Hippocampal γCaMKII dopaminylation promotes synaptic-to-nuclear signaling and memory formation DOI Creative Commons
Andrew F. Stewart, Sasha L. Fulton, Romain Durand-de Cuttoli

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Сен. 20, 2024

ABSTRACT Protein monoaminylation is a class of posttranslational modification (PTM) that contributes to transcription, physiology and behavior. While recent analyses have focused on histones as critical substrates monoaminylation, the broader repertoire monoaminylated proteins in brain remains unclear. Here, we report development/implementation chemical probe for bioorthogonal labeling, enrichment proteomics-based detection dopaminylated brain. We identified 1,557 – many synaptic including γCaMKII, which mediates Ca 2+ -dependent cellular signaling hippocampal-dependent memory. found γCaMKII dopaminylation largely synaptic-to-nuclear signaling, neuronal gene expression intrinsic excitability, contextual These results indicate role adaptive plasticity, may suggest roles these phenomena pathologies associated with altered monoaminergic signaling.

Язык: Английский

Bioorthogonal Labeling and Enrichment of Histone Monoaminylation Reveal Its Accumulation and Regulatory Function in Cancer Cell Chromatin DOI
Nan Zhang, Jinghua Wu, Farzana Hossain

и другие.

Journal of the American Chemical Society, Год журнала: 2024, Номер 146(24), С. 16714 - 16720

Опубликована: Июнь 7, 2024

Histone monoaminylation (

Язык: Английский

Процитировано

9

Construction of an anaplastic thyroid cancer stratification signature to guide immune therapy selection and validation of the pivotal gene HLF through in vitro experiments DOI Creative Commons
Pengping Li,

Kexin Yin,

Xie Yuwei

и другие.

Frontiers in Immunology, Год журнала: 2025, Номер 15

Опубликована: Янв. 13, 2025

While most thyroid cancer patients have a favorable prognosis, anaplastic carcinoma (ATC) remains particularly aggressive form with median survival time of just five months. Conventional therapies offer limited benefits for this type cancer. Our study aims to identify ATC who might bene t from immunotherapy. uses multiple algorithms by R4.2.0, and gene expression clinical data are collected TCGA, GEO local cohort. In vitro experiments, such as western blot immunofluorescence staining, performed. Using set genes uniquely expressed across various types cancer, we developed machine-learning model distinguish each within the dataset (GSE60542, GSE76039, GSE33630, GSE53157, GSE65144, GSE29265, GSE82208, GSE27155, GSE58545, GSE54958, GSE32662). These allowed us stratify into three distinct groups, exhibiting significantly different responses anti-PD1 therapy determined consensus clustering. Through weighted co-expression network analysis (WGCNA), identified 12 differentially closely associated immunotherapy outcomes. This led creation refined signature predicting ATC's immune responsiveness therapy, which was further validated using cohorts TCGA nine melanoma trials. Among genes, HLF stood out due its strong association hallmarks. revealed that impedes progression down-regulating epithelial-to-mesenchymal transition (EMT) pathway, reducing T cell exhaustion, increasing sensitivity sorafenib, demonstrated through our in-vitro experiments.

Язык: Английский

Процитировано

0

Phenethylaminylation: Preliminary In Vitro Evidence for the Covalent Transamidation of Psychedelic Phenethylamines to Glial Proteins using 3,5-Dimethoxy-4-(2-Propynyloxy)-Phenethylamine as a Model Compound DOI Creative Commons
Rajiv Rangan, Russell Petty, Suchismita Acharya

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2025, Номер unknown

Опубликована: Фев. 17, 2025

Abstract Psychedelics are well known for their ability to produce profoundly altered states of consciousness. But, more importantly, the effects psychedelics can influence neurobehavioral changes that last after these acute subjective end. This phenomenon is currently being leveraged in development psychedelic-assisted psychotherapies treatment multiple neuropsychiatric disorders. The cellular and molecular mechanisms by which single doses able mediate long-term cognitive an active area research. We hypothesize contribute long term state covalently modifying proteins. post-translational modification possible through transglutaminase-mediated transamidation amine termini glutamine carboxamide residues. Here, we synthesize utilize a propargylated analogue mescaline – classic serotonergic psychedelic phenethylamine found cacti species identify putative protein targets modifications use click-chemistry primary human astrocyte cell culture model. Our preliminary findings indicate diverse array glial proteins may be substrates transglutaminase 2-mediated monoaminylation our model (“phenethylaminylation”). Based on points, speculatively highlight new directions study this noncanonical activity.

