Tumor Biology Hides Novel Therapeutic Approaches to Diffuse Large B-Cell Lymphoma: A Narrative Review DOI Open Access
Romana Masnikosa, Zorica Cvetković, David Pirić

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(21), С. 11384 - 11384

Опубликована: Окт. 23, 2024

Diffuse large B-cell lymphoma (DLBCL) is a malignancy of immense biological and clinical heterogeneity. Based on the transcriptomic or genomic approach, several different classification schemes have evolved over years to subdivide DLBCL into clinically (prognostically) relevant subsets, but each leaves unclassified samples. Herein, we outline tumor biology behind actual potential drug targets address challenges drawbacks coupled with their (potential) use. Therapeutic modalities are discussed, including small-molecule inhibitors, naked antibodies, antibody-drug conjugates, chimeric antigen receptors, bispecific antibodies T-cell engagers, immune checkpoint inhibitors. Candidate drugs explored in ongoing trials diverse toxicity issues refractoriness drugs. According literature DLBCL, promise for new therapeutic lies epigenetic alterations, receptor NF-κB pathways. present putative hiding lipid pathways, ferroptosis, gut microbiome that could be used addition immuno-chemotherapy improve general health status patients, thus increasing chance being cured. It may time devote more effort exploring metabolism discover novel druggable targets. We also performed bibliometric knowledge-map analysis published from 2014-2023.

Язык: Английский

Mechanisms and technologies in cancer epigenetics DOI Creative Commons
Zaki A. Sherif, Olorunseun O. Ogunwobi, Habtom W. Ressom

и другие.

Frontiers in Oncology, Год журнала: 2025, Номер 14

Опубликована: Янв. 7, 2025

Cancer's epigenetic landscape, a labyrinthine tapestry of molecular modifications, has long captivated researchers with its profound influence on gene expression and cellular fate. This review discusses the intricate mechanisms underlying cancer epigenetics, unraveling complex interplay between DNA methylation, histone chromatin remodeling, non-coding RNAs. We navigate through tumultuous seas dysregulation, exploring how these processes conspire to silence tumor suppressors unleash oncogenic potential. The narrative pivots cutting-edge technologies, revolutionizing our ability decode epigenome. From granular insights single-cell epigenomics holistic view offered by multi-omics approaches, we examine tools are reshaping understanding heterogeneity evolution. also highlights emerging techniques, such as spatial long-read sequencing, which promise unveil hidden dimensions regulation. Finally, probed transformative potential CRISPR-based epigenome editing computational analysis transmute raw data into biological insights. study seeks synthesize comprehensive yet nuanced contemporary landscape future directions research.

Язык: Английский

Процитировано

0

FCRL1 and BAFF mRNA Expression as Novel Diagnostic and Prognostic Biomarkers in Diffuse Large B-Cell Lymphoma: Expression Signatures Predict R-CHOP Therapy Response and Survival DOI Open Access

Hiba S. Al-Amodi,

Hanan M. Bedair, Suzy Gohar

и другие.

International Journal of Molecular Sciences, Год журнала: 2025, Номер 26(3), С. 1269 - 1269

Опубликована: Янв. 31, 2025

This study investigated the diagnostic, prognostic, and therapeutic significance of Fc receptor-like 1 (FCRL1) B-cell activating factor (BAFF) mRNA expression in Egyptian patients with diffuse large lymphoma (DLBCL) undergoing standard R-CHOP regimen (rituximab, cyclophosphamide, doxorubicin, vincristine, prednisone) using quantitative real-time PCR (RT-qPCR). The results demonstrated that FCRL1 BAFF were significantly elevated DLBCL compared to healthy controls. A strong positive correlation existed between levels. Diagnostic performance assessed through combined ROC curve analysis revealed BAFF, FCRL1, lactate dehydrogenase (LDH) achieved perfect diagnostic accuracy (AUC = 1.0), demonstrating 100% sensitivity, specificity, predictive values. Further prognostic COX regression identified as most significant predictor poor clinical outcomes. Kaplan–Meier survival reinforced this finding, high showing associations reduced overall (OS, p 0.031) progression-free (PFS, 0.038). underscores potential utility markers for DLBCL, emerging a promising marker target enabling more tailored treatment approaches common type non-Hodgkin lymphoma, receiving therapy.

Язык: Английский

Процитировано

0

Bioequivalence of generic and branded ibrutinib capsules in healthy Chinese volunteers under fasting and fed conditions: a randomized, four-period, fully replicated, crossover study DOI
Rong Zhang, Rachel Y. Chong,

Shaomei Yang

и другие.

Expert Opinion on Drug Metabolism & Toxicology, Год журнала: 2025, Номер unknown

Опубликована: Апрель 23, 2025

The aim of this study was to evaluate the bioequivalence test and reference products ibrutinib capsule (140 mg). This a fully replicated crossover that included 100 healthy Chinese volunteers (50 in fasting BE 50 fed study). Subjects were assigned receive single dose or product each treatment period. main PK parameters (Cmax, AUC0-t, AUC0-∞) evaluated using either average (ABE) approach reference-scaled (RSABE) approach, depending on within-subject standard deviation (SWR) estimated study. RSABE applied Cmax as corresponding SWR value exceeded cutoff 0.294, while ABE AUC0-t AUC0-∞ values less than 0.294. All three met acceptance criteria both studies. are bioequivalent under conditions. also confirmed high intra-subject variability for ibrutinib. http://www.chinadrugtrials.org.cn/index.html identifier is CTR20202168.

Язык: Английский

Процитировано

0

Tumor Biology Hides Novel Therapeutic Approaches to Diffuse Large B-Cell Lymphoma: A Narrative Review DOI Open Access
Romana Masnikosa, Zorica Cvetković, David Pirić

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(21), С. 11384 - 11384

Опубликована: Окт. 23, 2024

Diffuse large B-cell lymphoma (DLBCL) is a malignancy of immense biological and clinical heterogeneity. Based on the transcriptomic or genomic approach, several different classification schemes have evolved over years to subdivide DLBCL into clinically (prognostically) relevant subsets, but each leaves unclassified samples. Herein, we outline tumor biology behind actual potential drug targets address challenges drawbacks coupled with their (potential) use. Therapeutic modalities are discussed, including small-molecule inhibitors, naked antibodies, antibody-drug conjugates, chimeric antigen receptors, bispecific antibodies T-cell engagers, immune checkpoint inhibitors. Candidate drugs explored in ongoing trials diverse toxicity issues refractoriness drugs. According literature DLBCL, promise for new therapeutic lies epigenetic alterations, receptor NF-κB pathways. present putative hiding lipid pathways, ferroptosis, gut microbiome that could be used addition immuno-chemotherapy improve general health status patients, thus increasing chance being cured. It may time devote more effort exploring metabolism discover novel druggable targets. We also performed bibliometric knowledge-map analysis published from 2014-2023.

Язык: Английский

Процитировано

1