Polyglucosan body density in the aged mouse hippocampus is controlled by a novel modifier locus on chromosome 1 DOI Creative Commons
Alicia Gómez-Pascual, Dow Michael Glikman,

H.-H. Ng

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2023, Номер unknown

Опубликована: Ноя. 22, 2023

In aged humans and mice, aggregates of hypobranched glycogen molecules called polyglucosan bodies (PGBs) accumulate in hippocampal astrocytes. PGBs are known to drive cognitive decline neurological diseases but remain largely unstudied the context typical brain aging. Here, we show that arise autophagy-dysregulated astrocytes C57BL/6J mouse hippocampus. To map genetic cause age-related PGB accumulation, quantified burden 32 fully sequenced BXD-recombinant inbred strains, which display a 400-fold variation at 16-18 months age. A major modifier locus was mapped chromosome 1 72-75 Mb, defined as

Язык: Английский

Glia: The Cellular Glue that binds Circadian Rhythms and Sleep DOI Creative Commons

Catarina Cavalhas-Almeida,

Amita Sehgal

SLEEP, Год журнала: 2025, Номер unknown

Опубликована: Янв. 15, 2025

Glia are increasingly appreciated as serving an important function in the control of sleep and circadian rhythms. Glial cells Drosophila mammals regulate daily rhythms locomotor activity well homeostatic rebound following deprivation. In addition, they contribute to proposed functions sleep, with different mapping varied glial subtypes. Here, we discuss recent findings rodent models establishing a role glia or regulation synaptic plasticity, brain metabolism, removal cellular debris immune challenges. These underscore relevance for benefits attributed have implications understanding neurobiological mechanisms underlying associated disorders.

Язык: Английский

Процитировано

0

Adopting whole-brain computational modelling to investigate neurophysiological features associated with cognition DOI

Ching Hei Kuang,

Niall W. Duncan

˜The œPsychology of learning and motivation/˜The œpsychology of learning and motivation, Год журнала: 2025, Номер unknown

Опубликована: Янв. 1, 2025

Язык: Английский

Процитировано

0

Modeling of blood flow dynamics in the rat somatosensory cortex DOI Creative Commons
S. Battini, Nicola Cantarutti, Christos Kotsalos

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Ноя. 14, 2024

Abstract The cerebral microvasculature forms a dense network of interconnected blood vessels where flow is modulated partly by astrocytes. Increased neuronal activity stimulates astrocytes to release vasoactive substances at the endfeet, altering diameters connected vessels. Our study simulated coupling between variations and vessel diameter changes driven astrocytic in rat somatosensory cortex. We developed framework with three key components: vasculature synthesized morphologies, fluid dynamics model compute each vascular segment, stochastic process replicating effect endfeet on radii. was validated against experimental values from literature across cortical depths. found that local vasodilation astrocyte increased flow, especially capillaries, exhibiting layer-specific response deeper layers. Additionally, highest variability occurred emphasizing their role perfusion regulation. discovered impacts localized, clustered manner, most segments influenced two neighboring endfeet. These insights enhance our understanding neurovascular guide future research flow-related diseases.

Язык: Английский

Процитировано

1

ATP Restoration by ATP-Deprived Cultured Primary Astrocytes DOI Creative Commons
Gabriele Karger, Johanna Elisabeth Willker, Antonia Regina Harders

и другие.

Neurochemical Research, Год журнала: 2024, Номер 50(1)

Опубликована: Ноя. 16, 2024

Abstract A high cellular concentration of adenosine triphosphate (ATP) is essential to fuel many important functions brain astrocytes. Although ATP depletion has frequently been reported for astrocytes, little known on the metabolic pathways that contribute restoration by ATP-depleted Incubation cultured primary rat astrocytes in glucose-free buffer 60 min with mitochondrial uncoupler BAM15 lowered content around 70%, total amount phosphates 50% and adenylate energy charge (AEC) from 0.9 0.6. Testing after removal revealed presence glucose as exclusive substrate allowed cells restore within 6 h 80% initial content, while coapplication plus enabled fully their min. rapid but incomplete transient was found had exposed alone. This completely prevented application pyruvate uptake inhibitor UK5099, respiratory chain antimycin or continuous BAM15. However, these compounds strongly accelerated release lactate cells, suggesting ribose moiety can serve some via metabolism. Finally, adenosine-accelerated glucose-fed inhibited kinase ABT-702. These data demonstrate require a complete both AMP precursor.

Язык: Английский

Процитировано

1

Cognitive disorders: potential astrocyte-based mechanism DOI Creative Commons
Shiyu Li,

Yeru Chen,

Gang Chen

и другие.

