Bioinformatics based exploration of the anti-NAFLD mechanism of Wang’s empirical formula via TLR4/NF-κB/COX2 pathway DOI Creative Commons
Suhong Chen,

Chuanjie Zhou,

Jia-Hui Huang

и другие.

Molecular Medicine, Год журнала: 2024, Номер 30(1)

Опубликована: Дек. 27, 2024

Abstract Background Nonalcoholic fatty liver disease (NAFLD) has developed as a leading public wellness challenge result of changes in dietary patterns. Unfortunately, there is still lack effective pharmacotherapy methods for NAFLD. Wang’s empirical formula (WSF) demonstrated considerable clinical efficacy treating metabolic disorders years. Nevertheless, the protective effect WSF against NAFLD and its underlying mechanism remains poorly understood. Methods The model was established using 17-week high-sucrose high-fat (HSHF) diet with 32 ICR mice. In assessing therapeutic on NAFLD, we detected body weight, viscera biomarkers glycolipid metabolism serum liver, transaminase levels histopathology H&E Oil Red O staining after oral administration. chemical components were extensively identified gathered utilizing HPLC-Q-TOF/MS system, database mining from HMDB, MassBank, TCMSP databases, alongside literature searches CNKI, Wanfang VIP databases. forecast network pharmacology approach then utilized to investigate probable mechanisms by which improves based performance prospective target identification pathway enrichment analysis. Besides, molecular docking also conducted verification combination activities between active core proteins related final, validation experiments obtained pathways through ELISA, immunohistochemistry (IHC), western blot (WB) Results Pharmacodynamic outcomes indicated that intervention effectively mitigated obesity, fat accumulation organs, lipid disorders, abnormal pathology injury mice ( P < 0.05, 0.01). A total 72 existent ingredients acquired database, 254 common targets (11.6% targets) identified. Network revealed presses via modulating TNF, IL6, AKT1, IL1B, PTGS2 (COX2), other targets, primarily inflammatory signaling pathways, confirmed docking. Molecular biology further conformed could decrease factors like IL-1β, IL-6 TNF-α 0.01) expression TLR4, NF-κB COX-2 liver. Conclusion treatment protects inflammation HSHF diet-induced mice, might be suppressing TLR4/NF-κB/COX-2 reduce release cytokines Graphical abstract

Язык: Английский

AlphaFold-Based AI Docking Reveals AMPK/SIRT1-TFEB Pathway Modulation by Traditional Chinese Medicine in Metabolic-Associated Fatty Liver Disease DOI Creative Commons

Lulu Zhang,

Yi Zheng, Mingyan Shao

и другие.

Pharmacological Research, Год журнала: 2025, Номер unknown, С. 107617 - 107617

Опубликована: Янв. 1, 2025

Язык: Английский

Процитировано

1

The efficacy of traditional Chinese medicine sequential therapy in non-alcoholic fatty liver disease DOI Creative Commons
Zhang Li, Ying Qian,

Hongdi Wu

и другие.

Pakistan Journal of Medical Sciences, Год журнала: 2025, Номер 41(2), С. 512 - 518

Опубликована: Янв. 23, 2025

Objective: Sequential therapy in traditional Chinese medicine (TCM) refers to a combination of internal and external treatment methods certain order based on syndrome differentiation therapy. The aim this study was evaluate the efficacy TCM sequential that is given basis conventional management patients with non-alcoholic fatty liver disease (NAFLD). Methods: Medical records one hundred NAFLD who received at Zhejiang Provincial Tongde Hospital between February 2023 April 2024 were retrospectively analyzed. Of them, 48 routine intervention (Control group), 52 additionally treated by (TCM group). levels blood lipid indicators, function efficacy, quality life (QOL) scores compared two groups before after intervention. Results: After intervention, indicators both significantly improved preintervention considerably better Control group (P<0.05). overall higher (94.23%) (79.17%) QOL increased than Conclusions: For patients, adopting addition strategies can effectively regulate patients’ levels, restore function, improve effectiveness, life. doi: https://doi.org/10.12669/pjms.41.2.11352 How cite this: Zhang L, Qian Y, Wu H, Xu H. disease. Pak J Med Sci. 2025;41(2):512-518. This an Open Access article distributed under terms Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution, reproduction any medium, provided original work properly cited.

