Characterization of Social and Repetitive Behaviors of Mllt11/Af1q/TcF7c Conditional Knockout Mice DOI Creative Commons
Emily A. Witt,

Danielle Stanton-Turcotte,

Pristine Garay

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Авг. 1, 2024

Mllt11 (myeloid/lymphoid or mixed-lineage leukemia translocated to chromosome 11; also known as Af1q/TcF7c) has been identified a novel regulator of neural development, playing role in the migration and outgrowth cortical projection neurons. We previously reported that conditional inactivation gene mouse superficial cortex resulted reduced connectivity corpus callosum white fiber tracts, resulting thickness. However, behavioral consequences loss are unknown. Callosal abnormalities thought be present 3-5% all neurodevelopmental disorders volume correlates with core symptoms autism spectrum disorder (ASD) humans. Cortical thickness dysregulation is likewise shared among various including ASD. therefore investigated knockout upper layer 2/3 neurons using transgenic Cux2iresCre mice. Utilizing tasks designed reflect ASD symptoms, we examined behaviors both male female animals. These tests included olfaction habituation/dishabituation, three-chambered social approach, marble burying, nestlet shredding. found sex-dependent disruptions preference, shredding animals lacking Mllt11, mice presenting more than males. Understanding phenotype associated genes interest specifically context sex differences crucial individualized treatment for disorders.

Язык: Английский

Differential modulation of freezing and 22-kHz USVs by shock intensity, tone-duration matching, and anxiety levels in rodent fear-conditioning paradigms DOI

Benhuiyuan Zheng,

J. Murti Rao, Lili Bao

и другие.

Learning & Behavior, Год журнала: 2025, Номер unknown

Опубликована: Апрель 9, 2025

Язык: Английский

Процитировано

0

Exploring the combined neuroprotective effects of resveratrol and hesperidin in a scopolamine-induced rat model of cognitive impairment DOI
Bedanta Bhattacharjee, Anshul Shakya,

Naveen Shivavedi

и другие.

Neuroscience, Год журнала: 2025, Номер 579, С. 54 - 68

Опубликована: Май 26, 2025

Язык: Английский

Процитировано

0

Decoding the language of fear: Unveiling objective and subjective indicators in rodent models through a systematic review and meta-analysis DOI
Lili Bao, J. Murti Rao, Delin Yu

и другие.

Neuroscience & Biobehavioral Reviews, Год журнала: 2024, Номер 157, С. 105537 - 105537

Опубликована: Янв. 11, 2024

Язык: Английский

Процитировано

2

ChAT::Cre transgenic rats show sex‐dependent altered fear behaviors, ultrasonic vocalizations and cholinergic marker expression DOI Creative Commons

Sarah C. Tryon,

Iris M. Sakamoto,

Kris F. Kaigler

и другие.

Genes Brain & Behavior, Год журнала: 2023, Номер 22(1)

Опубликована: Янв. 13, 2023

Abstract The cholinergic system is a critical regulator of Pavlovian fear learning and extinction. As such, we have begun investigating the system's involvement in individual differences cued extinction using transgenic ChAT::Cre rat model. current study extends behavioral phenotyping line by examining both freezing behavior ultrasonic vocalizations (USVs) during paradigm. Freezing, 22 kHz USVs, 50 USVs were compared between male female ChAT::Cre+ rats their wildtype (Cre‐) littermates learning, contextual cue‐conditioned recall, extinction, generalization to novel tone. During recall froze slightly more than Cre‐ littermates, displayed significant sex freezing, USVs. Females showed males trials, but fewer distress calls recall. also produced exposure testing chambers prior tone (or shock) presentation with males, this effect was blunted females. Corroborating previous studies, overexpressed vesicular acetylcholine transporter immunolabeling basal forebrain, striatum, basolateral amygdala, hippocampus, had similar levels acetylcholinesterase numbers ChAT+ neurons as rats. This suggests that variance dependent advances theories distinct neural circuits processes regulate sexually divergent responses.

Язык: Английский

Процитировано

5

Characterization of Social and Repetitive Behaviors of Mllt11/Af1q/TcF7c Conditional Knockout Mice DOI Creative Commons
Emily A. Witt,

Danielle Stanton-Turcotte,

Pristine Garay

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Авг. 1, 2024

Mllt11 (myeloid/lymphoid or mixed-lineage leukemia translocated to chromosome 11; also known as Af1q/TcF7c) has been identified a novel regulator of neural development, playing role in the migration and outgrowth cortical projection neurons. We previously reported that conditional inactivation gene mouse superficial cortex resulted reduced connectivity corpus callosum white fiber tracts, resulting thickness. However, behavioral consequences loss are unknown. Callosal abnormalities thought be present 3-5% all neurodevelopmental disorders volume correlates with core symptoms autism spectrum disorder (ASD) humans. Cortical thickness dysregulation is likewise shared among various including ASD. therefore investigated knockout upper layer 2/3 neurons using transgenic Cux2iresCre mice. Utilizing tasks designed reflect ASD symptoms, we examined behaviors both male female animals. These tests included olfaction habituation/dishabituation, three-chambered social approach, marble burying, nestlet shredding. found sex-dependent disruptions preference, shredding animals lacking Mllt11, mice presenting more than males. Understanding phenotype associated genes interest specifically context sex differences crucial individualized treatment for disorders.

Язык: Английский

Процитировано

0