Anti-atherosclerotic effects of natural compounds targeting lipid metabolism and inflammation: focus on PPARs, LXRs, and PCSK9 DOI Creative Commons
Marcella Palumbo, M. Ugolotti, Francesca Zimetti

и другие.

Atherosclerosis Plus, Год журнала: 2024, Номер unknown

Опубликована: Дек. 1, 2024

A large body of evidence has shown that modulation the nuclear receptors peroxisome proliferator-activated (PPARs), liver X (LXRs), proprotein convertase subtilisin/kexin type 9 (PCSK9) and inflammatory processes by natural compounds hypolipidemic anti-atherosclerotic effects. These beneficial outcomes are certainly related to crucial function these targets in maintaining cholesterol homeostasis regulating systemic inflammation. Currently, therapeutic scenario for cardiovascular diseases (CVD) offers a plethora widely validated functional pharmacological treatments improve health status patients. However, patients increasingly sceptical which often associated with moderate severe side The aim our review is provide collection most recent scientific on common phytochemicals, used centuries Mediterranean diet traditional chinese medicine act key regulators inflammation, could constitute important tools CVD management.

Язык: Английский

Natural products in atherosclerosis therapy by targeting PPARs: a review focusing on lipid metabolism and inflammation DOI Creative Commons
Yan Zhang, Xueying Zhang,

Shan-Rui Shi

и другие.

Frontiers in Cardiovascular Medicine, Год журнала: 2024, Номер 11

Опубликована: Апрель 18, 2024

Inflammation and dyslipidemia are critical inducing factors of atherosclerosis. Peroxisome proliferator-activated receptors (PPARs) ligand-activated transcription control the expression multiple genes that involved in lipid metabolism inflammatory responses. However, synthesized PPAR agonists exhibit contrary therapeutic effects various side atherosclerosis therapy. Natural products structural diversity have a good safety. Recent studies find natural herbs compounds attractive on by alleviating hyperlipidemia inflammation through modulation PPARs. Importantly, preparation generally causes significantly lower environmental pollution compared to chemical compounds. Therefore, it is interesting discover novel modulator develop alternative strategies for therapy based This article reviews recent findings, mainly from year 2020 present, about roles regulation PPARs their provides theoretical basis using targeting PPARs, offers valuable information researchers interested developing modulators.

Язык: Английский

Процитировано

11

Cancer cachexia: multilevel metabolic dysfunction DOI
Mauricio Berriel Díaz, Maria Rohm, Stephan Herzig

и другие.

Nature Metabolism, Год журнала: 2024, Номер unknown

Опубликована: Ноя. 22, 2024

Язык: Английский

Процитировано

6

Design, Synthesis, and Pharmacological Evaluation of Dual FXR-LIFR Modulators for the Treatment of Liver Fibrosis DOI
Pasquale Rapacciuolo, Claudia Finamore, Cristina Di Giorgio

и другие.

Journal of Medicinal Chemistry, Год журнала: 2024, Номер 67(20), С. 18334 - 18355

Опубликована: Окт. 9, 2024

Although multiple approaches have been suggested, treating mild-to-severe fibrosis in the context of metabolic dysfunction associated with liver disease (MASLD) remains a challenging area drug discovery. Pathogenesis is multifactorial, and pathogenic mechanisms are deeply intertwined; thus, it well accepted that future treatment requires development multitarget modulators. Harnessing 3,4,5-trisubstituted isoxazole scaffold, previously described as key moiety Farnesoid X receptor (FXR) agonism, herein we report discovery novel class hybrid molecules endowed dual activity toward FXR leukemia inhibitory factor (LIFR). Up to 27 new derivatives were designed synthesized. The pharmacological characterization this series resulted identification

Язык: Английский

Процитировано

4

Systemic metabolic crosstalk as driver of cancer cachexia DOI
Elisabeth Wyart, Giovanna Carrà, Elia Angelino

и другие.

Trends in Endocrinology and Metabolism, Год журнала: 2025, Номер unknown

Опубликована: Янв. 1, 2025

Язык: Английский

Процитировано

0

Recent trends in research on the role of cholesterol in leukemia: a bibliometric and visualization study DOI Creative Commons

LV Hui-juan,

Ke Lu,

Ximing Wang

и другие.

Frontiers in Immunology, Год журнала: 2025, Номер 16

Опубликована: Янв. 23, 2025

Background Cholesterol metabolism significantly impacts leukemia pathophysiology, affecting tumor cell survival, proliferation, and treatment resistance. This study employs bibliometric analysis visualization techniques to investigate research trends regarding cholesterol in identify key hotspots. Methods A systematic search of the Web Science Core Collection was performed for literature published from 1980 2024 using keywords “cholesterol” “leukemia,” yielding 1,220 articles. Bibliometric tools like VOSviewer CiteSpace were utilized visualizing citation networks thematic clusters. Results The comprised publications produced by 6,771 researchers across 1,756 institutions 68 countries, 576 journals with 5,903 unique keywords. Publication output demonstrated a significant rise 2024, peaking 2022. United States led total (381) citations (40,462), followed China (137 articles) Japan (102). Notably, U.S. had lower average than those Germany Brazil. Key included University São Paulo, Medical College Wisconsin, National Cancer Institute, prominent authors such as Maranhao Raul C. Girotti Albert W. journal Research most prolific, while Blood highest frequency. Major areas encompassed molecular biology, immunology, medicine, focusing on cholesterol-leukemia link. Keyword co-occurrence co-citation analyses reveal increasing interest topics STAT3, multidrug resistance, interactions. These insights suggest crucial further research. Discussion Our findings emphasize cholesterol’s significance leukemia, indicating its potential therapeutic target. Further exploration at intersection requires multidisciplinary collaboration. Conclusion delineates evolving landscape role pinpointing emerging future directions inform effective strategies.