Язык: Английский

Процитировано

0

Patent research in academic literature. Landscape and trends with a focus on patent analytics DOI Creative Commons
Cristian Mejía, Yuya Kajikawa

Frontiers in Research Metrics and Analytics, Год журнала: 2025, Номер 9

Опубликована: Янв. 8, 2025

Patent analytics is crucial for understanding innovation dynamics and technological trends. However, a comprehensive overview of this rapidly evolving field lacking. This study presents data-driven analysis patent research, employing citation network to categorize examine research clusters. Here, we show that characterized by interconnected themes spanning fundamental systems, indicator development, methodological advancements, intellectual property management practices, diverse applications. We reveal central areas in strategies, impact, while identifying emerging focuses on environmental sustainability corporate innovation. The integration advanced analytical techniques, including AI machine learning, observed across various domains. provides insights researchers practitioners, highlighting opportunities cross-disciplinary collaboration future directions.

Язык: Английский

Процитировано

0

Exploring the mechanism of SLXG for treating nonalcoholic fatty liver disease based on network pharmacology and molecular docking DOI Creative Commons
Yang Wang, Jiaxing Wang, Zitong Chen

и другие.

Medicine, Год журнала: 2025, Номер 104(6), С. e40255 - e40255

Опубликована: Фев. 7, 2025

Background: The Shugan Lidan Decoction and Chaihu formula are traditional Chinese medicine formulas for treating liver diseases, with a history of over 1000 years. By comprehensively improving 2 medicinal formulas, Xiaoshi Granules (SLXG) has been developed the treatment nonalcoholic fatty disease (NAFLD) other liver-related metabolic diseases. Methods: First, effective active ingredients targets SLXG were determined using Traditional Medicine Systems Pharmacology Database Analysis Platform database. NAFLD identified GeneCards, OMIM, CTD databases, intersection decoction was obtained. then subjected to protein–protein interaction network analysis, Kyoto encyclopedia genes genomes (KEGG) enrichment gene ontology analysis. KEGG analysis revealed pathway. Molecular docking performed validate binding between crucial enriched in this pathway corresponding SLXG. Results: A total 219 related from 239 non-duplicated drug obtained 24 target both drug- disease-related 6 results showed that 13 gene–active ingredient bindings had energy less than −6.5. Conclusion: use pharmacology molecular technology mechanism action treatment, thus laying theoretical foundation clinical application therapy.

Язык: Английский

Процитировано

0

Analysis the Mechanism of <i>Herba</i> <i>Artemisiae</i> <i>s</i><i>copariae</i> and <i>Folium </i><i>n</i><i>elumbinis</i> in the Treatment of Non-Alcoholic Fatty Liver Disease Based on Network Pharmacology DOI

鑫凤 娄

Traditional Chinese Medicine, Год журнала: 2025, Номер 14(04), С. 1253 - 1262

Опубликована: Янв. 1, 2025

Процитировано

0

Deciphering the mechanism of Chaihu Shugan San in the treatment of nonalcoholic steatohepatitis using network pharmacology and molecular docking DOI
Yi Ren,

Kaihui Xiao,

Yujia Lu

и другие.

Journal of Pharmacy and Pharmacology, Год журнала: 2024, Номер unknown

Опубликована: Сен. 9, 2024

Abstract Objectives In China, there is a long history and rich clinical experience in treating nonalcoholic steatohepatitis (NASH) with traditional Chinese herbal medicines, including Chai Hu Shu Gan San. This study aims to investigate the potential regulatory effects of Chaihu Shugan San (CSS) on liver lipid metabolism inflammatory damage mice experimental induced by choline-deficient high-fat diet (CDHFD). Utilizing network pharmacology, we systematically explore mechanisms action therapeutic CSS against NASH. Methods Potential targets for NASH were identified using online databases. Functional enrichment protein–protein interaction analyses conducted identify hub-targeted genes elucidate underlying molecular mechanisms. The affinities active compounds evaluated docking. Finally, validated through real-time polymerase chain reaction, western blotting, immunofluorescence mice, both without treatment. Key findings reduces serum ALT AST levels mice(P &lt; 0.05) ameliorates ballooning degeneration livers thereby lowering NAS score(P 0.05). Including naringenin, high-performance liquid chromatography/mass spectrometrys 12 chromatographic peaks. Based pharmacology analysis, contains total 103 877 target genes. Transferase activity represents mechanism intervention transcriptional protein expression SIRT1 gene are significantly increased (P Conclusions Naringenin probable compound hub which involved

