Subfunctionalization of NRC3 altered the genetic structure of the Nicotiana NRC network DOI Creative Commons
Ching‐Yi Huang,

Yu-Seng Huang,

Yu Sugihara

и другие.

PLoS Genetics, Год журнала: 2024, Номер 20(9), С. e1011402 - e1011402

Опубликована: Сен. 12, 2024

Nucleotide-binding domain and leucine-rich repeat (NLR) proteins play crucial roles in immunity against pathogens both animals plants. In solanaceous plants, activation of several sensor NLRs triggers their helper NLRs, known as NLR-required for cell death (NRC), to form resistosome complexes initiate immune responses. While the downstream NRC helpers display diverse genetic compatibility, molecular evolutionary events leading complex network architecture remained elusive. Here, we showed that NRC3 variants underwent subfunctionalization after divergence Solanum Nicotiana , altering . Natural three allelic groups displaying distinct compatibilities with NLR Rpi-blb2. Ancestral sequence reconstruction analyses natural chimeric identified six key amino acids involved sensor-helper compatibility. These residues are positioned on multiple surfaces resting homodimer, collectively contributing compatibility Upon activation, Rpi-blb2-compatible membrane-associated punctate high weight complexes, confer resistance late blight pathogen Phytophthora infestans Our findings revealed how mutations alleles lead subfunctionalization, increased complexity network.

Язык: Английский

Oligomerization-mediated autoinhibition and cofactor binding of a plant NLR DOI Creative Commons
Ma ShouCai, Chunpeng An, Aaron W. Lawson

и другие.

Nature, Год журнала: 2024, Номер 632(8026), С. 869 - 876

Опубликована: Июнь 12, 2024

Abstract Nucleotide-binding leucine-rich repeat (NLR) proteins play a pivotal role in plant immunity by recognizing pathogen effectors 1,2 . Maintaining balanced immune response is crucial, as excessive NLR expression can lead to unintended autoimmunity 3,4 Unlike most NLRs, the required for cell death 2 (NRC2) belongs small group characterized constitutively high without self-activation 5 The mechanisms underlying NRC2 autoinhibition and activation are not yet understood. Here we show that Solanum lycopersicum (tomato) ( Sl NRC2) forms dimers tetramers higher-order oligomers at elevated concentrations. Cryo-electron microscopy shows an inactive conformation of these oligomers. Dimerization oligomerization only stabilize state but also sequester from assembling into active form. Mutations dimeric or interdimeric interfaces enhance pathogen-induced Nicotiana benthamiana cryo-electron structures unexpectedly inositol hexakisphosphate (IP 6 ) pentakisphosphate bound inner surface C-terminal domain NRC2, confirmed mass spectrometry. phosphate-binding site impair phosphate binding NRC2-mediated N. Our study indicates negative regulatory mechanism suggests phosphates cofactors NRCs.

Язык: Английский

Процитировано

22

Mixed Plant Viral Infections: Complementation, Interference and Their Effects, a Review DOI Creative Commons

Mónica Roció Sánchez-Tovar,

Rafael F. Rivera-Bustamante,

Diana L. Saavedra-Trejo

и другие.

Agronomy, Год журнала: 2025, Номер 15(3), С. 620 - 620

Опубликована: Фев. 28, 2025

Viral diseases are a frequent problem in the agricultural sector, causing significant economic losses, so their management is constant challenge for producers and researchers. One of factors that often complicates control viral plants mixed infections, which occur when two or more viruses present plant, generating complex expression symptoms. During infection, following types interactions basically occur: complementation interference, effect produces synergism, antagonism, no effect. However, there also subcategories effects. This makes early detection difficult, this infection can give competitive advantage to pathogens involved. review presents updated information on infections plants, interaction categories, severity symptoms, impact vectors. The intention share better understand etiology diseases.

Язык: Английский

Процитировано

4

Balanced plant helper NLR activation by a modified host protein complex DOI
Shijia Huang, Junli Wang, Ruirui Song

и другие.

