Blood biomarkers differentiate AD‐related versus non‐AD‐related cognitive deficits DOI Creative Commons
Kelsey R. Sewell, Lauren E. Oberlin, Thomas K. Karikari

et al.

Alzheimer s & Dementia, Journal Year: 2025, Volume and Issue: 21(3)

Published: March 1, 2025

Abstract INTRODUCTION The utility of blood‐based biomarkers for discriminating Alzheimer's disease (AD)‐related versus non‐AD‐related cognitive deficits in preclinical populations remains poorly understood. Here, we tested the capability blood markers to detect and discriminate variation performance across multiple domains a cognitively unimpaired sample. METHODS Participants ( n = 648, aged 69.9 ± 3.8, 71% female) underwent comprehensive assessment assays plasma‐based amyloid beta (Aβ)1‐42/1‐40 by mass spectrometry, phosphorylated tau (p‐tau) 181 217, p‐tau217/Aβ1‐42, glial fibrillary acidic protein (GFAP), neurofilament light (NfL). RESULTS Greater p‐tau217 was exclusively associated with poorer episodic memory (β −0.11, SE 0.04, p .003), remained so after covarying NfL. Higher NfL non‐specifically range p‐tau217. DISCUSSION Blood‐based may differentiate AD‐related deficits. This characterization will be important early intervention monitoring AD. Highlights There is heterogeneity causes decline aging. help characterize these causes. Elevated memory. cognition broad domains. Blood AD‐

Language: Английский

Influence of medical conditions on the diagnostic accuracy of plasma p‐tau217 and p‐tau217/Aβ42 DOI Creative Commons
Marcos Olvera‐Rojas, Kelsey R. Sewell, Thomas K. Karikari

et al.

Alzheimer s & Dementia, Journal Year: 2024, Volume and Issue: unknown

Published: Dec. 6, 2024

Abstract INTRODUCTION Blood‐based biomarkers (BBMs) can enable early detection of brain amyloid beta (Aβ) pathology in cognitively unimpaired individuals. However, the extent to which common medical conditions affect biomarker performance remains unclear. METHODS Participants ( n = 348) included individuals without cognitive impairment. We studied how Aβ associated with BBMs (Aβ42/40, phosphorylated tau [p‐tau] 181 and 217, p‐tau217/Aβ42, glial fibrillary acidic protein [GFAP], neurofilament light [NfL]) optimal BBM thresholds for predicting positivity whether they are obscured by presence conditions. RESULTS Plasma Aβ42/40, p‐tau181, p‐tau217, GFAP, but not NfL, were significantly Aβ. P‐tau217/Aβ42 showed best discriminative (area under curve: 0.91). The strength p‐tau217–brain associations diabetes cardiovascular DISCUSSION These results suggest may help detect caution their use due that could accuracy. Highlights GFAP NfL significant more strongly level those (AUC 0.91) discriminating an cut‐off >1.2, followed p‐tau217 at >0.46 pg/mL, slightly improving when excluding participants

Language: Английский

Citations

6

Blood biomarkers differentiate AD‐related versus non‐AD‐related cognitive deficits DOI Creative Commons
Kelsey R. Sewell, Lauren E. Oberlin, Thomas K. Karikari

et al.

Alzheimer s & Dementia, Journal Year: 2025, Volume and Issue: 21(3)

Published: March 1, 2025

Abstract INTRODUCTION The utility of blood‐based biomarkers for discriminating Alzheimer's disease (AD)‐related versus non‐AD‐related cognitive deficits in preclinical populations remains poorly understood. Here, we tested the capability blood markers to detect and discriminate variation performance across multiple domains a cognitively unimpaired sample. METHODS Participants ( n = 648, aged 69.9 ± 3.8, 71% female) underwent comprehensive assessment assays plasma‐based amyloid beta (Aβ)1‐42/1‐40 by mass spectrometry, phosphorylated tau (p‐tau) 181 217, p‐tau217/Aβ1‐42, glial fibrillary acidic protein (GFAP), neurofilament light (NfL). RESULTS Greater p‐tau217 was exclusively associated with poorer episodic memory (β −0.11, SE 0.04, p .003), remained so after covarying NfL. Higher NfL non‐specifically range p‐tau217. DISCUSSION Blood‐based may differentiate AD‐related deficits. This characterization will be important early intervention monitoring AD. Highlights There is heterogeneity causes decline aging. help characterize these causes. Elevated memory. cognition broad domains. Blood AD‐

Language: Английский

Citations

0