Язык: Английский

Процитировано

1

Elucidation of the anti-fibrotic diseases mechanism of chelerythrine by integrative approach of network pharmacology and experimental verification DOI
Wei Han,

Gui-yun Qian,

Qinrong Wang

и другие.

Natural Product Research, Год журнала: 2024, Номер unknown, С. 1 - 7

Опубликована: Авг. 18, 2024

In the present study, we conducted an integrative approach of network pharmacology and experimental validation study to elucidate underlying mechanisms Chelerythrine (CLT), in treating fibrotic diseases (FD), which are disorders characterised by excessive accumulation extracellular matrix. 27 common targets CLT against FD were analysed, these used construct PPI network. The results GO KEGG enrichment analyses suggested that exerted pharmacological effects on regulating mTOR signalling pathway, AKT-PI3K pathway apoptosis pathway. Finally, molecular docking confirmed a strong binding affinity between core target proteins. has inhibitory proliferation migration L929 cells, could promote cell apoptosis. decreased levels Bcl-2, p-AKT/AKT, p-mTOR/mTOR p-PI3K/PI3K, meanwhile increased Bax. Taken together, indicate may be potential drug for anti-fibrotic therapy.

Язык: Английский

Процитировано

0

Bioinformatics based exploration of the anti-NAFLD mechanism of Wang’s empirical formula via TLR4/NF-κB/COX2 pathway DOI Creative Commons
Suhong Chen,

Chuanjie Zhou,

Jia-Hui Huang

и другие.

Molecular Medicine, Год журнала: 2024, Номер 30(1)

Опубликована: Дек. 27, 2024

Abstract Background Nonalcoholic fatty liver disease (NAFLD) has developed as a leading public wellness challenge result of changes in dietary patterns. Unfortunately, there is still lack effective pharmacotherapy methods for NAFLD. Wang’s empirical formula (WSF) demonstrated considerable clinical efficacy treating metabolic disorders years. Nevertheless, the protective effect WSF against NAFLD and its underlying mechanism remains poorly understood. Methods The model was established using 17-week high-sucrose high-fat (HSHF) diet with 32 ICR mice. In assessing therapeutic on NAFLD, we detected body weight, viscera biomarkers glycolipid metabolism serum liver, transaminase levels histopathology H&E Oil Red O staining after oral administration. chemical components were extensively identified gathered utilizing HPLC-Q-TOF/MS system, database mining from HMDB, MassBank, TCMSP databases, alongside literature searches CNKI, Wanfang VIP databases. forecast network pharmacology approach then utilized to investigate probable mechanisms by which improves based performance prospective target identification pathway enrichment analysis. Besides, molecular docking also conducted verification combination activities between active core proteins related final, validation experiments obtained pathways through ELISA, immunohistochemistry (IHC), western blot (WB) Results Pharmacodynamic outcomes indicated that intervention effectively mitigated obesity, fat accumulation organs, lipid disorders, abnormal pathology injury mice ( P < 0.05, 0.01). A total 72 existent ingredients acquired database, 254 common targets (11.6% targets) identified. Network revealed presses via modulating TNF, IL6, AKT1, IL1B, PTGS2 (COX2), other targets, primarily inflammatory signaling pathways, confirmed docking. Molecular biology further conformed could decrease factors like IL-1β, IL-6 TNF-α 0.01) expression TLR4, NF-κB COX-2 liver. Conclusion treatment protects inflammation HSHF diet-induced mice, might be suppressing TLR4/NF-κB/COX-2 reduce release cytokines Graphical abstract

Язык: Английский

Процитировано

0