Brain Penetrant NLRP3 Inhibitors: The Discovery of a Panacea? DOI
David Harrison

Journal of Medicinal Chemistry, Journal Year: 2024, Volume and Issue: unknown

Published: Dec. 2, 2024

The NLRP3 inflammasome has attracted much interest as a drug target; however, many of the first wave inhibitors were derived from single aryl sulfonylurea starting point. physicochemical properties this molecule and most derivatives are not amenable to high brain penetration, thus limiting their potential effectiveness against disease targets where is required. disclosure novel pyridazine phenol scaffold facilitated second research toward brain-penetrant molecules, which may enable discovery treatments for Alzheimer's disease, Parkinson's multiple sclerosis cardiometabolic diseases.

Language: Английский

Discovery of Potent and Brain-Penetrant Bicyclic NLRP3 Inhibitors with Peripheral and Central In Vivo Activity DOI
Oscar Mammoliti, Rodrigo J. Carbajo, Laura Pérez‐Benito

et al.

Journal of Medicinal Chemistry, Journal Year: 2025, Volume and Issue: unknown

Published: Feb. 11, 2025

NLRP3 is a danger sensor protein responsible for inflammasome activation. This leads to pro-inflammatory cytokines release, like IL-1β, and pyroptosis, regulated cell death. Mounting evidence associates excessive activation neurodegenerative conditions, such as Alzheimer's Parkinson's diseases. Thus, inhibitors could potentially provide therapeutic benefit these disorders. We describe here the evolution of relying on pyridazine-based motif their key interactions with NLRP3. A Cryo-EM structure helped optimizing protein-ligand complementarity. Subsequently, conformational NMR studies pointed efforts toward 5,6-bicyclic cores that allowed balance between brain penetration undesirable properties, hERG inhibition. The effort culminated in compound 19, which showed moderate (mouse) good (rat) was active at low dose an LPS challenge model. Importantly, earlier central neuroinflammation model providing valuable proof concept

Language: Английский

Citations

0

Understanding a Potential Role for the NLRP3 Inflammasome in Placenta‐Mediated Pregnancy Complications DOI Creative Commons
Chloe Moss, Mark Dilworth, Lynda K. Harris

et al.

American Journal of Reproductive Immunology, Journal Year: 2025, Volume and Issue: 93(4)

Published: April 1, 2025

ABSTRACT Stillbirth affects approximately 2 million pregnancies annually and is closely linked to placental dysfunction, which may also present clinically as foetal growth restriction (FGR) or pre‐eclampsia (PE). Placental dysfunction can arise from a range of insults, including the inflammatory conditions villitis unknown aetiology (VUE) chronic histiocytic intervillositis (CHI). Despite ample research regarding pathophysiology literature surrounding inflammation more limited, with no currently established treatments. In absence infection, hypothesised be stimulated by damage‐associated molecular patterns (DAMPs), known sterile inflammation. The NLRP3 inflammasome, protein scaffold that unites within cytosol cells, proposed contributor. inflammasome dysregulated in numerous diseases has shown evidence activation through pathway via DAMPs. Studies have demonstrated upregulation its components placentally‐mediated pregnancy pathologies. However, link between seen these disorders not yet firmly established. This manuscript aims review discuss association identify potential therapeutic interventions for this pathological response.

Language: Английский

Citations

0

[18F]NP3‐627, a candidate PET imaging agent targeting the NLRP3 inflammasome in the central nervous system DOI
David M. Whitehead, Christian Fischer, Emmanuelle Briard

et al.

ChemMedChem, Journal Year: 2024, Volume and Issue: unknown

Published: Nov. 14, 2024

We describe the identification of a candidate positron emission tomography (PET) imaging agent for NLRP3 protein. plays critical role in immune system and has proven difficult target development agents due to its low cell-specific expression profile. A recently described series pyridazine-based inhibitors, with improved permeability brain-penetration properties, was used as starting point suitable PET agent. Optimization affinity, non-specific binding pharmacokinetic properties led aminopyridazine (R)-2-(6-((1-cyclopropylpiperidin-3-yl)amino)pyridazin-3-yl)-5-fluoro-3-methylphenol (17 b), which meets preclinical profile successful agent, whose tritiated version demonstrated excellent specificity radioligand saturation assay, confirming potential.

Language: Английский

Citations

0

Brain Penetrant NLRP3 Inhibitors: The Discovery of a Panacea? DOI
David Harrison

Journal of Medicinal Chemistry, Journal Year: 2024, Volume and Issue: unknown

Published: Dec. 2, 2024

The NLRP3 inflammasome has attracted much interest as a drug target; however, many of the first wave inhibitors were derived from single aryl sulfonylurea starting point. physicochemical properties this molecule and most derivatives are not amenable to high brain penetration, thus limiting their potential effectiveness against disease targets where is required. disclosure novel pyridazine phenol scaffold facilitated second research toward brain-penetrant molecules, which may enable discovery treatments for Alzheimer's disease, Parkinson's multiple sclerosis cardiometabolic diseases.

Language: Английский

Citations

0