Knockdown of USP7 alleviates atherosclerosis in ApoE-deficient mice by regulating EZH2 expression DOI Creative Commons
Yu Zhang, Yanchun Zhang

Open Life Sciences, Journal Year: 2024, Volume and Issue: 19(1)

Published: Jan. 1, 2024

Atherosclerosis (AS) is a chronic vascular disease associated with lipid accumulation. Understanding the molecular mechanisms of AS essential. Ubiquitin-specific protease 7 (USP7) deubiquitination enzyme involved in various cellular processes, including metabolism. In this study, we aimed to elucidate role USP7 progression and its underlying mechanism using ApoE-deficient mice. We found that ablation improved morphological characteristics these knockdown reduced inflammation, evidenced by decreases inflammatory markers IL-6, TNF-α, IL-1β 35, 40, 38%, respectively (

Language: Английский

Tumor-intrinsic regulators of the immune-cold microenvironment of prostate cancer DOI
Lourdes Brea, Jindan Yu

Trends in Endocrinology and Metabolism, Journal Year: 2025, Volume and Issue: unknown

Published: Jan. 1, 2025

Language: Английский

Citations

4

Systematic multi-omics investigation of androgen receptor driven gene expression and epigenetics changes in prostate cancer DOI
Lin Li, Kyung Hyun Cho, Xiuping Yu

et al.

Computers in Biology and Medicine, Journal Year: 2025, Volume and Issue: 189, P. 110000 - 110000

Published: March 8, 2025

Language: Английский

Citations

0

Regulation of Molecular Biomarkers Associated with the Progression of Prostate Cancer DOI Open Access
Miguel Martin‐Caraballo

International Journal of Molecular Sciences, Journal Year: 2024, Volume and Issue: 25(8), P. 4171 - 4171

Published: April 10, 2024

Androgen receptor signaling regulates the normal and pathological growth of prostate. In particular, survival prostate cancer cells is initially dependent on androgen signaling. Exposure to deprivation therapy leads development castration-resistant cancer. There a multitude molecular cellular changes that occur in tumor cells, including expression neuroendocrine features various biomarkers, which promotes switch androgen-independent growth. These biomarkers include transcription factors (TP53, REST, BRN2, INSM1, c-Myc), molecules (PTEN, Aurora kinases, retinoblastoma suppressor, calcium-binding proteins), receptors (glucocorticoid, receptor-variant 7), among others. It believed genetic modifications, therapeutic treatments, microenvironment are contributing progression cancers with significant heterogeneity their phenotypic characteristics. However, it not well understood how these characteristics modifications arise under specific treatment conditions. this work, we summarize some most important associated describe involved processes.

Language: Английский

Citations

2

Promising therapy for neuroendocrine prostate cancer: current status and future directions DOI Creative Commons
Fei Xin,

Jia-Wei Xue,

Ji-zhongrong Wu

et al.

Therapeutic Advances in Medical Oncology, Journal Year: 2024, Volume and Issue: 16

Published: Jan. 1, 2024

Neuroendocrine prostate cancer (NEPC) is a highly aggressive variant of castration-resistant cancer. It characterized by low or no expression the androgen receptor (AR), activation AR-independent signaling, and increased neuroendocrine phenotype. Most NEPC induced treatment deprivation therapy pathway inhibitors (ARPIs). Currently, follows strategy for small-cell lung cancer, lacking effective drugs specific options. This review summarizes potential novel targets therapies treatment, including epigenetic regulators (zeste homolog 2 inhibitors, lysine-specific demethylase 1 inhibitors), aurora kinase A poly-ADP-ribose polymerase delta-like ligand 3 targeted therapies, combination immunotherapies, etc. Other promising future directions are also discussed in this review. These may provide new opportunities NEPC.

Language: Английский

Citations

1

Knockdown of USP7 alleviates atherosclerosis in ApoE-deficient mice by regulating EZH2 expression DOI Creative Commons
Yu Zhang, Yanchun Zhang

Open Life Sciences, Journal Year: 2024, Volume and Issue: 19(1)

Published: Jan. 1, 2024

Atherosclerosis (AS) is a chronic vascular disease associated with lipid accumulation. Understanding the molecular mechanisms of AS essential. Ubiquitin-specific protease 7 (USP7) deubiquitination enzyme involved in various cellular processes, including metabolism. In this study, we aimed to elucidate role USP7 progression and its underlying mechanism using ApoE-deficient mice. We found that ablation improved morphological characteristics these knockdown reduced inflammation, evidenced by decreases inflammatory markers IL-6, TNF-α, IL-1β 35, 40, 38%, respectively (

Language: Английский

Citations

0