Brain Research Bulletin, Год журнала: 2024, Номер unknown, С. 111181 - 111181

Опубликована: Дек. 1, 2024

Cognitive disorders are a common clinical manifestation, including deterioration in the patient's memory ability, attention, executive power, language, and other functions. The contributing factors of cognitive numerous diverse nature, organic diseases mental disorders. Neurodegenerative type disease related to pathology neuronal death disruption glial cell balance, ultimately accompanied with impairment. Thus, disorder frequently serves as an extremely critical indicator neurodegenerative impairments negatively affect patients' daily lives. However, our understanding precise pathogenic pathways defects remains incomplete. most prevalent kind cells central nervous system called astrocytes. They have unique significance cerebral function because their wide range functions maintaining homeostasis system, regulating synaptic plasticity, so on. Dysfunction astrocytes is intimately linked disorders, we attempting understand this phenomenon predominantly from those perspectives: neuroinflammation, astrocytic senescence, connexin, Ca2+ signaling, mitochondrial dysfunction, glymphatic system.

Язык: Английский

Процитировано

1

VII Congress of Russian Biophysicists—2023, Krasnodar, Russia DOI Open Access
Anastasia A. Anashkina, Andrey B. Rubin, Nikita B. Gudimchuk

и другие.

Biophysical Reviews, Год журнала: 2023, Номер unknown

Опубликована: Окт. 13, 2023

Язык: Английский

Процитировано

3

Commentary: Early-in-life Isoflurane Exposure Alters Resting-state Functional connectivity in Juvenile Non-human Primates DOI Open Access
Viola Neudecker, Jose F. Perez‐Zoghbi, Ansgar M. Brambrink

и другие.

Journal of Immunological Sciences, Год журнала: 2024, Номер 8(2), С. 1 - 5

Опубликована: Май 17, 2024

The concern about anesthesia-induced developmental neurotoxicity (AIDN) in infants and young children arises from animal studies indicating potential long-term neurobehavioral impairments following early-in-life anesthesia exposure. While initial clinical provided ambiguous results, recent prospective assessments indicate associations between exposure later behavioral alterations. Ethical constraints confounding factors pose challenges establishing a direct causal link investigating its mechanisms. This commentary on study non-human primates (NHPs) focuses exploring the role of neuroinflammation alterations brain functional connectivity In juvenile NHPs, chronic astrogliosis amygdala correlates with this area other regions impairments, suggesting mechanism for AIDN. Despite acknowledging study's limitations, these findings emphasize need further research larger cohorts to confirm establish associated

Язык: Английский

Процитировано

0

Modeling of Blood Flow Dynamics in Rat Somatosensory Cortex DOI Creative Commons
S. Battini, Nicola Cantarutti, Christos Kotsalos

и другие.

Biomedicines, Год журнала: 2024, Номер 13(1), С. 72 - 72

Опубликована: Дек. 31, 2024

Background: The cerebral microvasculature forms a dense network of interconnected blood vessels where flow is modulated partly by astrocytes. Increased neuronal activity stimulates astrocytes to release vasoactive substances at the endfeet, altering diameters connected vessels. Methods: Our study simulated coupling between variations and vessel diameter changes driven astrocytic in rat somatosensory cortex. We developed framework with three key components: vasculature synthesized morphologies, fluid dynamics model compute each vascular segment, stochastic process replicating effect endfeet on radii. Results: was validated against experimental values from literature across cortical depths. found that local vasodilation astrocyte increased flow, especially capillaries, exhibiting layer-specific response deeper layers. Additionally, highest variability occurred emphasizing their role perfusion regulation. discovered impacted localized, clustered manner, most segments influenced two neighboring endfeet. Conclusions: These insights enhance our understanding neurovascular guide future research flow-related diseases.

Язык: Английский

Процитировано

0

Polyglucosan body density in the aged mouse hippocampus is controlled by a novel modifier locus on chromosome 1 DOI Creative Commons
Alicia Gómez-Pascual, Dow Michael Glikman,

H.-H. Ng

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2023, Номер unknown

Опубликована: Ноя. 22, 2023

In aged humans and mice, aggregates of hypobranched glycogen molecules called polyglucosan bodies (PGBs) accumulate in hippocampal astrocytes. PGBs are known to drive cognitive decline neurological diseases but remain largely unstudied the context typical brain aging. Here, we show that arise autophagy-dysregulated astrocytes C57BL/6J mouse hippocampus. To map genetic cause age-related PGB accumulation, quantified burden 32 fully sequenced BXD-recombinant inbred strains, which display a 400-fold variation at 16-18 months age. A major modifier locus was mapped chromosome 1 72-75 Mb, defined as

Язык: Английский

Процитировано

0