Bu Shen Huo Xue Formula Provides Neuroprotection Against Spinal Cord Injury by Inhibits Oxidative Stress by Activating the Nrf2 Signaling Pathway DOI Creative Commons
Dan Luo,

Yonghui Hou,

Jiheng Zhan

и другие.

Drug Design Development and Therapy, Год журнала: 2024, Номер Volume 18, С. 4779 - 4797

Опубликована: Окт. 1, 2024

Spinal cord injury (SCI) is an irreversible neurological disease that can result in severe dysfunction. The Bu Shen Huo Xue Formula (BSHXF) has been clinically shown to assist the recovery of limb function patients with SCI. However, underlying mechanisms BSHXF's therapeutic effects remain unclear. This study aimed evaluate BSHXF a mouse model SCI and identify potential targets.

Язык: Английский

Agathisflavone Modulates Reactive Gliosis After Trauma and Increases the Neuroblast Population at the Subventricular Zone DOI Open Access
Juliana Helena Castro e Silva, Francesca Pieropan, Andrea Rivera

и другие.

Nutrients, Год журнала: 2024, Номер 16(23), С. 4053 - 4053

Опубликована: Ноя. 26, 2024

Background: Reactive astrogliosis and microgliosis are coordinated responses to CNS insults pathological hallmarks of traumatic brain injury (TBI). In these conditions, persistent reactive gliosis can impede tissue repopulation limit neurogenesis. Thus, modulating this phenomenon has been increasingly recognized as potential therapeutic approach. Methods: study, we investigated the flavonoid agathisflavone modulate astroglial microglial promote neurogenesis in subventricular zone (SVZ) neurogenic niche. Agathisflavone, or vehicle controls, was administered directly into lateral ventricles postnatal day (P)8-10 mice by twice daily intracerebroventricular (ICV) injections for 3 days, brains were examined at P11. Results: ICV injection caused glial reactivity along needle track, characterised immunohistochemically increased astrocyte expression fibrillary protein (GFAP) number Iba-1+ microglia lesion site. Treatment with decreased GFAP expression, reduced both core site penumbra, induced a 2-fold increase on ratio anti-inflammatory CD206+ pro-inflammatory CD16/32+ microglia. Notably, population neuroblasts (GFAP+ type B cells) all SVZ microdomains up double, without significantly increasing neuronal progenitors (DCX+). Conclusions: Although future studies should investigate underlying molecular mechanisms driving effects polarization different timepoints, data indicate that could be adjuvant treatment TBI central nervous system disorders have common feature.

Язык: Английский

Процитировано

2

Bu Shen Huo Xue Formula Provides Neuroprotection Against Spinal Cord Injury by Inhibits Oxidative Stress by Activating the Nrf2 Signaling Pathway DOI Creative Commons
Dan Luo,

Yonghui Hou,

Jiheng Zhan

и другие.

Drug Design Development and Therapy, Год журнала: 2024, Номер Volume 18, С. 4779 - 4797

Опубликована: Окт. 1, 2024

Spinal cord injury (SCI) is an irreversible neurological disease that can result in severe dysfunction. The Bu Shen Huo Xue Formula (BSHXF) has been clinically shown to assist the recovery of limb function patients with SCI. However, underlying mechanisms BSHXF's therapeutic effects remain unclear. This study aimed evaluate BSHXF a mouse model SCI and identify potential targets.

Язык: Английский

Процитировано

0