Язык: Английский

Процитировано

0

Protective Vaccination of Mice Against Blood-Stage Malaria Impacts Hepatic Expression of Genes Encoding Acute-Phase Proteins and IL-6 Family Members DOI Open Access
F. Thomas Wunderlich, Daniela Gerovska, Denis Delić

и другие.

International Journal of Molecular Sciences, Год журнала: 2025, Номер 26(7), С. 3173 - 3173

Опубликована: Март 29, 2025

In response to vaccination and/or infectious agents, the liver produces acute-phase proteins (APPs) driven by IL-6, which circulate in blood plasma as components of humoral innate defense. This study investigates mice for possible effects protective against primary blood-stage infections Plasmodium chabaudi malaria on expression genes encoding APPs and IL-6 family members. Female Balb/c were vaccinated with a non-infectious vaccine prior challenge 106 P. chabaudi-infected erythrocytes, resulting about 80% survival otherwise lethal infections. Gene microarrays used determine relative transcript levels livers unvaccinated days 0, 1, 4, 8, 11 p.i. (post infectionem). Vaccination induced significant (p-value < 0.05) differences malaria-responsive toward end crisis day p.i., when recovered from These include Saa4, Apcs, Cp, Crp, described inhibitorily interact parasitic stages; F2, F7, F8, F9, F10, F13b, Plg, Plat, Serpina5, balancing coagulation vs. fibrinolysis dysregulated malaria, respectively; Hc, C8a, C8b, C8g, C9, lytic complement membrane attack complex (MAC); Cfh, Cfi, C4bp, complement-regulatory proteins. accelerated, albeit differently, malaria-induced activation all three pathways, evidenced higher C1qa, C1qb, C1qc, Fcna, Cfp, C3, C9 4 C1ra, C1s, C2 1 Serping1, multifunctional protease inhibitor C1INH, 0 Protective may also accelerate downregulation malaria-promoting lethality trans-signaling, contribute an overall accelerated recovery malaria.

Язык: Английский

Процитировано

0

Skeletal muscle endothelial dysfunction through the activin A–PGC1α axis drives progression of cancer cachexia DOI Creative Commons
Young-Mee Kim, Mark A. Sanborn, Shaluah Vijeth

и другие.

Nature Cancer, Год журнала: 2025, Номер unknown

Опубликована: Май 26, 2025

Язык: Английский

Процитировано

0

Leukemia inhibitory factor drives transcriptional programs that promote lipid accumulation and M2 polarization in macrophages DOI Creative Commons
Visnú Chaparro, Louis‐Philippe Leroux,

Aurore Lebourg

и другие.

Journal of Leukocyte Biology, Год журнала: 2024, Номер 117(1)

Опубликована: Авг. 23, 2024

Leukemia inhibitory factor, a member of the interleukin-6 cytokine family, plays central role in homeostasis and disease. Interestingly, some pleiotropic effects leukemia factor have been attributed to modulation macrophage functions although molecular underpinnings not explored at genome-wide scale. Herein, we investigated factor-driven transcriptional changes murine bone marrow-derived macrophages by RNA sequencing. In silico analyses revealed selective time-dependent remodeling gene expression programs associated with lipid metabolism cell activation. Accordingly, subset factor-upregulated transcripts related cholesterol internalization was validated real-time quantitative polymerase chain reaction. This accompanied factor-enhanced capacity for accumulation upon incubation oxidized low-density lipoprotein. Mechanistically, triggered phosphorylation (Y705 S727) nuclear translocation transcription STAT3 macrophages. Consistent this, ingenuity pathway analysis identified as an upstream regulator transcripts, including Il4ra, factor-treated Notably, priming enhanced responses interleukin-4-mediated M2 polarization (i.e. increased arginase activity encoding markers). Conversely, stimulation had no significant effect M1-polarizing stimuli (interferon-γ lipopolysaccharide). Thus, our study provides insight into landscape macrophages, shedding light on its responses. A better understanding regulatory mechanisms governing might help informing novel therapeutic approaches aiming reprogram phenotypes diseased states (e.g. cancer, atherosclerosis, infection).

Язык: Английский

Процитировано

1

FGF21 Counteracts Adipose Tissue Wasting in Cancer-Associated Cachexia DOI

Tamina Österreicher,

Kristina Kührer,

Anna-Lena Höbler

и другие.

Опубликована: Янв. 1, 2024

Язык: Английский

Процитировано

0

Anti-atherosclerotic effects of natural compounds targeting lipid metabolism and inflammation: focus on PPARs, LXRs, and PCSK9 DOI Creative Commons
Marcella Palumbo, M. Ugolotti, Francesca Zimetti

и другие.

Atherosclerosis Plus, Год журнала: 2024, Номер unknown

Опубликована: Дек. 1, 2024

A large body of evidence has shown that modulation the nuclear receptors peroxisome proliferator-activated (PPARs), liver X (LXRs), proprotein convertase subtilisin/kexin type 9 (PCSK9) and inflammatory processes by natural compounds hypolipidemic anti-atherosclerotic effects. These beneficial outcomes are certainly related to crucial function these targets in maintaining cholesterol homeostasis regulating systemic inflammation. Currently, therapeutic scenario for cardiovascular diseases (CVD) offers a plethora widely validated functional pharmacological treatments improve health status patients. However, patients increasingly sceptical which often associated with moderate severe side The aim our review is provide collection most recent scientific on common phytochemicals, used centuries Mediterranean diet traditional chinese medicine act key regulators inflammation, could constitute important tools CVD management.

Язык: Английский

Процитировано

0