Oxidative Stress in Cataract Formation: Is There a Treatment Approach on the Horizon? DOI Creative Commons
Jingyan Li, Francesco Buonfiglio, Ying Zeng

и другие.

Antioxidants, Год журнала: 2024, Номер 13(10), С. 1249 - 1249

Опубликована: Окт. 16, 2024

Cataracts, a leading cause of blindness worldwide, are closely linked to oxidative stress-induced damage lens epithelial cells (LECs). Key factors contributing cataract formation include aging, arterial hypertension, and diabetes mellitus. Given the high global prevalence cataracts, burden cataract-related visual impairment is substantial, highlighting need for pharmacological strategies supplement surgical interventions. Understanding molecular pathways involved in stress during development may offer valuable insights designing novel therapeutic approaches. This review explores role formation, focusing on critical mechanisms, such as mitochondrial dysfunction, endoplasmic reticulum stress, loss gap junctions, various cell death LECs. Additionally, we discuss emerging potential targeting options, including antioxidant-based treatments.

Язык: Английский

Aging of the eye: Lessons from cataracts and age-related macular degeneration DOI Creative Commons
Aleš Cvekl, Jan Vijg

Ageing Research Reviews, Год журнала: 2024, Номер 99, С. 102407 - 102407

Опубликована: Июль 6, 2024

Aging is the greatest risk factor for chronic human diseases, including many eye diseases. Geroscience aims to understand effects of aging process on these genetic, molecular, and cellular mechanisms that underlie increased disease over lifetime. Understanding increases general knowledge physiology impacted by processes at various biological extremes. Two major age-related cataract macular degeneration (AMD) are caused dysfunction lens retina, respectively. Lens transparency light refraction mediated fiber cells lacking nuclei other organelles, which provides a unique opportunity study single hallmark, i.e., loss proteostasis, within an environment limited metabolism. In AMD, local photoreceptors/retinal pigmented epithelium/Bruch's membrane/choriocapillaris complex in macula leads photoreceptors eventually central vision, driven nearly all hallmarks shares features with Alzheimer's disease, Parkinson's cardiovascular diabetes. The can function as model studying basic and, vice versa, well-defined be used tools disease.

Язык: Английский

Процитировано

12

Oxidative Stress and Cataract Formation: Evaluating the Efficacy of Antioxidant Therapies DOI Creative Commons
Merve Kulbay, Kevin Y. Wu, Gurleen K. Nirwal

и другие.

Biomolecules, Год журнала: 2024, Номер 14(9), С. 1055 - 1055

Опубликована: Авг. 25, 2024

This comprehensive review investigates the pivotal role of reactive oxygen species (ROS) in cataract formation and evaluates potential antioxidant therapies mitigating this ocular condition. By elucidating mechanisms oxidative stress, article examines how ROS contribute to deterioration lens proteins lipids, leading characteristic aggregation, cross-linking, light scattering observed cataracts. The provides a thorough assessment various strategies aimed at preventing managing cataracts, such as dietary antioxidants (i.e., vitamins C E, lutein, zeaxanthin), well pharmacological agents with antioxidative properties. Furthermore, explores innovative therapeutic approaches, including gene therapy nanotechnology-based delivery systems, designed bolster defenses tissues. Concluding critical analysis current research, offers evidence-based recommendations for optimizing therapies. literature on use prevent is sparse. There lack conclusions; further clinical studies are needed endorse patients cataractogenesis. However, personalized treatment plans considering individual patient factors disease stages can be applied. serves valuable resource, providing insights into alleviate burden

Язык: Английский

Процитировано

7

Oxidative Stress in Cataract Formation: Is There a Treatment Approach on the Horizon? DOI Creative Commons
Jingyan Li, Francesco Buonfiglio, Ying Zeng

и другие.

Antioxidants, Год журнала: 2024, Номер 13(10), С. 1249 - 1249

Опубликована: Окт. 16, 2024

Cataracts, a leading cause of blindness worldwide, are closely linked to oxidative stress-induced damage lens epithelial cells (LECs). Key factors contributing cataract formation include aging, arterial hypertension, and diabetes mellitus. Given the high global prevalence cataracts, burden cataract-related visual impairment is substantial, highlighting need for pharmacological strategies supplement surgical interventions. Understanding molecular pathways involved in stress during development may offer valuable insights designing novel therapeutic approaches. This review explores role formation, focusing on critical mechanisms, such as mitochondrial dysfunction, endoplasmic reticulum stress, loss gap junctions, various cell death LECs. Additionally, we discuss emerging potential targeting options, including antioxidant-based treatments.

Язык: Английский

Процитировано

2