
Nature Communications, Journal Year: 2025, Volume and Issue: 16(1)
Published: April 9, 2025
Lipid dyshomeostasis and tau pathology are present in frontotemporal lobar degeneration (FTLD) Alzheimer's disease (AD). However, the relationship between lipid remains unclear. We report that GRAM Domain Containing 1B (GRAMD1B), a nonvesicular cholesterol transporter, is increased excitatory neurons of human neural organoids (HNOs) with MAPT R406W mutation. Human FTLD, AD cases, PS19 mice also have GRAMD1B expression. show overexpression increases levels free cholesterol, droplets, impairs autophagy flux. Modulating iPSC-derived alters key autophagy-related components such as PI3K, phospho-AKT, p62, well phosphorylated tau, CDK5R1. Blocking function decreases droplets. Knocking down additionally reduces CDK5R1 Our findings elucidate role nervous system highlight its relevance to FTLD AD.
Language: Английский