Anti-tumor mechanism of artesunate DOI Creative Commons
Xiaoyu Fan, Yiming Yan, Yafei Li

и другие.

Frontiers in Pharmacology, Год журнала: 2024, Номер 15

Опубликована: Окт. 25, 2024

Artesunate (ART) is a classic antimalarial drug with high efficiency, low toxicity and tolerance. It has been shown to be safe good anti-tumor effect. Existing clinical studies have that the mechanisms of ART mainly include inducing apoptosis autophagy tumor cells, affecting microenvironment, regulating immune response, overcoming resistance, as well inhibiting cell proliferation, migration, invasion, angiogenesis. proven fight against lung cancer, hepatocarcinoma, lymphoma, multiple myeloma, leukemia, colorectal ovarian cervical malignant melanoma, oral squamous carcinoma, bladder prostate cancer other neoplasms. In this review, we highlight effects on various tumors an emphasis its mechanism, which helpful propose potential research directions expand application.

Язык: Английский

Dihydroartemisinin suppresses COX-2-mediated apoptosis resistance in hepatocellular carcinoma under endoplasmic reticulum stress DOI
Lulu Cao, Jun Lin, Yun Fang

и другие.

Cytotechnology, Год журнала: 2025, Номер 77(2)

Опубликована: Фев. 13, 2025

Язык: Английский

Процитировано

0

Editorial: Drugs and methods that enhance the anti-cancer efficacy of artesunate DOI Creative Commons
Tuğçenur Uzun

Frontiers in Pharmacology, Год журнала: 2025, Номер 16

Опубликована: Март 17, 2025

Ferroptosis, a regulated cell death mechanism triggered by iron-dependent lipid peroxidation, has emerged as promising target in cancer therapy. This process is distinct from other forms of death, such apoptosis, necrosis, and autolysis, due to its unique morphological biochemical characteristics. The high iron demand susceptibility cells ferroptosis make this particularly advantageous addressing drug resistance metastasis. Liu et al. have explored the role therapy metastasis, detailing molecular mechanisms associated with potential overcoming preventing metastasis (Liu al.).Several studies shown that reduced sensitivity leads (9,10). Conversely, inducing been found enhance efficacy standard chemotherapy, targeted therapies, radiotherapy, immunotherapy (11,12). Furthermore, biological processes epithelial-mesenchymal transition (EMT) noncoding RNAs regulate ferroptosis, thereby influencing (13)(14)(15). In context, targeting represents crucial strategy for both spread re-sensitizing resistant therapy.As an antimalarial drug, artesunate recently gained attention researchers effects on (16,17). Artesunate triggers increasing ROS production disrupting metabolism (17,18). Particularly ferroptosis-sensitive types, cytotoxic are further enhanced when combined treatments platinum-based drugs, paclitaxel, temozolomide (19)(20)(21).The extends beyond resistance. It also capacity inhibit metastatic cells. For example, suppressing EMT production, offers significant advantage cells.While ferroptosis-based offer strategies treatment, development therapeutic approaches requires innovative designs diversify efficacy. regard, combining different pharmacophores provides novel pathway address fundamental challenges selective toxicity. Pharmacophore hybrids represent tools not only complement existing treatment but expand clinical benefits current multiple pathways while sparing healthy tissues.The combination active advantages, including modulation targets, efficacy, prevention development, reduction side effects. Dong designed synthesized 15 dihydroartemisinin isatin derivatives three-carbon linker, demonstrating their potent anti-lung activity. These were extensively tested against drug-sensitive (A549), doxorubicin-resistant (A549/DOX), cisplatin-resistant (A549/DDP) lung lines (Dong al.).Biological evaluations revealed 6a 6e exhibited comparable IC50 values doxorubicin cisplatin lines. non-toxic profile normal epithelial highlighted selectivity. Notably, hybrid displayed demonstrated good stability mouse human microsomes superior pharmacokinetic properties, making it candidate preclinical studies.The study underscores developing pharmacophore solutions enhancing They anticipated hold place future applications.In study, Lin discussed diverse activities artemisitin (ATT), natural compound isolated Artemisia annua L. analogue artemisinin. ATT, identified endoperoxide, promise diseases inflammation, oxidative stress, cancer. instance, ATT bind cysteine residues Keap1 protein, stabilize Nrf2, reduce stress-induced damage. Additionally, reported NLRP3 inflammasome activation, inflammatory processes. context anticancer effects, induce NEDD4/c-Myc/topoisomerase pathways, tumor growth. However, unclear proteins remain barrier application. emphasize identifying ATT's critical structural optimization elucidation pharmacological mechanisms. Ma comprehensively investigated (ART) choroidal melanoma (CM) using network pharmacology, docking, experimental validation. addressed limited options poor prognosis CM, highly malignant ocular tumor.Artesunate, broad-spectrum antitumor properties. ART targets pharmacology key apoptosis induction, cycle arrest, stress regulation. Molecular docking confirmed ART's strong binding ability these pathways.Experimental validation supported ART. vitro experiments induced via p53 signaling pathway, inhibited PI3K/AKT/mTOR arrest at G0/G1 phase, increased levels activate NRF2/HO-1 pathway. findings corroborated vivo studies, which showed suppression CM growth ART.The was proposed interfere migration through interactions MMP2 MMP9, play roles extracellular matrix degradation metastasis.In conclusion, presented compelling evidence multifaceted oncogenic promoting arresting cycle, levels. highlight agent paving way strategies.The scope artemisinin malaria offering opportunities managing complex like molecules create integration into strategies. scientific journey, bridging traditional knowledge modern biotechnological innovation, demonstrates profound outcomes can arise synthesis. Successfully translating applications challenges, delivery systems, mechanisms, improvement formulation bioavailability Moreover, deeper understanding clarification safety profiles essential ensure effective safe implementation. expected pivotal more therapies.