Язык: Английский

Процитировано

0

Transglutaminase 2-mediated Histone Monoaminylation and Its Role in Cancer DOI Creative Commons
Huapeng Li, Jinghua Wu, Nan Zhang

и другие.

Bioscience Reports, Год журнала: 2024, Номер 44(8)

Опубликована: Авг. 1, 2024

Abstract Transglutaminase 2 (TGM2) has been known as a well-characterized factor regulating the progression of multiple types cancer, due to its multifunctional activities and ubiquitous signaling pathways it is involved in. As member transglutaminase family, TGM2 catalyzes protein post-translational modifications (PTMs), including monoaminylation, amide hydrolysis, cross-linking, etc., through transamidation variant glutamine-containing substrates. Recent discoveries revealed histone an important category substrates, thus identifying monoaminylation emerging epigenetic mark, which highly enriched in cancer cells possesses significant regulatory functions gene transcription. In this review, we will summarize recent advances TGM2-mediated well role discuss key research methodologies better understand unique thereby shedding light on therapeutic potential druggable target treatment.

Язык: Английский

Процитировано

2

pH-Controlled Chemoselective Rapid Azo-Coupling Reaction (CRACR) Enables Global Profiling of Serotonylation Proteome in Cancer Cells DOI
Nan Zhang, Jinghua Wu, Shuaixin Gao

и другие.

Journal of Proteome Research, Год журнала: 2024, Номер 23(10), С. 4457 - 4466

Опубликована: Авг. 29, 2024

Serotonylation has been identified as a novel protein posttranslational modification for decades, where an isopeptide bond is formed between the glutamine residue and serotonin through transamination. Transglutaminase 2 (also known TGM2 or TGase2) was proven to act main "writer" enzyme this PTM, number of key regulatory proteins (including small GTPases, fibronectin, fibrinogen, transporter, histone H3) have characterized substrates serotonylation. However, due lack pan-specific antibodies serotonylated glutamine, precise enrichment proteomic profiling serotonylation still remain challenging. In our previous research, we developed aryldiazonium probe specifically label in bioorthogonal manner, which depended on pH-controlled chemoselective rapid azo-coupling reaction. Here, report application photoactive aryldiazonium-biotin global proteome cancer cells. Thus, over 1,000 were from HCT 116 cells, many are highly related carcinogenesis. Moreover, sites these determined, attributed successful chemical approach. Overall, findings provided new insights into significant association cellular development, further suggesting that target TGM2-mediated monoaminylation may serve promising strategy therapeutics.

Язык: Английский

Процитировано

1

Hippocampal γCaMKII dopaminylation promotes synaptic-to-nuclear signaling and memory formation DOI Creative Commons
Andrew F. Stewart, Sasha L. Fulton, Romain Durand-de Cuttoli

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Сен. 20, 2024

ABSTRACT Protein monoaminylation is a class of posttranslational modification (PTM) that contributes to transcription, physiology and behavior. While recent analyses have focused on histones as critical substrates monoaminylation, the broader repertoire monoaminylated proteins in brain remains unclear. Here, we report development/implementation chemical probe for bioorthogonal labeling, enrichment proteomics-based detection dopaminylated brain. We identified 1,557 – many synaptic including γCaMKII, which mediates Ca 2+ -dependent cellular signaling hippocampal-dependent memory. found γCaMKII dopaminylation largely synaptic-to-nuclear signaling, neuronal gene expression intrinsic excitability, contextual These results indicate role adaptive plasticity, may suggest roles these phenomena pathologies associated with altered monoaminergic signaling.

Язык: Английский

Процитировано

0