Oxidative stress and cellular senescence: Roles in tumor progression and therapeutic opportunities DOI Creative Commons
Ping Jin, Xudong Feng, Chongan Huang

и другие.

MedComm – Oncology, Год журнала: 2024, Номер 3(4)

Опубликована: Дек. 1, 2024

Abstract Oxidative stress results from an imbalance between the production and neutralization of reactive oxygen species. It induces oxidative damage to cellular components including proteins, lipids, nucleic acids, membranes, therefore intrinsically linking aging‐related diseases such as cancer, cardiovascular disease, neurological disorders. Emerging evidence suggests that may promote tumor development by influencing various aspects senescence, its onset, pro‐inflammatory secretion, alteration function structure. Modulating target senescence offers a novel strategy for cancer prevention treatment. However, thorough grasp specific mechanisms at play is lacking. This review will present association their regulatory role in progression treatment, with emphasis on senescence‐associated secretory phenotype, immunosenescence therapy‐induced senescence. Current agents strategies remove side effects via killing senescent cells or modulating improve antitumor efficacy be summarized. help readers better understand complex relationship also provide basis further research this area.

Язык: Английский

Real-Time Dynamic Tracking of Multiple Base Excision Repair Enzymes in Living Cells DOI
Junqiu Zhai, Han Zhang, Wenxuan Zhu

и другие.

Analytical Chemistry, Год журнала: 2025, Номер unknown

Опубликована: Фев. 27, 2025

Simultaneous in situ monitoring of base excision repair (BER) correlated enzymes like apurinic/apyrimidinic endonuclease 1 (APE1) and flap (FEN1) living cells offers valuable insights into their roles disease development cytotoxicity caused by pollutants, but comprehensive analysis is currently hindered diverse enzyme functions limited methods. In this study, we developed a dual-activatable DNA fluorescent probe (AP-FLAP) to simultaneously visualize APE1 FEN1 activities, revealing the BER-related damage various environmental pollutants within cells. The AP-FLAP was designed ingeniously integrating dumbbell structure containing 5' hairpin AP sites single oligonucleotide probe. specifically hydrolyzed sites, releasing 5-carboxy-X-rhodamine (ROX) signal, while recognized cleaved flap, 6-carboxyfluorescein (FAM) signal. allowed for independent determination activities with good specificity sensitivity. Subsequently, applied investigate induced 1-methylphenanthrene (1-MP) 6-chlorobenzo[a]pyrene (6-Cl-BaP) human umbilical vein endothelial (HUVECs). Significant 1-MP 6-Cl-BaP exposure revealed, positive correlation degree different concentrations from 0.1 100 μM. Notably, significant even at μM, concentration-dependent manner. Our work provides powerful tool elucidating BER molecular mechanisms under opens new avenues developing multifunctional nucleic acid probes wide range applications chemical biology biomedical research.

Язык: Английский

Процитировано

1

Effects of POPs-induced SIRT6 alteration on intestinal mucosal barrier function: A comprehensive review DOI Creative Commons
Guangcan Chen, Tze‐Sing Huang, Yifeng Dai

и другие.

Ecotoxicology and Environmental Safety, Год журнала: 2025, Номер 289, С. 117705 - 117705

Опубликована: Янв. 1, 2025

Persistent organic pollutants (POPs) are pervasive chemicals with significant environmental and ecological ramifications, extending to adverse human health effects due their toxicity persistence. The intestinal mucosal barrier, a sophisticated defense mechanism comprising the epithelial layer, chemistry, cellular immunity, shields host from external threats fosters symbiotic relationship bacteria. Sirtuin 6 (SIRT6), sirtuin family member, is pivotal in genome telomere stability, inflammation regulation, metabolic processes. Result shows POPs have been implicated diseases, particularly barrier dysfunction, through mechanisms such as damage, epigenetic alterations, inflammation, microbiota changes, disruptions. While impact of SIRT6 expression changes on functions has reviewed, linking remain elusive. This review summarized latest research results discussed role multiple perspectives, proposed new directions POPs, health, explored therapeutic potential SIRT6.

Язык: Английский

Процитировано

0

Genotoxicity and Cytotoxicity Induced In Vitro by Airborne Particulate Matter (PM2.5) from an Open-Cast Coal Mining Area DOI Creative Commons
Claudia Galeano–Páez, Hugo Brango, Karina Pastor–Sierra

и другие.

Atmosphere, Год журнала: 2024, Номер 15(12), С. 1420 - 1420

Опубликована: Ноя. 26, 2024

This study evaluates the cytotoxic and genotoxic effects of PM2.5 collected from an open-cast coal mining area in northern Colombia. Cyclohexane (CH), dichloromethane (DCM), acetone (ACE) extracts were obtained using Soxhlet extraction to isolate compounds different polarities. Human lymphocytes exposed extracted compounds, cytotoxicity genotoxicity assessed cytokinesis block micronucleus (CBMN) comet assays, incorporating FPG ENDO III enzymes detect oxidative DNA damage. Chemical analysis revealed that organic fractions consisted mainly modified hydrocarbons volatile compounds. The CBMN assay showed a significant increase micronuclei binucleated (MNBN) mononucleated (MNMONO) cells nucleoplasmic bridges (NPB) lymphocytes. substantial damage, particularly with ACE extract, which significantly increased oxidized purines pyrimidines. DCM induced similar effects, while CH moderate effects. CREST immunostaining aneugenic activity, extracts. These results suggest polar PM2.5, likely containing metals PAHs, contribute damage chromosomal instability. highlights need monitor composition regions implement stricter environmental policies reduce exposure health risks.

Язык: Английский

Процитировано

1

Association between chronic long-term exposure to airborne dioxins and breast cancer DOI Creative Commons
Delphine Praud,

Amina Amadou,

Thomas Coudon

и другие.

International Journal of Hygiene and Environmental Health, Год журнала: 2024, Номер 263, С. 114489 - 114489

Опубликована: Ноя. 22, 2024

Язык: Английский

Процитировано

0

Oxidative stress and cellular senescence: Roles in tumor progression and therapeutic opportunities DOI Creative Commons
Ping Jin, Xudong Feng, Chongan Huang

и другие.

MedComm – Oncology, Год журнала: 2024, Номер 3(4)

Опубликована: Дек. 1, 2024

Abstract Oxidative stress results from an imbalance between the production and neutralization of reactive oxygen species. It induces oxidative damage to cellular components including proteins, lipids, nucleic acids, membranes, therefore intrinsically linking aging‐related diseases such as cancer, cardiovascular disease, neurological disorders. Emerging evidence suggests that may promote tumor development by influencing various aspects senescence, its onset, pro‐inflammatory secretion, alteration function structure. Modulating target senescence offers a novel strategy for cancer prevention treatment. However, thorough grasp specific mechanisms at play is lacking. This review will present association their regulatory role in progression treatment, with emphasis on senescence‐associated secretory phenotype, immunosenescence therapy‐induced senescence. Current agents strategies remove side effects via killing senescent cells or modulating improve antitumor efficacy be summarized. help readers better understand complex relationship also provide basis further research this area.

Язык: Английский

Процитировано

0