Язык: Английский

Процитировано

0

Multidimensional regulation of transcription factors: decoding the comprehensive signals of plant secondary metabolism DOI Creative Commons
Hongwei Li, Nana Chen,

Hong‐Bin Zhang

и другие.

Frontiers in Plant Science, Год журнала: 2025, Номер 16

Опубликована: Март 26, 2025

Plants synthesize an extensive array of secondary metabolites in response to diverse biotic and abiotic stresses. These function not only as defensive compounds but also constitute significant sources nutrition pharmaceuticals. However, the mechanisms governing synthesis these have long been a central focus research continue pose challenges. Transcription factors (TFs), serving key regulators metabolite plants, exhibit action that are still fully understood. This review summarizes latest advancements on how plant transcription mediate regulation biosynthesis through various signaling pathways, including light signaling, hormone MAPK ubiquitin-proteasome pathway, epigenetic regulation, microbial interactions, climate change. A deeper understanding regulating is expected provide new insights into metabolites.

Язык: Английский

Процитировано

0

New Roles of Artemisinins in Atherosclerosis Progression DOI
Hamidreza Majidiani, Maryam Musavi, Amir Abbas Momtazi‐Borojeni

и другие.

Phytotherapy Research, Год журнала: 2025, Номер unknown

Опубликована: Апрель 8, 2025

ABSTRACT Artemisinin is a natural compound derived from the Chinese plant Artemisia annua , which was officially approved by FDA for its antimalarial effects. In recent years, growing body of studies has shown novel function artemisinin in atherosclerosis therapy. vivo have that can inhibit progression plaque. present review, evidence showing inhibitory effects on plaque and underlying mechanisms discussed. Mechanistically, derivatives act modulating various atherosclerosis‐mediating risk factors, including hyperlipidemia, inflammation, oxidative stress, malfunctioning vascular smooth muscle cells (VSMCs). Notably, artesunate, but not artemisinin, attenuate plasma levels TG, TC, VLDL‐C, LDL‐c, along with substantial decline arterial lipid deposition through enhancing LDPL activity via inducing KFL2/NRF2/TCF7L2 axis. found to ameliorate inflammation reducing monocyte adhesion subsequent transmigration intima, inhibiting expression ICAM‐1 VCAM‐1, diminishing NLRP3 inflammasome activation, inflammatory factors such as IL‐1β, IL‐18, TNF‐α, MCP‐1, TGF‐β1 mechanistically mainly suppressing NF‐κB activity. could exert antioxidant activating PI3K/Akt/eNOS signaling pathway ROS‐mediated signal pathway. also improve VSMC These findings suggest new therapeutic agent treating atherosclerosis; however, future clinical trials are warranted validate efficiency patients atherosclerosis.

Язык: Английский

Процитировано

0

Artemisinin-loaded polylactic acid nanoparticles alleviate 1.2 N,N-dimethylhydrazine-induced colorectal cancer in Albino rats DOI Creative Commons

Zeinab Dwidar,

Walaa Awadin, Mohamed El‐Adl

и другие.

Cancer Nanotechnology, Год журнала: 2025, Номер 16(1)

Опубликована: Апрель 19, 2025

Язык: Английский

Процитировано

0

Updating the Pharmacological Effects of α-Mangostin Compound and Unraveling Its Mechanism of Action: A Computational Study Review DOI Creative Commons
Cecep Suhandi, Gofarana Wilar, Angga Cipta Narsa

и другие.

Drug Design Development and Therapy, Год журнала: 2024, Номер Volume 18, С. 4723 - 4748

Опубликована: Окт. 1, 2024

α-Mangostin, initially identified in 1855, is a xanthone derivative compound predominantly located the pericarp of mangosteen fruit (

Язык: Английский

Процитировано

2

Anti-tumor mechanism of artesunate DOI Creative Commons
Xiaoyu Fan, Yiming Yan, Yafei Li

и другие.

Frontiers in Pharmacology, Год журнала: 2024, Номер 15

Опубликована: Окт. 25, 2024

Artesunate (ART) is a classic antimalarial drug with high efficiency, low toxicity and tolerance. It has been shown to be safe good anti-tumor effect. Existing clinical studies have that the mechanisms of ART mainly include inducing apoptosis autophagy tumor cells, affecting microenvironment, regulating immune response, overcoming resistance, as well inhibiting cell proliferation, migration, invasion, angiogenesis. proven fight against lung cancer, hepatocarcinoma, lymphoma, multiple myeloma, leukemia, colorectal ovarian cervical malignant melanoma, oral squamous carcinoma, bladder prostate cancer other neoplasms. In this review, we highlight effects on various tumors an emphasis its mechanism, which helpful propose potential research directions expand application.

Язык: Английский

Процитировано

0