Nature, Год журнала: 2025, Номер unknown

Опубликована: Фев. 12, 2025

Язык: Английский

Процитировано

3

A disease resistance protein triggers oligomerization of its NLR helper into a hexameric resistosome to mediate innate immunity DOI Creative Commons
Jogi Madhuprakash, AmirAli Toghani, Mauricio P. Contreras

и другие.

Science Advances, Год журнала: 2024, Номер 10(45)

Опубликована: Ноя. 6, 2024

NRCs are essential helper NLR (nucleotide-binding domain and leucine-rich repeat) proteins that execute immune responses triggered by sensor NLRs. The resting state of NbNRC2 was recently shown to be a homodimer, but the sensor-activated remains unclear. Using cryo-EM, we determined structure NbNRC2, which forms hexameric inflammasome-like resistosome. Mutagenesis oligomerization interface abolished signaling, confirming functional significance Comparative structural analyses between homodimer homohexamer revealed substantial rearrangements, providing insights into activation mechanisms. Furthermore, comparisons hexamer previously reported CC-NLR pentameric assemblies features allowing an additional protomer integration. structure, assessed released AlphaFold 3 for predicting activated oligomers, revealing high-confidence modeling other amino-terminal α1 helices, region proven difficult resolve structurally. Overall, our work sheds light on mechanisms expands understanding diversity.

Язык: Английский

Процитировано

11

NLR‐mediated antiviral immunity in plants DOI Open Access

Min Zhu,

Mingfeng Feng, Xiaorong Tao

и другие.

Journal of Integrative Plant Biology, Год журнала: 2025, Номер unknown

Опубликована: Янв. 7, 2025

ABSTRACT Plant viruses cause substantial agricultural devastation and economic losses worldwide. nucleotide‐binding domain leucine‐rich repeat receptors (NLRs) play a pivotal role in detecting viral infection activating robust immune responses. Recent advances, including the elucidation of interaction mechanisms between NLRs pathogen effectors, discovery helper NLRs, resolution ZAR1 resistosome structure, have significantly deepened our understanding NLR‐mediated responses, marking new era NLR research. In this scenario, significant progress has been made study antiviral immunity. This review comprehensively summarizes plant research over past decades, with focus on recognition activation regulation, downstream signaling, engineering NLRs.

Язык: Английский

Процитировано

0

A hierarchical immune receptor network in lettuce reveals contrasting patterns of evolution in sensor and helper NLRs DOI Creative Commons
Hsuan Pai, Toshiyuki Sakai, Andrés Posbeyikian

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2025, Номер unknown

Опубликована: Фев. 25, 2025

ABSTRACT Nucleotide-binding domain and leucine-rich repeat immune receptors (NLRs) are known for their rapid evolution, even at the intraspecific level, yet rates of evolution differ significantly across various NLR classes. Within NRC (NLR Required Cell Death) network, NLRs operate in complex sensor-helper configurations to confer immunity against a diverse array pathogens, particularly Asterids. While helper typically conserved evolve slowly, sensor tend more rapidly. However, functional connections between slow fast-evolving remain poorly understood, notably important crop species. We conducted comparative analysis 40 Solanales 29 Asterales genomes explore network expansion diversification within less-studied order. Our findings reveal that has expanded less compared Solanales. functionally validated minimal with 2 helpers 9 sensors common lettuce ( Lactuca sativa ). Through selection structural modeling subclades genus, we found varying evolutionary sensors. correlation dependency, reliant on phylogenetically experiencing limited pressure. results highlight lineage- function-specific offering insights into pressures shaping plant receptor networks.

Язык: Английский

Процитировано

0

Helper NLR immune protein NRC3 evolved to evade inhibition by a cyst nematode virulence effector DOI Creative Commons
Yu Sugihara, Jiorgos Kourelis, Mauricio P. Contreras

и другие.

