The Problem of Assessing the Extent of Somatic Comorbidity in Patients with Major Depressive Disorder and Bipolar Disorder: Metabolic Syndrome, Cardiovascular Diseases, and Type II Diabetes Mellitus DOI Creative Commons

V. E. Makeenko,

Diana S. Shumskaia, А.О. Кибитов

et al.

V M BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, Journal Year: 2024, Volume and Issue: 58(4-2), P. 29 - 38

Published: Dec. 16, 2024

The deleterious impact of depression on the course and outcome somatic illness is well understood, but subject how diseases affect clinical picture, course, prognosis has received little attention. There a lack awareness actual level comorbidity in patients with depression, as relationship features disease, patient age, sex, diagnostic variants depression. Metabolic syndrome (MS), cardiovascular (CVDs), type II diabetes (T2D) are most common comorbidities, causing significant impairment contributing to mortality rates. All these closely related through shared pathophysiologic mechanisms neuro-immuno-metabolic cardio-metabolic nature. Inflammatory processes genetic risk factors play crucial role realization mechanisms. Objective: aim this study analyze existent knowledge prevalence MS, CVDs, T2D major depressive disorder (MDD) bipolar (BD) comparative aspect, taking into account possible effects sex age. Additionally, we strive highlight specific comorbid patients. Methods: We conducted literature review topic MDD BD T2D. mostly selected meta-analyses prospective studies large samples. Results: According current research, MS affects an average 30-35% individuals both MDD, minor predominance male CVDs detected 18-20% 25-45% also slight 8-10% people no gender differences. consistent identifiable signs that distinguish from non-comorbid for either or MDD. Conclusion: More research chronic required, particularly given scarcity scientific data Russian population.

Language: Английский

Treatment and long-term outcome of mental disorders: the grim picture from a quasi-epidemiological investigation in 54,826 subjects from 40 countries DOI Creative Commons
Konstantinos Ν. Fountoulakis, Grigorios N. Karakatsoulis, Seri Abraham

et al.

Psychiatry Research, Journal Year: 2025, Volume and Issue: unknown, P. 116459 - 116459

Published: March 1, 2025

Language: Английский

Citations

0

Unravelling the joint genetic architecture between psychiatric and insulin-related traits in the general population DOI Creative Commons
B. Šakić, Izel Erdogan, Giuseppe Fanelli

et al.

medRxiv (Cold Spring Harbor Laboratory), Journal Year: 2024, Volume and Issue: unknown

Published: Oct. 6, 2024

Abstract Attention-deficit/hyperactivity disorder (ADHD), autism spectrum (ASD), and obsessive-compulsive (OCD) are heritable disorders that frequently co-occur with insulin resistance (IR)-related conditions. Traditional genetic case-control comparisons challenged by the extent of heterogeneity comorbidity within across these In this study we step away from univariate analyses to let biology guide us potential links between psychiatry-related traits. We used large-scale population-based studies (N= 17,666-697,734) applied genomic structural equation modeling identify factor structure best representing joint architecture symptom scores ADHD, ASD, OCD, five IR-related traits: body mass index (BMI), fasting plasma glucose (FPG), insulin, glycated haemoglobin (HbA1c), homeostatic model assessment for IR. Subsequently performed multivariate genome-wide association on psychiatry-IR related factors explore associations unravel its biological basis. Factor indicated a three-factor fitted data (x2(df=9)=18.79, AIC=56.8, CFI=0.99, SRMR=0.068). One included ADHD traits three (BMI, FPG, HbA1c), while another encompassed OCD HbA1c. The last solely Gene-wide revealed 57 genes significantly associated ADHD-IR (p< 2.961e-06) one gene, MTNR1B (p=3.44e-07), OCD/OCS-IR factor. Gene-set found neurodevelopmental pathways. Our findings suggest shared liability psychiatric symptoms in general population, offering new perspectives molecular genetics underlying overlap somatic conditions as well biologically informed clustering psychiatry.

Language: Английский

Citations

0

The Problem of Assessing the Extent of Somatic Comorbidity in Patients with Major Depressive Disorder and Bipolar Disorder: Metabolic Syndrome, Cardiovascular Diseases, and Type II Diabetes Mellitus DOI Creative Commons

V. E. Makeenko,

Diana S. Shumskaia, А.О. Кибитов

et al.

V M BEKHTEREV REVIEW OF PSYCHIATRY AND MEDICAL PSYCHOLOGY, Journal Year: 2024, Volume and Issue: 58(4-2), P. 29 - 38

Published: Dec. 16, 2024

The deleterious impact of depression on the course and outcome somatic illness is well understood, but subject how diseases affect clinical picture, course, prognosis has received little attention. There a lack awareness actual level comorbidity in patients with depression, as relationship features disease, patient age, sex, diagnostic variants depression. Metabolic syndrome (MS), cardiovascular (CVDs), type II diabetes (T2D) are most common comorbidities, causing significant impairment contributing to mortality rates. All these closely related through shared pathophysiologic mechanisms neuro-immuno-metabolic cardio-metabolic nature. Inflammatory processes genetic risk factors play crucial role realization mechanisms. Objective: aim this study analyze existent knowledge prevalence MS, CVDs, T2D major depressive disorder (MDD) bipolar (BD) comparative aspect, taking into account possible effects sex age. Additionally, we strive highlight specific comorbid patients. Methods: We conducted literature review topic MDD BD T2D. mostly selected meta-analyses prospective studies large samples. Results: According current research, MS affects an average 30-35% individuals both MDD, minor predominance male CVDs detected 18-20% 25-45% also slight 8-10% people no gender differences. consistent identifiable signs that distinguish from non-comorbid for either or MDD. Conclusion: More research chronic required, particularly given scarcity scientific data Russian population.

Language: Английский

Citations

0