mechanism impaired hemoglobin saturation in damaged lung. change affinity for oxygen, which is most important factor compensating deficiency various pathological conditions, underlies processes adaptation hypoxia. question significance shift oxyhemoglobin dissociation curve this pathology actively discussed, its left arterial blood and right venous are noted, conditions hypoxic syndrome reflects a-ctivation body’s mechanisms compensate deficiency. position not specific pathology, but determined by severity imbalance cardiorespiratory system, as well peculiarities status patients analyzed sample.

Язык: Английский

Процитировано

0