The Potential Related Genes and Mechanisms Involved in Improving the Treadmill Exercise Ability of APP/PS1 Mice DOI Open Access
Zhe Zhao,

Xingqing Wu,

Weijia Wu

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(19), С. 10244 - 10244

Опубликована: Сен. 24, 2024

Alzheimer’s disease (AD) causes a decline in skeletal muscle function, which can further exacerbate the cognitive dysfunction of patients with AD. It has been widely established that exercise improves AD brain pathology, but role is still poorly understood. In this study, we investigated effects treadmill on ability APP/PS1 transgenic mice and explored potential gene expression changes their muscle. The were subjected to for 12 weeks, followed by Morris water maze open field test. After behavioral experiments, morphology, area, collagen fiber deposition, ultrastructure determined; balance protein synthesis decomposition was analyzed; using RNA-Seq. We found strategy promote learning memory abilities mice, reduce anxiety-like behavior, improve ability, alleviate atrophy, optimize microstructure. also enhance several signaling pathways, such as JAK–STAT, Wnt, NOD-like receptors while decreasing calcium, cAMP, cGMP–PKG, other pathways. Six KEGG enrichment pathways downregulated five upregulated compared wild-type these precisely reversed after exercise. transcription factors Fosb Egr1 decreased, decrease regulated target genes Socs1, Srrm4, Il1b, trend following intervention. exercise, exhibited similar indicating enhanced ability. regulatory related identified study provide valuable insights clinical management treatment

Язык: Английский

Physical exercise for brain plasticity promotion an overview of the underlying oscillatory mechanism DOI Creative Commons
Xueyang Li,

Xuehong Qu,

Kaixuan Shi

и другие.

Frontiers in Neuroscience, Год журнала: 2024, Номер 18

Опубликована: Авг. 8, 2024

The global recognition of the importance physical exercise (PE) for human health has resulted in increased research on its effects cortical activity. Neural oscillations, which are prominent features brain activity, serve as crucial indicators studying PE function. Existing studies support idea that modifies various types neural oscillations. While EEG-related literature science exists, a comprehensive review specifically healthy populations not yet been conducted. Given demonstrated influence plasticity, particularly oscillatory it is imperative to consolidate this phenomenon. Therefore, aims summarize numerous neuromodulatory mechanisms over past decade, covering (1) resistance and aerobic training via oscillations; (2) how mind-body affects activity cognitive functioning; (3) age-Related neurodegenerative disease rehabilitation oscillation mechanisms; (4) conclusion future direction. In conclusion, effect multifaceted process, seeks comprehensively examine existing studies' understanding regulates brain, providing more scientific theoretical foundation development personalized programs further research.

Язык: Английский

Процитировано

5

Research progress on resistance exercise therapy for improving cognitive function in patients with AD and muscle atrophy DOI Creative Commons
Wenyao Li, Wei Fang,

Yier Zhang

и другие.

Frontiers in Aging Neuroscience, Год журнала: 2025, Номер 17

Опубликована: Апрель 8, 2025

Alzheimer's disease (AD) significantly reduces the quality of life patients and exacerbates burden on their families society. Resistance exercise enhances overall cognitive function elderly with AD while positively improving memory, executive function, muscle strength, reducing fall risks, alleviating psychological symptoms. As is a neurodegenerative disorder, some nerve factors are readily activated released during exercise. Therefore, several prior studies have concentrated exploring molecular mechanisms resistance impact brain neural plasticity. Recent investigations identified an intrinsic relationship between individuals pathological skeletal atrophy, establishing correlation level content. primarily targets muscle, which improves impairment in by vascular neuroinflammatory further restoring structural muscle. Furthermore, effects training vary among distinct subgroups impairment. Individuals exhibiting lower demonstrate more pronounced adaptive responses physical performance over time. Consequently, investigation warranted to determine whether tailored guidelines—such as variations frequency duration exercise—should be established for varying levels dementia, order optimize benefits those experiencing This study aimed review atrophy cognition, mechanism cognition through improvement, optimal mode elucidate additional advantages treating atrophy.

Язык: Английский

Процитировано

0

Resistance Exercise Training as a New Trend in Alzheimer’s Disease Research: From Molecular Mechanisms to Prevention DOI Open Access
Alexis Sepúlveda-Lara, Paulina Sepúlveda, Gabriel Nasri Marzuca-Nassr

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(13), С. 7084 - 7084

Опубликована: Июнь 27, 2024

Alzheimer's disease is a pathology characterized by the progressive loss of neuronal connections, which leads to gray matter atrophy in brain. most prevalent type dementia and has been classified into two types, early onset, associated with genetic factors, late environmental factors. One greatest challenges regarding high economic cost involved, why number studies aimed at prevention treatment have increased. possible approach use resistance exercise training, given that it shown neuroprotective effects disease, such as increasing cortical hippocampal volume, improving neuroplasticity, promoting cognitive function throughout life cycle. However, how training specifically prevents or ameliorates not fully characterized. Therefore, aim this review was identify molecular basis could prevent treat disease.

Язык: Английский

Процитировано

1

The Potential Related Genes and Mechanisms Involved in Improving the Treadmill Exercise Ability of APP/PS1 Mice DOI Open Access
Zhe Zhao,

Xingqing Wu,

Weijia Wu

и другие.

International Journal of Molecular Sciences, Год журнала: 2024, Номер 25(19), С. 10244 - 10244

Опубликована: Сен. 24, 2024

Alzheimer’s disease (AD) causes a decline in skeletal muscle function, which can further exacerbate the cognitive dysfunction of patients with AD. It has been widely established that exercise improves AD brain pathology, but role is still poorly understood. In this study, we investigated effects treadmill on ability APP/PS1 transgenic mice and explored potential gene expression changes their muscle. The were subjected to for 12 weeks, followed by Morris water maze open field test. After behavioral experiments, morphology, area, collagen fiber deposition, ultrastructure determined; balance protein synthesis decomposition was analyzed; using RNA-Seq. We found strategy promote learning memory abilities mice, reduce anxiety-like behavior, improve ability, alleviate atrophy, optimize microstructure. also enhance several signaling pathways, such as JAK–STAT, Wnt, NOD-like receptors while decreasing calcium, cAMP, cGMP–PKG, other pathways. Six KEGG enrichment pathways downregulated five upregulated compared wild-type these precisely reversed after exercise. transcription factors Fosb Egr1 decreased, decrease regulated target genes Socs1, Srrm4, Il1b, trend following intervention. exercise, exhibited similar indicating enhanced ability. regulatory related identified study provide valuable insights clinical management treatment

Язык: Английский

Процитировано

0