PKM2-mediated metabolic reprogramming of microglia in neuroinflammation DOI Creative Commons
Qi Zhang, Shasha Wang, Zhao Zhang

et al.

Cell Death Discovery, Journal Year: 2025, Volume and Issue: 11(1)

Published: April 6, 2025

Abstract Microglia, the resident immune cells of central nervous system, undergo metabolic reprogramming during neuroinflammation, playing a crucial role in pathogenesis neurological disorders such as Parkinson’s disease. This review focuses on Pyruvate Kinase M2 (PKM2), key glycolytic enzyme, and its impact microglial subsequent neuroinflammation. We explore regulatory mechanisms governing PKM2 activity, influence activation responses, contribution to progression various diseases. Finally, we highlight therapeutic potential targeting novel strategy for treating neuroinflammation-driven disorders. provides insights into molecular aiming inform development future interventions.

Language: Английский

Coptisine improves LPS-induced anxiety-like behaviors by regulating the Warburg effect in microglia via PKM2 DOI Open Access

Yiyu Qi,

Xin Zhao,

Weizhen Wu

et al.

Biomedicine & Pharmacotherapy, Journal Year: 2025, Volume and Issue: 183, P. 117837 - 117837

Published: Jan. 16, 2025

Language: Английский

Citations

1

Triclosan exposure induces liver fibrosis in mice: The heterogeneous nuclear ribonucleoprotein A1/pyruvate kinase M2 axis drives hepatic stellate cell activation DOI Creative Commons
Xiao Ni, Ziyun Wei,

Yuxuan Peng

et al.

Ecotoxicology and Environmental Safety, Journal Year: 2025, Volume and Issue: 294, P. 118113 - 118113

Published: March 30, 2025

Language: Английский

Citations

0

PKM2-mediated metabolic reprogramming of microglia in neuroinflammation DOI Creative Commons
Qi Zhang, Shasha Wang, Zhao Zhang

et al.

Cell Death Discovery, Journal Year: 2025, Volume and Issue: 11(1)

Published: April 6, 2025

Abstract Microglia, the resident immune cells of central nervous system, undergo metabolic reprogramming during neuroinflammation, playing a crucial role in pathogenesis neurological disorders such as Parkinson’s disease. This review focuses on Pyruvate Kinase M2 (PKM2), key glycolytic enzyme, and its impact microglial subsequent neuroinflammation. We explore regulatory mechanisms governing PKM2 activity, influence activation responses, contribution to progression various diseases. Finally, we highlight therapeutic potential targeting novel strategy for treating neuroinflammation-driven disorders. provides insights into molecular aiming inform development future interventions.

Language: Английский

Citations

0