PLoS Genetics, Год журнала: 2025, Номер 21(4), С. e1011653 - e1011653

Опубликована: Апрель 9, 2025

Parasites can counteract host immunity by suppressing nucleotide binding and leucine-rich repeat (NLR) proteins that function as immune receptors. We previously showed a cyst nematode virulence effector SPRYSEC15 (SS15) binds inhibits oligomerisation of helper NLR in the expanded NRC1/2/3 clade preventing intramolecular rearrangements required for NRC into an activated resistosome. Here we examined degree to which from multiple Solanaceae species are sensitive suppression SS15 tested hypotheses about adaptive evolution interface between inhibitor proteins. Whereas all orthologs NRC2 were inhibited SS15, some natural variants NRC1 NRC3 insensitive suppression. Ancestral sequence reconstruction combined with functional assays revealed transitioned ancestral suppressed form one over 19 million years ago. Our analyses evolutionary trajectory receptor against parasite inhibitor, identifying key transitions NLRs this inhibition. This work reveals distinct type gene-for-gene interaction or pathogen immunosuppressors receptors contrasts coevolution AVR effectors

Язык: Английский

Процитировано

0

Helper NLR immune protein NRC3 evolved to evade inhibition by a cyst nematode virulence effector DOI Creative Commons
Yu Sugihara, Jiorgos Kourelis, Mauricio P. Contreras

и другие.

bioRxiv (Cold Spring Harbor Laboratory), Год журнала: 2024, Номер unknown

Опубликована: Июнь 16, 2024

ABSTRACT Parasites can counteract host immunity by suppressing nucleotide binding and leucine-rich repeat (NLR) proteins that function as immune receptors. We previously showed a cyst nematode virulence effector SPRYSEC15 (SS15) binds inhibits oligomerisation of helper NLR in the expanded NRC1/2/3 clade preventing intramolecular rearrangements required for NRC into an activated resistosome. Here we examined degree to which from multiple Solanaceae species are sensitive suppression SS15 tested hypotheses about adaptive evolution interface between inhibitor proteins. Whereas all orthologs NRC2 were inhibited SS15, some natural variants NRC1 NRC3 insensitive suppression. Ancestral sequence reconstruction combined with functional assays revealed transitioned ancestral suppressed form one over 19 million years ago. Our analyses evolutionary trajectory coevolution parasite its receptor target, identifying key transitions NLRs this inhibition. This work reveals distinct type gene-for-gene interaction or pathogen immunosuppressors receptors contrasts AVR effectors

Язык: Английский

Процитировано

1

Subfunctionalization of NRC3 altered the genetic structure of the Nicotiana NRC network DOI Creative Commons
Ching‐Yi Huang,

Yu-Seng Huang,

Yu Sugihara

и другие.

PLoS Genetics, Год журнала: 2024, Номер 20(9), С. e1011402 - e1011402

Опубликована: Сен. 12, 2024

Nucleotide-binding domain and leucine-rich repeat (NLR) proteins play crucial roles in immunity against pathogens both animals plants. In solanaceous plants, activation of several sensor NLRs triggers their helper NLRs, known as NLR-required for cell death (NRC), to form resistosome complexes initiate immune responses. While the downstream NRC helpers display diverse genetic compatibility, molecular evolutionary events leading complex network architecture remained elusive. Here, we showed that NRC3 variants underwent subfunctionalization after divergence Solanum Nicotiana , altering . Natural three allelic groups displaying distinct compatibilities with NLR Rpi-blb2. Ancestral sequence reconstruction analyses natural chimeric identified six key amino acids involved sensor-helper compatibility. These residues are positioned on multiple surfaces resting homodimer, collectively contributing compatibility Upon activation, Rpi-blb2-compatible membrane-associated punctate high weight complexes, confer resistance late blight pathogen Phytophthora infestans Our findings revealed how mutations alleles lead subfunctionalization, increased complexity network.

Язык: Английский

Процитировано

1